日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Laquinimod treatment attenuates EAU by inhibiting both the inductive and effector phases in an APC-dependent manner.

Laquinimod 治疗通过以 APC 依赖的方式抑制诱导期和效应期来减轻 EAU

Xu Biying, Mattapallil Mary J, Jia Xiuzhi, Tang Jihong, Horai Reiko, Caspi Rachel R, Gery Igal

Tolerance Induction in Relation to the Eye

眼睛相关的耐受性诱导

Gery, Igal; Caspi, Rachel R

TMP778, a selective inhibitor of RORγt, suppresses experimental autoimmune uveitis development, but affects both Th17 and Th1 cell populations

TMP778 是一种 RORγt 选择性抑制剂,可抑制实验性自身免疫性葡萄膜炎的发展,但对 Th17 和 Th1 细胞群均有影响。

Lyu, Cancan; Bing, So Jin; Wandu, Wambui S; Xu, Biying; Shi, Guangpu; Hinshaw, Samuel J; Lobera, Mercedes; Caspi, Rachel R; Lu, Lin; Yang, Jianfei; Gery, Igal

Unlike Th1/Th17 cells, Th2/Th9 cells selectively migrate to the limbus/conjunctiva and initiate an eosinophilic infiltration process

与 Th1/Th17 细胞不同,Th2/Th9 细胞选择性地迁移至角膜缘/结膜,并启动嗜酸性粒细胞浸润过程。

Tan, Cuiyan; Wandu, Wambui S; St Leger, Anthony; Kielczewski, Jennifer; Wawrousek, Eric F; Chan, Chi-Chao; Gery, Igal

Shedding New Light on the Process of "Licensing" for Pathogenicity by Th Lymphocytes

揭示Th淋巴细胞致病性“许可”过程的新机制

Tan, Cuiyan; Wandu, Wambui S; Lee, R Steven; Hinshaw, Samuel H; Klinman, Dennis M; Wawrousek, Eric; Gery, Igal

Robert Burton Nussenblatt, MD, MPH (1948-2016), Physician-Scientist, Leading Ophthalmologist and Exemplary Teacher

罗伯特·伯顿·努森布拉特,医学博士,公共卫生硕士(1948-2016),医师科学家,杰出眼科医生和卓越教师

Gery, Igal

Inflammasomes Induced by 7-Ketocholesterol and Other Stimuli in RPE and in Bone Marrow-Derived Cells Differ Markedly in Their Production of IL-1β and IL-18

7-酮胆固醇和其他刺激物在视网膜色素上皮细胞和骨髓来源细胞中诱导的炎症小体在IL-1β和IL-18的产生方面存在显著差异

Shi, Guangpu; Chen, Siqi; Wandu, Wambui S; Ogbeifun, Osato; Nugent, Lindsey F; Maminishkis, Arvydas; Hinshaw, Samuel J H; Rodriguez, Ignacio R; Gery, Igal

Correction: The TNF-Family Ligand TL1A and Its Receptor DR3 Promote T Cell-Mediated Allergic Immunopathology by Enhancing Differentiation and Pathogenicity of IL-9-Producing T Cells

更正:TNF家族配体TL1A及其受体DR3通过增强IL-9产生T细胞的分化和致病性来促进T细胞介导的过敏性免疫病理学。

Richard, Arianne C; Tan, Cuiyan; Hawley, Eric T; Gomez-Rodriguez, Julio; Goswami, Ritobrata; Yang, Xiang-Ping; Cruz, Anthony C; Penumetcha, Pallavi; Hayes, Erika T; Pelletier, Martin; Gabay, Odile; Walsh, Matthew; Ferdinand, John R; Keane-Myers, Andrea; Choi, Yongwon; O'Shea, John J; Al-Shamkhani, Aymen; Kaplan, Mark H; Gery, Igal; Siegel, Richard M; Meylan, Françoise

The definition of lymphocyte activating factor: giving a helping hand to serendipity

淋巴细胞激活因子的定义:为意外发现提供助力

Gery, Igal

Cell-cell interaction with APC, not IL-23, is required for naive CD4 cells to acquire pathogenicity during Th17 lineage commitment

在Th17谱系分化过程中,幼稚CD4细胞获得致病性需要与抗原呈递细胞(APC)的细胞间相互作用,而非IL-23。

Shi, Guangpu; Lovaas, Jenna D; Tan, Cuiyan; Vistica, Barbara P; Wawrousek, Eric F; Aziz, Mehak K; Rigden, Rachael C; Caspi, Rachel R; Gery, Igal