日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Fructose-2,6-bisphosphate restores TDP-43 pathology-driven genome repair deficiency in motor neuron diseases

果糖-2,6-二磷酸可恢复运动神经元疾病中由TDP-43病理驱动的基因组修复缺陷

Chakraborty, Anirban; Mitra, Joy; Malojirao, Vikas H; Kodavati, Manohar; Mandal, Santi M; Gill, Satkarjeet K; Sreenivasmurthy, Sravan Gopalkrishnashetty; Vasquez, Velmarini; Mankevich, Mikita; Van Den Bosch, Ludo; Garruto, Ralph M; Robey, Ian; Krishnan, Balaji; Ghosh, Gourisankar; Hegde, Muralidhar L; Hazra, Tapas

F2,6BP restores mitochondrial genome integrity in Huntington's disease.

F2,6BP 可恢复亨廷顿病患者的线粒体基因组完整性。

Chakraborty Anirban, Mandal Santi M, Mankevich Mikita, Mani Rajam S, Shahabi Shandy, Kodavati Manohar, Sreenivasmurthy Sravan Gopalkrishnashetty, Tapryal Nisha, Hamilton Dale J, Krishnan Balaji, Hegde Muralidhar L, Weinfeld Michael, Ghosh Gourisankar, Hazra Tapas

RNA control of reverse transcription in a diversity-generating retroelement

RNA 控制多样性生成逆转录元件中的逆转录

Handa, Sumit; Biswas, Tapan; Chakraborty, Jeet; Ghosh, Gourisankar; Paul, Blair G; Ghosh, Partho

Chromogranin A deficiency attenuates tauopathy by altering epinephrine-alpha-adrenergic receptor signaling in PS19 mice.

嗜铬粒蛋白 A 缺乏通过改变 PS19 小鼠的肾上腺素-α-肾上腺素能受体信号传导来减轻 tau 蛋白病

Jati Suborno, Munoz-Mayorga Daniel, Shahabi Shandy, Tang Kechun, Tao Yuren, Dickson Dennis W, Litvan Irene, Ghosh Gourisankar, Mahata Sushil K, Chen Xu

Teamwork of clustered low-affinity κB sites and accessory factors regulates transcriptional strength of NF-κB RelA dimers.

聚集的低亲和力 αB 位点和辅助因子的协同作用调节 NF-αB RelA 二聚体的转录强度。

Shahabi Shandy, Biswas Tapan, Shen Yuting, Sanahmadi Rose, Zou Yaya, Ghosh Gourisankar

Cooperativity among clustered κB sites within promoters and enhancers dictates transcriptional specificity of NF-κB RelA along with specific cofactors

启动子和增强子内聚集的κB位点之间的协同作用以及特定的辅助因子决定了NF-κB RelA的转录特异性。

Shahabi, Shandy; Biswas, Tapan; Shen, Yuting; Sanahmadi, Rose; Zou, Yaya; Ghosh, Gourisankar

Fructose-2,6-bisphosphate restores DNA repair activity of PNKP and ameliorates neurodegenerative symptoms in Huntington's disease.

果糖-2,6-二磷酸可恢复 PNKP 的 DNA 修复活性,并改善亨廷顿病的神经退行性症状

Chakraborty Anirban, Sreenivasmurthy Sravan Gopalkrishnashetty, Miller Wyatt, Huai Weihan, Biswas Tapan, Mandal Santi Mohan, Boscá Lisardo, Krishnan Balaji, Ghosh Gourisankar, Hazra Tapas

Tumor suppressor NME1/NM23-H1 modulates DNA binding of NF-κB RelA.

肿瘤抑制因子 NME1/NM23-H1 调节 NF-κB RelA 的 DNA 结合

Shahabi Shandy, Maurya Mano, Subramaniam Shankar, Ghosh Gourisankar

Regulatory subunit NEMO promotes polyubiquitin-dependent induction of NF-κB through a targetable second interaction with upstream activator IKK2.

调节亚基 NEMO 通过与上游激活因子 IKK2 的可靶向的二次相互作用,促进多泛素依赖的 NF-κB 诱导

Ko Myung Soo, Cohen Samantha N, Polley Smarajit, Mahata Sushil K, Biswas Tapan, Huxford Tom, Ghosh Gourisankar

Immune Response to COVID-19 in India through Vaccination and Natural Infection.

印度通过疫苗接种和自然感染对 COVID-19 的免疫反应

Sarraf Tresa Rani, Maity Shreyasi, Ghosh Arjun, Bhattacharjee Suchandan, Pani Arijit, Saha Kaushik, Chattopadhyay Dhrubajyoti, Ghosh Gourisankar, Sen Malini