日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Parkinson's paradox: alpha-synuclein's selective strike on SNc dopamine neurons over VTA.

帕金森悖论:α-突触核蛋白选择性地攻击黑质致密部多巴胺神经元,而不是腹侧被盖区多巴胺神经元

Phan L, Miller D, Gopinath A, Lin M, Miller E J, Guenther D, Quintin S, Borg D, Hasanpour-Segherlou Z, Newman A, Sorrentino Z, Seibold J, Hoh B, Giasson B, Khoshbouei H

Parkinson's Paradox: Alpha-synuclein's Selective Strike on SNc Dopamine Neurons over VTA

帕金森悖论:α-突触核蛋白选择性地攻击黑质致密部(SNc)多巴胺能神经元而非腹侧被盖区(VTA)。

Phan, L; Miller, D; Gopinath, A; Lin, M; Gunther, D; Kiel, K; Quintin, S; Borg, D; Hasanpour-Segherlou, Z; Newman, A; Sorrentino, Z; Miller E, J; Seibold, J; Hoh, B; Giasson, B; Khoshbouei, H

Complement C1q-dependent engulfment of alpha-synuclein induces ENS-resident macrophage exhaustion and accelerates Parkinson's-like gut pathology

补体C1q依赖性α-突触核蛋白吞噬作用诱导肠神经系统驻留巨噬细胞耗竭,并加速帕金森样肠道病理。

Mackie, P M; Koshy, J; Bhogade, M; Hammoor, T; Hachmeister, W; Lloyd, G M; Paterno, G; Bolen, M; Tansey, M G; Giasson, B I; Khoshbouei, H

Neurodegeneration with brain iron accumulation, type 1 is characterized by alpha-, beta-, and gamma-synuclein neuropathology

型脑铁沉积性神经退行性疾病的特征是 α-、β- 和 γ-突触核蛋白神经病理学改变。

Galvin, J E; Giasson, B; Hurtig, H I; Lee, V M; Trojanowski, J Q

Activation of stress-activated MAP protein kinases up-regulates expression of transgenes driven by the cytomegalovirus immediate/early promoter

应激激活的MAP蛋白激酶的激活上调了由巨细胞病毒即刻/早期启动子驱动的转基因的表达。

Bruening, W; Giasson, B; Mushynski, W; Durham, H D