日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

CXCR6+ T cells promote apoptosis and necroptosis in proximal tubules during AKI-to-CKD transition

在急性肾损伤向慢性肾病过渡过程中,CXCR6+ T 细胞促进近端肾小管细胞凋亡和坏死性凋亡。

Li, Xiaoxu; Melchinger, Isabel; Chen, Yuchu; Guo, Jiankan; Cantley, Lloyd G; Xu, Leyuan

Macrophage Derived Galectin-3 Promotes Renal Fibrosis and Diabetic Kidney Disease by Enhancing TGFβ1 Signaling

巨噬细胞衍生的半乳糖凝集素-3通过增强TGFβ1信号传导促进肾纤维化和糖尿病肾病

Chen, Yibing; Jiang, Qian; Xing, Xiaowei; Xu, Liu; Zhao, Qijin; Zhang, Qidong; Chen, Jingwen; Ma, Chunxiao; Liu, Xingfeng; Lu, Yinfei; Wan, Yanjun; Kong, Lijuan; Hou, Shaocong; Lu, Qian; Cui, Bing; Guo, Jiankan; Huang, Hongdong; Li, Pingping

VCAM-1 mediates proximal tubule-immune cell cross talk in failed tubule recovery during AKI-to-CKD transition.

VCAM-1介导AKI向CKD过渡期间肾小管恢复失败中的近端肾小管免疫细胞相互作用

Melchinger Isabel, Guo Kailin, Li Xiaoxu, Guo Jiankan, Cantley Lloyd G, Xu Leyuan

Arginase-1 Is Required for Macrophage-Mediated Renal Tubule Regeneration

精氨酸酶-1是巨噬细胞介导的肾小管再生所必需的

Shin, Naomi S; Marlier, Arnaud; Xu, Leyuan; Doilicho, Natnael; Linberg, Daniel; Guo, Jiankan; Cantley, Lloyd G

Authors' Reply: Most Arginase-1 Positive Cells Are Likely Injured S3 Proximal Tubular Cells Carrying Upregulated Phagocytotic Capacity rather than M2 Macrophages-Too Many To Be True

作者回复:大多数精氨酸酶-1阳性细胞很可能是受损的S3近端肾小管细胞,其吞噬能力上调,而非M2巨噬细胞——数量太多,难以置信。

Shin, Naomi S; Marlier, Arnaud; Xu, Leyuan; Doilicho, Natnael; Linberg, Daniel; Guo, Jiankan; Cantley, Lloyd G