Giulia Chinetti相关文献与解析，生知库 | 链接生命科学的每一步

Michael T Patterson ，Maria M Firulyova ，Yingzheng Xu ，Hannah Hillman ，Courtney Bishop ，Alisha Zhu ，Grant H Hickok ，Patricia R Schrank ，Christine E Ronayne ，Zakariya Caillot ，Gavin Fredrickson ，Ainsley E Kennedy ，Nisha Acharya ，Jaap G Neels ，Giulia Chinetti ，Xavier Revelo ，Ingunn M Stromnes ，Stoyan Ivanov ，Tyler D Bold ，Konstantin Zaitsev ，Jesse W Williams

免疫/内分泌

心血管

细胞生物学

Diabetes-Induced Changes in Macrophage Biology Might Lead to Reduced Risk for Abdominal Aortic Aneurysm Development

糖尿病引起的巨噬细胞生物学变化可能会降低腹主动脉瘤发展的风险

期刊:Metabolites

影响因子:3.4

doi:10.3390/metabo12020128

Giulia Chinetti, Joseph Carboni, Joseph Murdaca, Claudine Moratal, Brigitte Sibille, Juliette Raffort, Fabien Lareyre, Elixène Jean Baptiste, Réda Hassen-Khodja, Jaap G Neels

Invalidation of the Transcriptional Modulator of Lipid Metabolism PPARβ/δ in T Cells Prevents Age-Related Alteration of Body Composition and Loss of Endurance Capacity

细胞中脂质代谢 PPARβ/δ 转录调节因子的失效可防止与年龄相关的身体成分改变和耐力下降

期刊:Frontiers in Physiology

影响因子:3.2

doi:10.3389/fphys.2021.587753

Anne-Sophie Rousseau, Joseph Murdaca, Gwenaëlle Le Menn, Brigitte Sibille, Walter Wahli, Sébastien Le Garf, Giulia Chinetti, Jaap G Neels, Isabelle Mothe-Satney

Complementary Immunometabolic Effects of Exercise and PPARβ/δ Agonist in the Context of Diet-Induced Weight Loss in Obese Female Mice

运动和 PPARβ/δ 激动剂在肥胖雌性小鼠饮食诱导减重过程中的互补免疫代谢作用

期刊:International Journal of Molecular Sciences

影响因子:0.7

doi:10.3390/ijms20205182

Sébastien Le Garf, Joseph Murdaca, Isabelle Mothe-Satney, Brigitte Sibille, Gwenaëlle Le Menn, Giulia Chinetti, Jaap G Neels, Anne-Sophie Rousseau

代谢

发表日期：2019

文献求助收藏

Human Alternative Macrophages Populate Calcified Areas of Atherosclerotic Lesions and Display Impaired RANKL-Induced Osteoclastic Bone Resorption Activity

人类替代巨噬细胞填充动脉粥样硬化病变的钙化区域并显示受损的 RANKL 诱导破骨细胞骨吸收活性

期刊:Circulation Research

影响因子:16.5

doi:10.1161/CIRCRESAHA.116.310262

Giulia Chinetti-Gbaguidi, Mehdi Daoudi, Mickael Rosa, Manjula Vinod, Loïc Louvet, Corinne Copin, Mélanie Fanchon, Jonathan Vanhoutte, Bruno Derudas, Loic Belloy, Stephan Haulon, Christophe Zawadzki, Sophie Susen, Ziad A Massy, Jérôme Eeckhoute, Bart Staels

DHA-derived oxylipins, neuroprostanes and protectins, differentially and dose-dependently modulate the inflammatory response in human macrophages: Putative mechanisms through PPAR activation

DHA 衍生的氧化脂质、神经前列腺素和保护素以不同的剂量依赖性方式调节人类巨噬细胞的炎症反应：通过 PPAR 激活的推定机制

期刊:Free Radical Biology and Medicine

影响因子:7.1

doi:10.1016/j.freeradbiomed.2016.12.018

Rémy Bosviel, Laurie Joumard-Cubizolles, Giulia Chinetti-Gbaguidi, Dominique Bayle, Corinne Copin, Nathalie Hennuyer, Isabelle Duplan, Bart Staels, Giuseppe Zanoni, Alessio Porta, Laurence Balas, Jean-Marie Galano, Camille Oger, Andrzej Mazur, Thierry Durand, Cécile Gladine

HDL in children with CKD promotes endothelial dysfunction and an abnormal vascular phenotype

CKD 儿童的 HDL 会促进内皮功能障碍和异常血管表型

期刊:Journal of the American Society of Nephrology

影响因子:10.3

doi:10.1681/ASN.2013111212

Rukshana Shroff, Thimoteus Speer, Sophie Colin, Marietta Charakida, Stephen Zewinger, Bart Staels, Giulia Chinetti-Gbaguidi, Inga Hettrich, Lucia Rohrer, Francis O'Neill, Eve McLoughlin, David Long, Catherine M Shanahan, Ulf Landmesser, Danilo Fliser, John E Deanfield

心血管

发表日期：2014

文献求助收藏

miR-206 controls LXRα expression and promotes LXR-mediated cholesterol efflux in macrophages

miR-206 控制 LXRα 表达并促进巨噬细胞中 LXR 介导的胆固醇外流

期刊:

影响因子:3.3

doi:10.1016/j.bbalip.2014.02.006

Manjula Vinod, Indumathi Chennamsetty, Sophie Colin, Loic Belloy, Federica De Paoli, Helmut Schaider, Wolfgang F Graier, Saša Frank, Dagmar Kratky, Bart Staels, Giulia Chinetti-Gbaguidi, Gerhard M Kostner

Peroxisome proliferator-activated receptor-γ activation induces 11β-hydroxysteroid dehydrogenase type 1 activity in human alternative macrophages

过氧化物酶体增殖激活受体-γ 激活可诱导人类替代巨噬细胞中 11β-羟基类固醇脱氢酶 1 型活性

期刊:Arteriosclerosis Thrombosis and Vascular Biology

影响因子:7.4

doi:10.1161/ATVBAHA.111.241364

Giulia Chinetti-Gbaguidi, Mohamed Amine Bouhlel, Corinne Copin, Christian Duhem, Bruno Derudas, Bernadette Neve, Benoit Noel, Jerome Eeckhoute, Philippe Lefebvre, Jonathan R Seckl, Bart Staels

p16INK4a deficiency promotes IL-4-induced polarization and inhibits proinflammatory signaling in macrophages

p16INK4a 缺乏会促进 IL-4 诱导的极化并抑制巨噬细胞中的促炎信号传导

期刊:Blood

影响因子:21

doi:10.1182/blood-2010-10-313106

Céline Cudejko, Kristiaan Wouters, Lucía Fuentes, Sarah Anissa Hannou, Charlotte Paquet, Kadiombo Bantubungi, Emmanuel Bouchaert, Jonathan Vanhoutte, Sébastien Fleury, Patrick Remy, Anne Tailleux, Giulia Chinetti-Gbaguidi, David Dombrowicz, Bart Staels, Réjane Paumelle

Immunology/Inflammation

发表日期：2011

文献求助收藏

Trem2 promotes foamy macrophage lipid uptake and survival in atherosclerosis

Trem2促进泡沫巨噬细胞在动脉粥样硬化中的脂质摄取和存活

Diabetes-Induced Changes in Macrophage Biology Might Lead to Reduced Risk for Abdominal Aortic Aneurysm Development

糖尿病引起的巨噬细胞生物学变化可能会降低腹主动脉瘤发展的风险

Invalidation of the Transcriptional Modulator of Lipid Metabolism PPARβ/δ in T Cells Prevents Age-Related Alteration of Body Composition and Loss of Endurance Capacity

细胞中脂质代谢 PPARβ/δ 转录调节因子的失效可防止与年龄相关的身体成分改变和耐力下降

Complementary Immunometabolic Effects of Exercise and PPARβ/δ Agonist in the Context of Diet-Induced Weight Loss in Obese Female Mice

运动和 PPARβ/δ 激动剂在肥胖雌性小鼠饮食诱导减重过程中的互补免疫代谢作用

Human Alternative Macrophages Populate Calcified Areas of Atherosclerotic Lesions and Display Impaired RANKL-Induced Osteoclastic Bone Resorption Activity

人类替代巨噬细胞填充动脉粥样硬化病变的钙化区域并显示受损的 RANKL 诱导破骨细胞骨吸收活性

DHA-derived oxylipins, neuroprostanes and protectins, differentially and dose-dependently modulate the inflammatory response in human macrophages: Putative mechanisms through PPAR activation

DHA 衍生的氧化脂质、神经前列腺素和保护素以不同的剂量依赖性方式调节人类巨噬细胞的炎症反应：通过 PPAR 激活的推定机制

HDL in children with CKD promotes endothelial dysfunction and an abnormal vascular phenotype

CKD 儿童的 HDL 会促进内皮功能障碍和异常血管表型

miR-206 controls LXRα expression and promotes LXR-mediated cholesterol efflux in macrophages

miR-206 控制 LXRα 表达并促进巨噬细胞中 LXR 介导的胆固醇外流

Peroxisome proliferator-activated receptor-γ activation induces 11β-hydroxysteroid dehydrogenase type 1 activity in human alternative macrophages

过氧化物酶体增殖激活受体-γ 激活可诱导人类替代巨噬细胞中 11β-羟基类固醇脱氢酶 1 型活性

p16INK4a deficiency promotes IL-4-induced polarization and inhibits proinflammatory signaling in macrophages

p16INK4a 缺乏会促进 IL-4 诱导的极化并抑制巨噬细胞中的促炎信号传导