日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

A Blood-Brain Barrier-Penetrant Ag(III) Corrole Compound Rescues Alzheimer's Disease Pathology by Targeting Aβ42-Induced Oxidative Stress

一种能穿透血脑屏障的Ag(III)卟啉化合物通过靶向Aβ42诱导的氧化应激来改善阿尔茨海默病病理。

Tarai, Arup; Mitra, Tuhina; Pain, Tanmoy; Mallick, Jyotiprakash; Chakraborty, Rwiddhi; Tejaswi, Kallam; Ghatak, Swagata; Kar, Sanjib

Autophagy Activators Normalize Aberrant Tau Proteostasis and Rescue Synapses in Human Familial Alzheimer's Disease iPSC-Derived Cortical Organoids.

自噬激活剂可使异常的 Tau 蛋白稳态正常化,并挽救人类家族性阿尔茨海默病 iPSC 衍生皮质类器官中的突触。

Labra Sergio R, Compher Jadon, Prabhavalkar Akhil, Almaraz Mireya, Cedeño Kwong Claudia, Baal Christine, Talantova Maria, Dolatabadi Nima, Piña-Sanz Julian, Wang Yubo, Yoon Leonard, Ghatak Swagata, Gao Zi, Zhang Yuting, Trudler Dorit, Massey Lynee, Lin Wei, Balistreri Anthony, Bula Michael, Schork Nicholas J, Mondala Tony S, Head Steven R, Kelly Jeffery W, Lipton Stuart A

Dysregulation of miRNA expression and excitation in MEF2C autism patient hiPSC-neurons and cerebral organoids.

MEF2C自闭症患者hiPSC神经元和脑类器官中miRNA表达和兴奋失调

Trudler Dorit, Ghatak Swagata, Bula Michael, Parker James, Talantova Maria, Luevanos Melissa, Labra Sergio, Grabauskas Titas, Noveral Sarah Moore, Teranaka Mayu, Schahrer Emily, Dolatabadi Nima, Bakker Clare, Lopez Kevin, Sultan Abdullah, Patel Parth, Chan Agnes, Choi Yongwook, Kawaguchi Riki, Stankiewicz Pawel, Garcia-Bassets Ivan, Kozbial Piotr, Rosenfeld Michael G, Nakanishi Nobuki, Geschwind Daniel H, Chan Shing Fai, Lin Wei, Schork Nicholas J, Ambasudhan Rajesh, Lipton Stuart A

Editorial: Olfactory dysfunction in neurodegenerative diseases

社论:神经退行性疾病中的嗅觉功能障碍

Dasgupta, Debanjan; Heinbockel, Thomas; Ghatak, Swagata

Role of Ion Channels in Alzheimer's Disease Pathophysiology

离子通道在阿尔茨海默病病理生理学中的作用

Bhoi, Ranjit; Mitra, Tuhina; Tejaswi, Kallam; Manoj, Vaishnav; Ghatak, Swagata

Autophagy activators normalize aberrant Tau proteostasis and rescue synapses in human familial Alzheimer's disease iPSC-derived cortical organoids.

自噬激活剂可使异常的 Tau 蛋白稳态正常化,并挽救人类家族性阿尔茨海默病 iPSC 衍生皮质类器官中的突触

Labra Sergio R, Compher Jadon, Prabhavalkar Akhil, Almaraz Mireya, Kwong Claudia Cedeño, Baal Christine, Talantova Maria, Dolatabadi Nima, Piña-Sanz Julian, Wang Yubo, Yoon Leonard, Ghatak Swagata, Gao Zi, Zhang Yuting, Trudler Dorit, Massey Lynee, Lin Wei, Balistreri Anthony, Bula Michael, Schork Nicholas J, Mondala Tony S, Head Steven R, Kelly Jeffery W, Lipton Stuart A

Aberrant protein S-nitrosylation contributes to hyperexcitability-induced synaptic damage in Alzheimer's disease: Mechanistic insights and potential therapies

异常的蛋白质S-亚硝基化导致阿尔茨海默病中过度兴奋引起的突触损伤:机制解析和潜在疗法

Ghatak, Swagata; Nakamura, Tomohiro; Lipton, Stuart A

S-nitrosylated TDP-43 triggers aggregation, cell-to-cell spread, and neurotoxicity in hiPSCs and in vivo models of ALS/FTD.

S-亚硝基化的TDP-43可引发hiPSCs和ALS/FTD体内模型中的聚集、细胞间传播和神经毒性

Pirie Elaine, Oh Chang-Ki, Zhang Xu, Han Xuemei, Cieplak Piotr, Scott Henry R, Deal Amanda K, Ghatak Swagata, Martinez Fernando J, Yeo Gene W, Yates John R 3rd, Nakamura Tomohiro, Lipton Stuart A

Emerging hiPSC Models for Drug Discovery in Neurodegenerative Diseases

用于神经退行性疾病药物研发的新兴hiPSC模型

Trudler, Dorit; Ghatak, Swagata; Lipton, Stuart A

Ischaemic concentrations of lactate increase TREK1 channel activity by interacting with a single histidine residue in the carboxy terminal domain

缺血性乳酸浓度通过与羧基末端结构域中的单个组氨酸残基相互作用,增加 TREK1 通道活性。

Ghatak, Swagata; Banerjee, Aditi; Sikdar, Sujit Kumar