Harald Stephan相关文献与解析，生知库 | 链接生命科学的每一步

Reduction of the tumorigenic potential of human retinoblastoma cell lines by TFF1 overexpression involves p53/caspase signaling and miR-18a regulation

TFF1 过表达降低人类视网膜母细胞瘤细胞系的致瘤潜力涉及 p53/caspase 信号传导和 miR-18a 调控

Maike Busch, Jan Große-Kreul, Janina Jasmin Wirtz, Manfred Beier, Harald Stephan, Brigitte Royer-Pokora, Klaus Metz, Nicole Dünker

细胞生物学

Forced Trefoil Factor Family Peptide 3 (TFF3) Expression Reduces Growth, Viability, and Tumorigenicity of Human Retinoblastoma Cell Lines

强制三叶因子家族肽 3 (TFF3) 表达降低人类视网膜母细胞瘤细胞系的生长、活力和致瘤性

期刊:PLoS One

影响因子:2.9

doi:10.1371/journal.pone.0163025

Jan Große-Kreul, Maike Busch, Claudia Winter, Stefanie Pikos, Harald Stephan, Nicole Dünker

Additive Effects of Retinoic Acid (RA) and Bone Morphogenetic Protein 4 (BMP-4) Apoptosis Signaling in Retinoblastoma Cell Lines

视黄酸 (RA) 和骨形态发生蛋白 4 (BMP-4) 凋亡信号在视网膜母细胞瘤细胞系中的附加作用

期刊:PLoS One

影响因子:2.9

doi:10.1371/journal.pone.0131467

Patrick Müller, Rebekka Doliva, Maike Busch, Claudia Philippeit, Harald Stephan, Nicole Dünker

Sensitivity to cdk1-inhibition is modulated by p53 status in preclinical models of embryonal tumors

在胚胎肿瘤的临床前模型中，对 cdk1 抑制的敏感性受 p53 状态的调节

期刊:Oncotarget

影响因子:

doi:10.18632/oncotarget.3908

Melanie Schwermer, Sangkyun Lee, Johannes Köster, Tom van Maerken, Harald Stephan, Angelika Eggert, Katharina Morik, Johannes H Schulte, Alexander Schramm

肿瘤

发表日期：2015

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Neuroblastoma in dialog with its stroma: NTRK1 is a regulator of cellular cross-talk with Schwann cells

神经母细胞瘤与其基质的对话：NTRK1 是与施万细胞的细胞串扰的调节器

期刊:Oncotarget

影响因子:

doi:10.18632/oncotarget.2611

Kristian W Pajtler, Ellen Mahlow, Andrea Odersky, Sven Lindner, Harald Stephan, Ivo Bendix, Angelika Eggert, Alexander Schramm, Johannes H Schulte

Bone morphogenetic protein 4 (BMP4) signaling in retinoblastoma cells

视网膜母细胞瘤细胞中的骨形态发生蛋白 4 (BMP4) 信号传导

期刊:International Journal of Biological Sciences

影响因子:8.2

doi:10.7150/ijbs.6.700

Maike Haubold, Andreas Weise, Harald Stephan, Nicole Dünker

p53, miR-34a and EMP1-Newly Identified Targets of TFF3 Signaling in Y79 Retinoblastoma Cells

p53、miR-34a 和 EMP1-Y79 视网膜母细胞瘤细胞中 TFF3 信号传导的新靶点

Characterization of etoposide- and cisplatin-chemoresistant retinoblastoma cell lines

依托泊苷和顺铂化疗耐药视网膜母细胞瘤细胞系的表征

Reduction of the tumorigenic potential of human retinoblastoma cell lines by TFF1 overexpression involves p53/caspase signaling and miR-18a regulation

TFF1 过表达降低人类视网膜母细胞瘤细胞系的致瘤潜力涉及 p53/caspase 信号传导和 miR-18a 调控

Forced Trefoil Factor Family Peptide 3 (TFF3) Expression Reduces Growth, Viability, and Tumorigenicity of Human Retinoblastoma Cell Lines

强制三叶因子家族肽 3 (TFF3) 表达降低人类视网膜母细胞瘤细胞系的生长、活力和致瘤性

Additive Effects of Retinoic Acid (RA) and Bone Morphogenetic Protein 4 (BMP-4) Apoptosis Signaling in Retinoblastoma Cell Lines

视黄酸 (RA) 和骨形态发生蛋白 4 (BMP-4) 凋亡信号在视网膜母细胞瘤细胞系中的附加作用

Sensitivity to cdk1-inhibition is modulated by p53 status in preclinical models of embryonal tumors

在胚胎肿瘤的临床前模型中，对 cdk1 抑制的敏感性受 p53 状态的调节

Neuroblastoma in dialog with its stroma: NTRK1 is a regulator of cellular cross-talk with Schwann cells

神经母细胞瘤与其基质的对话：NTRK1 是与施万细胞的细胞串扰的调节器

Bone morphogenetic protein 4 (BMP4) signaling in retinoblastoma cells

视网膜母细胞瘤细胞中的骨形态发生蛋白 4 (BMP4) 信号传导