文献库，生知库 | 链接生命科学的每一步

CD11c⁺ cells primed with unrelated antigens facilitate an accelerated immune response to influenza virus in mice

用无关抗原预处理的CD11c⁺细胞可促进小鼠对流感病毒的加速免疫应答。

doi:10.1002/eji.201343587

Richert, Laura E; Rynda-Apple, Agnieszka; Harmsen, Ann L; Han, Soo; Wiley, James A; Douglas, Trevor; Larson, Kyle; Morton, Rachelle V; Harmsen, Allen G

Biomimetic antigenic nanoparticles elicit controlled protective immune response to influenza

仿生抗原纳米颗粒可诱导针对流感的可控保护性免疫反应

期刊:ACS Nano

影响因子:16

doi:10.1021/nn4006544

Patterson, Dustin P; Rynda-Apple, Agnieszka; Harmsen, Ann L; Harmsen, Allen G; Douglas, Trevor

Inducible bronchus-associated lymphoid tissue (iBALT) synergizes with local lymph nodes during antiviral CD4+ T cell responses

诱导型支气管相关淋巴组织 (iBALT) 在抗病毒 CD4+ T 细胞反应中与局部淋巴结协同作用

期刊:Lymphatic Research and Biology

影响因子:1.9

doi:10.1089/lrb.2013.0015

Richert, Laura E; Harmsen, Ann L; Rynda-Apple, Agnieszka; Wiley, James A; Servid, Amy E; Douglas, Trevor; Harmsen, Allen G

Pneumocystis elicits a STAT6-dependent, strain-specific innate immune response and airway hyperresponsiveness

肺孢子菌可引发STAT6依赖性的、菌株特异性的先天免疫反应和气道高反应性。

期刊:American Journal of Respiratory Cell and Molecular Biology

影响因子:5.3

doi:10.1165/rcmb.2011-0154OC

Swain, Steve D; Meissner, Nicole N; Siemsen, Dan W; McInnerney, Kate; Harmsen, Allen G

Virus-like particle-induced protection against MRSA pneumonia is dependent on IL-13 and enhancement of phagocyte function

病毒样颗粒诱导的针对耐甲氧西林金黄色葡萄球菌肺炎的保护作用依赖于IL-13和吞噬细胞功能的增强。

期刊:American Journal of Pathology

影响因子:3.6

doi:10.1016/j.ajpath.2012.03.018

Rynda-Apple, Agnieszka; Dobrinen, Erin; McAlpine, Mark; Read, Amanda; Harmsen, Ann; Richert, Laura E; Calverley, Matthew; Pallister, Kyler; Voyich, Jovanka; Wiley, James A; Johnson, Ben; Young, Mark; Douglas, Trevor; Harmsen, Allen G

Resident alveolar macrophages are susceptible to and permissive of Coxiella burnetii infection

肺泡驻留巨噬细胞易受伯氏考克斯体感染，并允许其感染。

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0051941

Calverley, Matthew; Erickson, Sara; Read, Amanda J; Harmsen, Allen G

Influenza virus infection decreases tracheal mucociliary velocity and clearance of Streptococcus pneumoniae

流感病毒感染会降低气管黏膜纤毛运动速度和肺炎链球菌的清除能力。

期刊:American Journal of Respiratory Cell and Molecular Biology

影响因子:5.3

doi:10.1165/rcmb.2007-0417OC

Pittet, Lynnelle A; Hall-Stoodley, Luanne; Rutkowski, Melanie R; Harmsen, Allen G

Pneumococcal surface protein A contributes to secondary Streptococcus pneumoniae infection after influenza virus infection

肺炎球菌表面蛋白A在流感病毒感染后可导致继发性肺炎链球菌感染。

期刊:Journal of Infectious Diseases

影响因子:4.5

doi:10.1086/600871

King, Quinton O; Lei, Benfang; Harmsen, Allen G

The Role of Altruistic Values in Motivating Underrepresented Minority Students for Biomedicine

利他主义价值观在激励少数族裔学生投身生物医学领域中的作用

Regulation of IFN-γ by IL-13 dictates susceptibility to secondary postinfluenza MRSA pneumonia

IL-13 对 IFN-γ 的调节决定了流感后继发性耐甲氧西林金黄色葡萄球菌肺炎的易感性

CD11c⁺ cells primed with unrelated antigens facilitate an accelerated immune response to influenza virus in mice

用无关抗原预处理的CD11c⁺细胞可促进小鼠对流感病毒的加速免疫应答。

Biomimetic antigenic nanoparticles elicit controlled protective immune response to influenza

仿生抗原纳米颗粒可诱导针对流感的可控保护性免疫反应

Inducible bronchus-associated lymphoid tissue (iBALT) synergizes with local lymph nodes during antiviral CD4+ T cell responses

诱导型支气管相关淋巴组织 (iBALT) 在抗病毒 CD4+ T 细胞反应中与局部淋巴结协同作用

Pneumocystis elicits a STAT6-dependent, strain-specific innate immune response and airway hyperresponsiveness

肺孢子菌可引发STAT6依赖性的、菌株特异性的先天免疫反应和气道高反应性。

Virus-like particle-induced protection against MRSA pneumonia is dependent on IL-13 and enhancement of phagocyte function

病毒样颗粒诱导的针对耐甲氧西林金黄色葡萄球菌肺炎的保护作用依赖于IL-13和吞噬细胞功能的增强。

Resident alveolar macrophages are susceptible to and permissive of Coxiella burnetii infection

肺泡驻留巨噬细胞易受伯氏考克斯体感染，并允许其感染。

Influenza virus infection decreases tracheal mucociliary velocity and clearance of Streptococcus pneumoniae

流感病毒感染会降低气管黏膜纤毛运动速度和肺炎链球菌的清除能力。

Pneumococcal surface protein A contributes to secondary Streptococcus pneumoniae infection after influenza virus infection

肺炎球菌表面蛋白A在流感病毒感染后可导致继发性肺炎链球菌感染。