日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Podocyte specific exon skipping after disease onset improves kidney pathology and function in a mouse model of Alport syndrome

在阿尔波特综合征小鼠模型中,疾病发作后足细胞特异性外显子跳跃可改善肾脏病理和功能。

Hashikami, Kentarou; Kobayashi, Ryosuke; Hori, Ryotaro; Danbayashi, Kenta; Nio, Yasunori

Anti-human-TIGIT agonistic antibody ameliorates autoimmune diseases by inhibiting Tfh and Tph cells and enhancing Treg cells

抗人TIGIT激动剂抗体通过抑制Tfh和Tph细胞并增强Treg细胞改善自身免疫性疾病

Marenori Kojima, Katsuya Suzuki, Masaru Takeshita, Masaki Ohyagi, Mana Iizuka, Humitsugu Yamane, Keiko Koga, Taku Kouro, Yoshiaki Kassai, Tomoki Yoshihara, Ryutaro Adachi, Kentarou Hashikami, Yuichiro Ota, Keiko Yoshimoto, Yuko Kaneko, Rimpei Morita, Akihiko Yoshimura, Tsutomu Takeuchi

Sbp2l contributes to oligodendrocyte maturation through translational control in Tcf7l2 signaling

Sbp2l 通过 Tcf7l2 信号传导中的翻译控制促进少突胶质细胞成熟

Masato Yugami, Yoshika Hayakawa-Yano, Takahisa Ogasawara, Kazumasa Yokoyama, Takako Furukawa, Hiroe Hara, Kentaro Hashikami, Isamu Tsuji, Hirohide Takebayashi, Shinsuke Araki, Hideyuki Okano, Masato Yano

Bidirectional, non-necrotizing glomerular crescents are the critical pathology in X-linked Alport syndrome mouse model harboring nonsense mutation of human COL4A5

双向、非坏死性肾小球新月体是携带人类 COL4A5 无义突变的 X 连锁 Alport 综合征小鼠模型的关键病理

Jiang Ying Song, Nobuyuki Saga, Kunio Kawanishi, Kentaro Hashikami, Michiyasu Takeyama, Michio Nagata

Chemically Crosslinked Bispecific Antibodies for Cancer Therapy: Breaking from the Structural Restrictions of the Genetic Fusion Approach

化学交联双特异性抗体用于癌症治疗:突破基因融合方法的结构限制

Asami Ueda, Mitsuo Umetsu, Takeshi Nakanishi, Kentaro Hashikami, Hikaru Nakazawa, Shuhei Hattori, Ryutaro Asano, Izumi Kumagai

Establishment of X-linked Alport syndrome model mice with a Col4a5 R471X mutation.

建立具有 Col4a5 R471X 突变的 X 连锁 Alport 综合征模型小鼠

Hashikami Kentarou, Asahina Makoto, Nozu Kandai, Iijima Kazumoto, Nagata Michio, Takeyama Michiyasu

Phospholamban Ablation Using CRISPR/Cas9 System Improves Mortality in a Murine Heart Failure Model

使用 CRISPR/Cas9 系统进行磷脂酰受精蛋白消融可改善小鼠心力衰竭模型的死亡率

Manami Kaneko, Kentarou Hashikami, Satoshi Yamamoto, Hirokazu Matsumoto, Tomoyuki Nishimoto