Giuseppe Aceto相关文献与解析，生知库 | 链接生命科学的每一步

Nobile Veronica, Niccolini Benedetta, Pucci Cecilia, Pietrobono Roberta, Modafferi Clarissa, Esposito Carla Lucia, Cappoli Natalia, Bianchetti Giada, Maulucci Giuseppe, Aceto Giuseppe, D'Ascenzo Marcello, Chiurazzi Pietro, Russo Cinzia Dello, Genuardi Maurizio, Neri Giovanni, Ruscio Annalisa Di, Tabolacci Elisabetta

表观遗传

DNMT1

DNA甲基化

发表日期：2026

文献求助收藏

Enhanced motivated behavior mediated by pharmacological targeting of the FGF14/Nav1.6 complex in nucleus accumbens neurons

通过药物靶向伏隔核神经元中的 FGF14/Nav1.6 复合物来增强动机行为

期刊:Nature Communications

影响因子:14.7

doi:10.1038/s41467-024-55554-7

Nolan M Dvorak #, Paul A Wadsworth #, Guillermo Aquino-Miranda #, Pingyuan Wang, Douglas S Engelke, Jingheng Zhou, Nghi Nguyen, Aditya K Singh, Giuseppe Aceto, Zahra Haghighijoo, Isabella I Smith, Nana Goode, Mingxiang Zhou, Yosef Avchalumov, Evan P Troendle, Cynthia M Tapia, Haiying Chen, Reid T Po

神经

发表日期：2025

文献求助收藏

Blockade of dopamine D3 receptors improves hippocampal synaptic function and rescues age-related cognitive phenotype

阻断多巴胺 D3 受体可改善海马突触功能并挽救与年龄相关的认知表型

期刊:Aging Cell

影响因子:8

doi:10.1111/acel.14291

Maria Rosaria Tropea, Marcello Melone, Domenica Donatella Li Puma, Valeria Vacanti, Giuseppe Aceto, Bruno Bandiera, Roberta Carmela Trovato, Sebastiano Alfio Torrisi, Gian Marco Leggio, Agostino Palmeri, Marcello D'Ascenzo, Fiorenzo Conti, Claudio Grassi, Daniela Puzzo

Structural and functional alterations of neurons derived from sporadic Alzheimer's disease hiPSCs are associated with downregulation of the LIMK1-cofilin axis

散发性阿尔茨海默病 hiPSC 来源的神经元的结构和功能改变与 LIMK1-cofilin 轴的下调有关

期刊:Alzheimers Research & Therapy

影响因子:7.9

doi:10.1186/s13195-024-01632-3

Raimondo Sollazzo, Domenica Donatella Li Puma, Giuseppe Aceto, Fabiola Paciello, Claudia Colussi, Maria Gabriella Vita, Guido Maria Giuffrè, Francesco Pastore, Alessia Casamassa, Jessica Rosati, Agnese Novelli, Sabrina Maietta, Francesco Danilo Tiziano, Camillo Marra, Cristian Ripoli, Claudio Grassi

神经

发表日期：2024

文献求助收藏

Nucleoporin 153 deficiency in adult neural stem cells defines a pathological protein-network signature and defective neurogenesis in a mouse model of AD

成人神经干细胞中的核孔蛋白 153 缺乏定义了 AD 小鼠模型中的病理性蛋白质网络特征和神经发生缺陷

期刊:Stem Cell Research

影响因子:7.1

doi:10.1186/s13287-024-03805-1

Claudia Colussi #, Alessia Bertozzi #, Lucia Leone, Marco Rinaudo, Raimondo Sollazzo, Federica Conte, Elena Paccosi, Luca Nardella, Giuseppe Aceto, Domenica Donatella Li Puma, Cristian Ripoli, Maria Gabriella Vita, Camillo Marra, Marcello D'Ascenzo, Claudio Grassi

Glycine-induced activation of GPR158 increases the intrinsic excitability of medium spiny neurons in the nucleus accumbens

甘氨酸诱导的 GPR158 激活增加了伏隔核中棘神经元的内在兴奋性

期刊:Cellular and Molecular Life Sciences

影响因子:6.2

doi:10.1007/s00018-024-05260-w

Giuseppe Aceto, Luca Nardella, Simona Nanni, Valeria Pecci, Alessia Bertozzi, Sofia Nutarelli, Maria Teresa Viscomi, Claudia Colussi, Marcello D'Ascenzo #, Claudio Grassi #

神经

发表日期：2024

文献求助收藏

Intracellular accumulation of tau oligomers in astrocytes and their synaptotoxic action rely on Amyloid Precursor Protein Intracellular Domain-dependent expression of Glypican-4

星形胶质细胞中 tau 寡聚体的细胞内积累及其突触毒性作用依赖于淀粉样蛋白前体细胞内结构域依赖的 Glypican-4 表达

期刊:Progress in Neurobiology

影响因子:6.7

doi:10.1016/j.pneurobio.2023.102482

Giulia Puliatti, Domenica Donatella Li Puma, Giuseppe Aceto, Giacomo Lazzarino, Erica Acquarone, Renata Mangione, Luciano D'Adamio, Cristian Ripoli, Ottavio Arancio, Roberto Piacentini, Claudio Grassi

Cytoplasmic HDAC4 recovers synaptic function in the 3×Tg mouse model of Alzheimer's disease

细胞质 HDAC4 在阿尔茨海默病 3×Tg 小鼠模型中恢复突触功能

期刊:Neuropathology and Applied Neurobiology

影响因子:4

doi:10.1111/nan.12861

Claudia Colussi, Giuseppe Aceto, Cristian Ripoli, Alessia Bertozzi, Domenica Donatella Li Puma, Elena Paccosi, Marcello D'Ascenzo, Claudio Grassi

Intranasal Administration of KYCCSRK Peptide Rescues Brain Insulin Signaling Activation and Reduces Alzheimer's Disease-like Neuropathology in a Mouse Model for Down Syndrome

KYCCSRK 肽鼻腔给药可挽救唐氏综合症小鼠模型中的脑胰岛素信号激活并减少阿尔茨海默病样神经病理学

期刊:Antioxidants

影响因子:

doi:10.3390/antiox12010111

Antonella Tramutola, Simona Lanzillotta, Giuseppe Aceto, Sara Pagnotta, Gabriele Ruffolo, Pierangelo Cifelli, Federico Marini, Cristian Ripoli, Eleonora Palma, Claudio Grassi, Fabio Di Domenico, Marzia Perluigi, Eugenio Barone

Activation of histamine type 2 receptors enhances intrinsic excitability of medium spiny neurons in the nucleus accumbens

组胺 2 型受体的激活增强了伏隔核中棘状神经元的内在兴奋性

期刊:Journal of Physiology-London

影响因子:2.1

doi:10.1113/JP282962

Giuseppe Aceto, Luca Nardella, Simona Nanni, Valeria Pecci, Alessia Bertozzi, Claudia Colussi, Marcello D'Ascenzo, Claudio Grassi

FMR1 RNA interaction with DNMT1 blocks DNA methylation at the FMR1 locus.

FMR1 RNA 与 DNMT1 相互作用，阻断 FMR1 位点的 DNA 甲基化。

Enhanced motivated behavior mediated by pharmacological targeting of the FGF14/Nav1.6 complex in nucleus accumbens neurons

通过药物靶向伏隔核神经元中的 FGF14/Nav1.6 复合物来增强动机行为

Blockade of dopamine D3 receptors improves hippocampal synaptic function and rescues age-related cognitive phenotype

阻断多巴胺 D3 受体可改善海马突触功能并挽救与年龄相关的认知表型

Structural and functional alterations of neurons derived from sporadic Alzheimer's disease hiPSCs are associated with downregulation of the LIMK1-cofilin axis

散发性阿尔茨海默病 hiPSC 来源的神经元的结构和功能改变与 LIMK1-cofilin 轴的下调有关

Nucleoporin 153 deficiency in adult neural stem cells defines a pathological protein-network signature and defective neurogenesis in a mouse model of AD

成人神经干细胞中的核孔蛋白 153 缺乏定义了 AD 小鼠模型中的病理性蛋白质网络特征和神经发生缺陷

Glycine-induced activation of GPR158 increases the intrinsic excitability of medium spiny neurons in the nucleus accumbens

甘氨酸诱导的 GPR158 激活增加了伏隔核中棘神经元的内在兴奋性

Intracellular accumulation of tau oligomers in astrocytes and their synaptotoxic action rely on Amyloid Precursor Protein Intracellular Domain-dependent expression of Glypican-4

星形胶质细胞中 tau 寡聚体的细胞内积累及其突触毒性作用依赖于淀粉样蛋白前体细胞内结构域依赖的 Glypican-4 表达

Cytoplasmic HDAC4 recovers synaptic function in the 3×Tg mouse model of Alzheimer's disease

细胞质 HDAC4 在阿尔茨海默病 3×Tg 小鼠模型中恢复突触功能

Intranasal Administration of KYCCSRK Peptide Rescues Brain Insulin Signaling Activation and Reduces Alzheimer's Disease-like Neuropathology in a Mouse Model for Down Syndrome

KYCCSRK 肽鼻腔给药可挽救唐氏综合症小鼠模型中的脑胰岛素信号激活并减少阿尔茨海默病样神经病理学

Activation of histamine type 2 receptors enhances intrinsic excitability of medium spiny neurons in the nucleus accumbens

组胺 2 型受体的激活增强了伏隔核中棘状神经元的内在兴奋性