ARL3 Enhances ERα Stability via USP10 Deubiquitination to Promote Endocrine Resistance and Drive Mitochondrial Metabolic Reprogramming in HR+ Breast Cancer.
ARL3 通过 USP10 去泛素化增强 ERα 稳定性，从而促进 HR+ 乳腺癌的内分泌耐药性并驱动线粒体代谢重编程。
期刊:Advanced Science
影响因子:14.1
doi:10.1002/advs.202509769
Li Han, Liu Yang, Cai Zehao, Li Kang, Gao Shun, Lan Ailin, Shu Dan, He Kuan, Liu Xin, Peng Yang, Guo Shipeng, Yu Haochen, Jin Aishun, Shen Meiying, Liu Shengchun
USP10
肿瘤
表观遗传
肿瘤免疫
泛素化
乳腺癌
代谢
代谢重编程
线粒体
SP1
