Angiotensin II-mediated improvement of renal mitochondrial function via the AMPK/PGC-1α/NRF-2 pathway is superior to norepinephrine in a rat model of septic shock associated with acute renal injury.

在与急性肾损伤相关的脓毒症休克大鼠模型中，血管紧张素 II 通过 AMPK/PGC-1α/NRF-2 通路介导的肾脏线粒体功能改善优于去甲肾上腺素