Goren Itamar相关文献与解析，生知库 | 链接生命科学的每一步

Shashi Gandhi, Raizy Mitterhoff, Rachel Rapoport, Marganit Farago, Avraham Greenberg, Lauren Hodge, Sharon Eden, Christopher Benner, Alon Goren, Itamar Simon

信号转导

IHC

Goat

IgG Fc

发表日期：2022

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Study of mitotic chromatin supports a model of bookmarking by histone modifications and reveals nucleosome deposition patterns

有丝分裂染色质的研究支持组蛋白修饰的书签模型并揭示核小体沉积模式

期刊:Genome Research

影响因子:6.2

doi:10.1101/gr.230300.117

Elisheva Javasky #, Inbal Shamir #, Shashi Gandhi, Shawn Egri, Oded Sandler, Scott B Rothbart, Noam Kaplan, Jacob D Jaffe, Alon Goren #, Itamar Simon #

表观遗传

发表日期：2018

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Wound healing in mice with high-fat diet- or ob gene-induced diabetes-obesity syndromes: a comparative study.

高脂饮食或 ob 基因诱导的糖尿病-肥胖综合征小鼠的伤口愈合：一项比较研究

期刊:Experimental Diabetes Research

影响因子:

doi:10.1155/2010/476969

Seitz Oliver, SchÃ¼rmann Christoph, Hermes Nadine, MÃ¼ller Elke, Pfeilschifter Josef, Frank Stefan, Goren Itamar

A transgenic mouse model of inducible macrophage depletion: effects of diphtheria toxin-driven lysozyme M-specific cell lineage ablation on wound inflammatory, angiogenic, and contractive processes.

诱导巨噬细胞耗竭的转基因小鼠模型：白喉毒素驱动的溶菌酶 M 特异性细胞谱系消融对伤口炎症、血管生成和收缩过程的影响

期刊:American Journal of Pathology

影响因子:3.6

doi:10.2353/ajpath.2009.081002

Goren Itamar, Allmann Nadine, Yogev Nir, SchÃ¼rmann Christoph, Linke Andreas, Holdener Martin, Waisman Ari, Pfeilschifter Josef, Frank Stefan

Biphasic regulation of HMG-CoA reductase expression and activity during wound healing and its functional role in the control of keratinocyte angiogenic and proliferative responses.

伤口愈合过程中 HMG-CoA 还原酶表达和活性的双相调节及其在控制角质形成细胞血管生成和增殖反应中的功能作用

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.M709841200

Schiefelbein Dana, Goren Itamar, Fisslthaler Beate, Schmidt Helmut, Geisslinger Gerd, Pfeilschifter Josef, Frank Stefan

Severely impaired insulin signaling in chronic wounds of diabetic ob/ob mice: a potential role of tumor necrosis factor-alpha.

糖尿病 ob/ob 小鼠慢性伤口中胰岛素信号传导严重受损：肿瘤坏死因子-α 的潜在作用

期刊:American Journal of Pathology

影响因子:3.6

doi:10.2353/ajpath.2006.050293

Goren Itamar, MÃ¼ller Elke, Pfeilschifter Josef, Frank Stefan

Mitotic H3K9ac is controlled by phase-specific activity of HDAC2, HDAC3, and SIRT1

有丝分裂 H3K9ac 受 HDAC2、HDAC3 和 SIRT1 的相位特异性活性控制

Study of mitotic chromatin supports a model of bookmarking by histone modifications and reveals nucleosome deposition patterns

有丝分裂染色质的研究支持组蛋白修饰的书签模型并揭示核小体沉积模式

Wound healing in mice with high-fat diet- or ob gene-induced diabetes-obesity syndromes: a comparative study.

高脂饮食或 ob 基因诱导的糖尿病-肥胖综合征小鼠的伤口愈合：一项比较研究

A transgenic mouse model of inducible macrophage depletion: effects of diphtheria toxin-driven lysozyme M-specific cell lineage ablation on wound inflammatory, angiogenic, and contractive processes.

诱导巨噬细胞耗竭的转基因小鼠模型：白喉毒素驱动的溶菌酶 M 特异性细胞谱系消融对伤口炎症、血管生成和收缩过程的影响

Biphasic regulation of HMG-CoA reductase expression and activity during wound healing and its functional role in the control of keratinocyte angiogenic and proliferative responses.

伤口愈合过程中 HMG-CoA 还原酶表达和活性的双相调节及其在控制角质形成细胞血管生成和增殖反应中的功能作用

Severely impaired insulin signaling in chronic wounds of diabetic ob/ob mice: a potential role of tumor necrosis factor-alpha.

糖尿病 ob/ob 小鼠慢性伤口中胰岛素信号传导严重受损：肿瘤坏死因子-α 的潜在作用