Hampson Peter相关文献与解析，生知库 | 链接生命科学的每一步

Montague, Samantha J; Delierneux, Céline; Lecut, Christelle; Layios, Nathalie; Dinsdale, Robert J; Lee, Christine S-M; Poulter, Natalie S; Andrews, Robert K; Hampson, Peter; Wearn, Christopher M; Maes, Nathalie; Bishop, Jonathan; Bamford, Amy; Gardiner, Chris; Lee, Woei Ming; Iqbal, Tariq; Moiemen, Naiem; Watson, Steve P; Oury, Cécile; Harrison, Paul; Gardiner, Elizabeth E

发表日期：2018

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Changes in the pattern of plasma extracellular vesicles after severe trauma

严重创伤后血浆细胞外囊泡模式的变化

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0183640

Kuravi, Sahithi J; Yates, Clara M; Foster, Mark; Harrison, Paul; Hazeldine, Jon; Hampson, Peter; Watson, Chris; Belli, Antonio; Midwinter, Mark; Nash, Gerard B

The diagnostic and prognostic value of systems biology research in major traumatic and thermal injury: a review

系统生物学研究在严重创伤和热损伤诊断和预后中的价值：综述

期刊:Burns & Trauma

影响因子:9.6

doi:10.1186/s41038-016-0059-3

Hazeldine, Jon; Hampson, Peter; Lord, Janet M

发表日期：2016

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NK cell immunesenescence is increased by psychological but not physical stress in older adults associated with raised cortisol and reduced perforin expression

老年人中，心理压力而非生理压力会加速NK细胞的免疫衰老，并伴有皮质醇水平升高和穿孔素表达降低。

期刊:

影响因子:

doi:10.1007/s11357-015-9748-2

Duggal, Niharika Arora; Upton, Jane; Phillips, Anna C; Hampson, Peter; Lord, Janet M

Cyclin-dependent kinase 9 activity regulates neutrophil spontaneous apoptosis

细胞周期蛋白依赖性激酶9活性调节中性粒细胞自发性凋亡

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0030128

Wang, Keqing; Hampson, Peter; Hazeldine, Jon; Krystof, Vladimir; Strnad, Miroslav; Pechan, Paul; M, Janet

Novel antileukemic compound ingenol 3-angelate inhibits T cell apoptosis by activating protein kinase Ctheta.

新型抗白血病化合物英格醇 3-天使酸酯通过激活蛋白激酶 Ctheta 抑制 T 细胞凋亡

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.M109.041962

Lee Wing-Yiu, Hampson Peter, Coulthard Lydia, Ali Farrah, Salmon Mike, Lord Janet M, Scheel-Toellner Dagmar

The anti-tumor agent, ingenol-3-angelate (PEP005), promotes the recruitment of cytotoxic neutrophils by activation of vascular endothelial cells in a PKC-delta dependent manner.

抗肿瘤药物英吉醇-3-天使酸酯（PEP005）通过以 PKC-δ 依赖的方式激活血管内皮细胞，促进细胞毒性中性粒细胞的募集

期刊:Cancer Immunology Immunotherapy

影响因子:5.1

doi:10.1007/s00262-008-0458-9

Hampson Peter, Kavanagh Dean, Smith Emily, Wang Keqing, Lord Janet M, Ed Rainger G

T cells remaining after intensive chemotherapy for acute myelogenous leukemia show a broad cytokine release profile including high levels of interferon-gamma that can be further increased by a novel protein kinase C agonist PEP005.

急性髓系白血病强化化疗后剩余的 T 细胞表现出广泛的细胞因子释放谱，包括高水平的干扰素-γ，而新型蛋白激酶 C 激动剂 PEP005 可以进一步增加干扰素-γ 的水平

期刊:Cancer Immunology Immunotherapy

影响因子:5.1

doi:10.1007/s00262-006-0236-5

Ersvaer Elisabeth, Hampson Peter, Hatfield Kimberley, Ulvestad Elling, Wendelbo Ãystein, Lord Janet M, Gjertsen BjÃ¸rn Tore, Bruserud Ãystein

Soluble GPVI is elevated in injured patients: shedding is mediated by fibrin activation of GPVI

受伤患者体内可溶性GPVI水平升高：GPVI的脱落是由纤维蛋白激活GPVI介导的。

Changes in the pattern of plasma extracellular vesicles after severe trauma

严重创伤后血浆细胞外囊泡模式的变化

The diagnostic and prognostic value of systems biology research in major traumatic and thermal injury: a review

系统生物学研究在严重创伤和热损伤诊断和预后中的价值：综述

NK cell immunesenescence is increased by psychological but not physical stress in older adults associated with raised cortisol and reduced perforin expression

老年人中，心理压力而非生理压力会加速NK细胞的免疫衰老，并伴有皮质醇水平升高和穿孔素表达降低。

Cyclin-dependent kinase 9 activity regulates neutrophil spontaneous apoptosis

细胞周期蛋白依赖性激酶9活性调节中性粒细胞自发性凋亡

Novel antileukemic compound ingenol 3-angelate inhibits T cell apoptosis by activating protein kinase Ctheta.

新型抗白血病化合物英格醇 3-天使酸酯通过激活蛋白激酶 Ctheta 抑制 T 细胞凋亡

The anti-tumor agent, ingenol-3-angelate (PEP005), promotes the recruitment of cytotoxic neutrophils by activation of vascular endothelial cells in a PKC-delta dependent manner.

抗肿瘤药物英吉醇-3-天使酸酯（PEP005）通过以 PKC-δ 依赖的方式激活血管内皮细胞，促进细胞毒性中性粒细胞的募集

T cells remaining after intensive chemotherapy for acute myelogenous leukemia show a broad cytokine release profile including high levels of interferon-gamma that can be further increased by a novel protein kinase C agonist PEP005.

急性髓系白血病强化化疗后剩余的 T 细胞表现出广泛的细胞因子释放谱，包括高水平的干扰素-γ，而新型蛋白激酶 C 激动剂 PEP005 可以进一步增加干扰素-γ 的水平