日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Structural dynamics insights into principles underlying the fitness of new broadly potent AAVs

结构动力学洞见揭示新型广谱高效AAV适应性的潜在原理

Johnson, Molly E; Ozturk, Bilge E; Tugwell, Thomas H; Lambros, Maryl; Janowitz, Haley N; Flohr, Keevon; Sedorovitz, Morgan; Campello, Laura; Hartung, Jane E; Hogle, Brett; Gillespie, Megan; Schriever, Hannah; Aweidah, Hamzah; Koester, Janis; Clausen, Inga; Seeber, Stefan; Revelant, Franco; Schreurs, Richard; Koechl, Fabian; Sieving, Paul A; Sahel, José-Alain; Stauffer, William R; Peixoto, Rui T; Fauser, Sascha; Lin, Rob; Conway, James F; da Silva, Susana; Krol, Jacek; Betegon, Miguel; Byrne, Leah C

Persistent (Na v 1.9) sodium currents in human dorsal root ganglion neurons

人类背根神经节神经元中持续存在的(Na v 1.9)钠电流

Zhang, Xiulin; Hartung, Jane E; Gold, Michael S

Photothermal Excitation of Neurons Using MXene: Cellular Stress and Phototoxicity Evaluation.

利用 MXene 对神经元进行光热激发:细胞应激和光毒性评价

Wang Yingqiao, Hartung Jane E, Goad Adam, Preisegger Matías A, Chacon Benjamin, Gold Michael S, Gogotsi Yury, Cohen-Karni Tzahi

Nicotine Evoked Currents in Human Primary Sensory Neurons

尼古丁诱发人类初级感觉神经元电流

Zhang, Xiulin; Hartung, Jane E; Friedman, Robert L; Koerber, H Richard; Belfer, Inna; Gold, Michael S

Sustained stimulation of β(2)- and β(3)-adrenergic receptors leads to persistent functional pain and neuroinflammation

β(2)-和β(3)-肾上腺素能受体的持续刺激会导致持续性功能性疼痛和神经炎症。

Zhang, Xin; Hartung, Jane E; Bortsov, Andrey V; Kim, Seungtae; O'Buckley, Sandra C; Kozlowski, Julia; Nackley, Andrea G

Circulating Omentin-1 and Chronic Painful Temporomandibular Disorders

循环中的网膜素-1与慢性疼痛性颞下颌关节紊乱

Harmon, Jennifer B; Sanders, Anne E; Wilder, Rebecca S; Essick, Greg K; Slade, Gary D; Hartung, Jane E; Nackley, Andrea G

Nuclear factor-kappa B regulates pain and COMT expression in a rodent model of inflammation

核因子-κB在啮齿动物炎症模型中调节疼痛和COMT表达。

Hartung, Jane E; Eskew, Olivia; Wong, Terrence; Tchivileva, Inna E; Oladosu, Folabomi A; O'Buckley, Sandra C; Nackley, Andrea G

β2- and β3-adrenergic receptors drive COMT-dependent pain by increasing production of nitric oxide and cytokines.

β2-和β3-肾上腺素能受体通过增加一氧化氮和细胞因子的产生来驱动COMT依赖性疼痛

Hartung Jane E, Ciszek Brittney P, Nackley Andrea G