Gong Da-Wei相关文献与解析，生知库 | 链接生命科学的每一步

Comparative Study of Elabela and Apelin on Apelin Receptor Activation Through β-Arrestin Recruitment

通过β-Arrestin募集对Apelin受体激活的Elabela和Apelin的比较研究

期刊:Molecular Biotechnology

影响因子:2.5

doi:10.1007/s12033-022-00529-6

Zhang, Hong; Chen, Juan; Shi, Min; Xu, Feng; Zhang, Xiangcheng; Gong, Da-Wei

信号转导

发表日期：2023

文献求助收藏

A potent risk model for predicting new-onset acute coronary syndrome in patients with type 2 diabetes mellitus in Northwest China

中国西北地区2型糖尿病患者新发急性冠脉综合征的有效风险预测模型

期刊:Acta Diabetologica

影响因子:2.9

doi:10.1007/s00592-020-01484-x

Lyu, Jun; Li, Zhiying; Wei, Huiyi; Liu, Dandan; Chi, Xiaoxian; Gong, Da-Wei; Zhao, Qingbin

Fc-apelin fusion protein attenuates lipopolysaccharide-induced liver injury in mice

Fc-apelin融合蛋白可减轻小鼠脂多糖诱导的肝损伤。

期刊:Scientific Reports

影响因子:3.9

doi:10.1038/s41598-018-29491-7

Zhou, Huifen; Yang, Rongze; Wang, Weimin; Xu, Feng; Xi, Yue; Brown, Robert A; Zhang, Hong; Shi, Lin; Zhu, Dalong; Gong, Da-Wei

FSTL1 as a Potential Mediator of Exercise-Induced Cardioprotection in Post-Myocardial Infarction Rats

FSTL1作为心肌梗死后大鼠运动诱导心脏保护作用的潜在介质

期刊:Scientific Reports

影响因子:3.9

doi:10.1038/srep32424

Xi, Yue; Gong, Da-Wei; Tian, Zhenjun

Hepatic ALT isoenzymes are elevated in gluconeogenic conditions including diabetes and suppressed by insulin at the protein level

肝脏ALT同工酶在包括糖尿病在内的糖异生性疾病中升高，并可在蛋白质水平上受到胰岛素的抑制。

期刊:Diabetes-Metabolism Research and Reviews

影响因子:6

doi:10.1002/dmrr.2655

Qian, Kun; Zhong, Shao; Xie, Keming; Yu, Daozhan; Yang, Rongze; Gong, Da-Wei

Loss of function mutation in glutamic pyruvate transaminase 2 (GPT2) causes developmental encephalopathy

谷丙转氨酶2 (GPT2) 功能丧失突变会导致发育性脑病

期刊:Journal of Inherited Metabolic Disease

影响因子:3.8

doi:10.1007/s10545-015-9824-x

Celis, Katrina; Shuldiner, Scott; Haverfield, Eden V; Cappell, Joshua; Yang, Rongze; Gong, Da-Wei; Chung, Wendy K

Sulforaphane inhibits mammary adipogenesis by targeting adipose mesenchymal stem cells.

萝卜硫素通过靶向脂肪间充质干细胞抑制乳腺脂肪生成

期刊:Breast Cancer Research and Treatment

影响因子:3

doi:10.1007/s10549-013-2672-1

Li Qinglin, Xia Jixiang, Yao Yuan, Gong Da-Wei, Shi Hongfei, Zhou Qun

Metabolic adaptive ALT isoenzyme response in livers of C57/BL6 mice treated with dexamethasone

地塞米松治疗的C57/BL6小鼠肝脏中ALT同工酶的代谢适应性反应

期刊:Toxicologic Pathology

影响因子:1.8

doi:10.1177/0192623312447550

Reagan, William J; Yang, Rong-Ze; Park, Soohyun; Goldstein, Richard; Brees, Dominique; Gong, Da-Wei

代谢

发表日期：2012

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Transmembrane emp24 protein transport domain 6 is selectively expressed in pancreatic islets and implicated in insulin secretion and diabetes

跨膜emp24蛋白转运结构域6选择性地在胰岛中表达，并与胰岛素分泌和糖尿病有关。

期刊:Pancreas

影响因子:1.7

doi:10.1097/MPA.0b013e318223c7e4

Wang, Xiaochun; Yang, Rongze; Jadhao, Sanjay B; Yu, Daozhan; Hu, Hong; Glynn-Cunningham, Nicole; Sztalryd, Carole; Silver, Kristi D; Gong, Da-Wei

Fc-Elabela mitigates heart failure without liver and renal toxicity in mice

Fc-Elabela 可减轻小鼠心力衰竭，且无肝肾毒性。

Comparative Study of Elabela and Apelin on Apelin Receptor Activation Through β-Arrestin Recruitment

通过β-Arrestin募集对Apelin受体激活的Elabela和Apelin的比较研究

A potent risk model for predicting new-onset acute coronary syndrome in patients with type 2 diabetes mellitus in Northwest China

中国西北地区2型糖尿病患者新发急性冠脉综合征的有效风险预测模型

Fc-apelin fusion protein attenuates lipopolysaccharide-induced liver injury in mice

Fc-apelin融合蛋白可减轻小鼠脂多糖诱导的肝损伤。

FSTL1 as a Potential Mediator of Exercise-Induced Cardioprotection in Post-Myocardial Infarction Rats

FSTL1作为心肌梗死后大鼠运动诱导心脏保护作用的潜在介质

Hepatic ALT isoenzymes are elevated in gluconeogenic conditions including diabetes and suppressed by insulin at the protein level

肝脏ALT同工酶在包括糖尿病在内的糖异生性疾病中升高，并可在蛋白质水平上受到胰岛素的抑制。

Loss of function mutation in glutamic pyruvate transaminase 2 (GPT2) causes developmental encephalopathy

谷丙转氨酶2 (GPT2) 功能丧失突变会导致发育性脑病

Sulforaphane inhibits mammary adipogenesis by targeting adipose mesenchymal stem cells.

萝卜硫素通过靶向脂肪间充质干细胞抑制乳腺脂肪生成

Metabolic adaptive ALT isoenzyme response in livers of C57/BL6 mice treated with dexamethasone

地塞米松治疗的C57/BL6小鼠肝脏中ALT同工酶的代谢适应性反应

Transmembrane emp24 protein transport domain 6 is selectively expressed in pancreatic islets and implicated in insulin secretion and diabetes

跨膜emp24蛋白转运结构域6选择性地在胰岛中表达，并与胰岛素分泌和糖尿病有关。