日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Fibroblast growth factor receptor inhibitors mitigate the neuropathogenicity of Borrelia burgdorferi or its remnants ex vivo

成纤维细胞生长因子受体抑制剂可减轻伯氏疏螺旋体或其残留物的体外神经致病性

Geetha Parthasarathy

The FGF/FGFR system in the microglial neuroinflammation with Borrelia burgdorferi: likely intersectionality with other neurological conditions

伯氏疏螺旋体引起的小胶质细胞神经炎症中的 FGF/FGFR 系统:可能与其他神经系统疾病存在交叉性

Geetha Parthasarathy, Melissa B Pattison, Cecily C Midkiff

Neuropathogenicity of non-viable Borrelia burgdorferi ex vivo

体外非存活伯氏疏螺旋体的神经致病性

Geetha Parthasarathy, Shiva Kumar Goud Gadila

Intracellular TLR7 is activated in human oligodendrocytes in response to Borrelia burgdorferi exposure

人类少突胶质细胞中的细胞内 TLR7 在暴露于伯氏疏螺旋体后被激活

Geetha Parthasarathy, Mario T Philipp

Receptor tyrosine kinases play a significant role in human oligodendrocyte inflammation and cell death associated with the Lyme disease bacterium Borrelia burgdorferi

受体酪氨酸激酶在莱姆病细菌伯氏疏螺旋体相关的人类少突胶质细胞炎症和细胞死亡中起重要作用

Geetha Parthasarathy, Mario T Philipp

The MEK/ERK pathway is the primary conduit for Borrelia burgdorferi-induced inflammation and P53-mediated apoptosis in oligodendrocytes

MEK/ERK 通路是伯氏疏螺旋体诱发炎症和 P53 介导少突胶质细胞凋亡的主要途径

Geetha Parthasarathy, Mario T Philipp

Non-viable Borrelia burgdorferi induce inflammatory mediators and apoptosis in human oligodendrocytes

无法存活的伯氏疏螺旋体在人类少突胶质细胞中诱导炎症介质和细胞凋亡

Geetha Parthasarathy, Helene B Fevrier, Mario T Philipp