日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Inhibition of NAD-GPx4 axis and MEK triggers ferroptosis to suppress pancreatic ductal adenocarcinoma.

抑制 NAD-GPx4 轴和 MEK 可触发铁死亡,从而抑制胰腺导管腺癌

Jiang Hui, Satoh Yusuke, Yamamura Ryodai, Ooshio Takako, Luo Yang, Hai Han, Otsuka Takuya, Hata Soichiro, Sato Reo, Hirata Taiga, Osawa Tsuyoshi, Goda Keisuke, Sonoshita Masahiro

Reduction in Acetylation of Superoxide Dismutase 2 in Skeletal Muscle Improves Exercise Capacity in Mice With Heart Failure

降低骨骼肌中超氧化物歧化酶2的乙酰化水平可改善心力衰竭小鼠的运动能力

Masunaga, Tomoka; Suenaga, Tomoyasu; Matsushima, Shouji; Hashimoto, Toru; Takada, Shingo; Noda, Eri; Fumoto, Yoshizuki; Hata, Soichiro; Yokota, Takashi; Kinugawa, Shintaro

O-GlcNAcase Inhibitor Improves Denervation-Induced Muscle Atrophy in Mice

O-GlcNAcase抑制剂可改善小鼠去神经支配引起的肌肉萎缩

Suenaga, Tomoyasu; Matsushima, Shouji; Masunaga, Tomoka; Hashimoto, Toru; Takada, Shingo; Fumoto, Yoshizuki; Hata, Soichiro; Abe, Kohtaro; Kinugawa, Shintaro

Orchestration of mesenchymal plasticity and immune evasiveness via rewiring of the metabolic program in pancreatic ductal adenocarcinoma

胰腺导管腺癌中通过代谢程序重编程来调控间质可塑性和免疫逃逸

Hashimoto, Ari; Handa, Haruka; Hata, Soichiro; Hashimoto, Shigeru

Inhibition of mutant KRAS-driven overexpression of ARF6 and MYC by an eIF4A inhibitor drug improves the effects of anti-PD-1 immunotherapy for pancreatic cancer

利用eIF4A抑制剂药物抑制突变型KRAS驱动的ARF6和MYC过表达,可增强抗PD-1免疫疗法治疗胰腺癌的效果。

Hashimoto, Ari; Handa, Haruka; Hata, Soichiro; Tsutaho, Akio; Yoshida, Takao; Hirano, Satoshi; Hashimoto, Shigeru; Sabe, Hisataka

ARF6 and AMAP1 are major targets of KRAS and TP53 mutations to promote invasion, PD-L1 dynamics, and immune evasion of pancreatic cancer

ARF6 和 AMAP1 是 KRAS 和 TP53 突变的主要靶点,这些突变会促进胰腺癌的侵袭、PD-L1 动态变化和免疫逃逸。

Hashimoto, Shigeru; Furukawa, Shotaro; Hashimoto, Ari; Tsutaho, Akio; Fukao, Akira; Sakamura, Yurika; Parajuli, Gyanu; Onodera, Yasuhito; Otsuka, Yutaro; Handa, Haruka; Oikawa, Tsukasa; Hata, Soichiro; Nishikawa, Yoshihiro; Mizukami, Yusuke; Kodama, Yuzo; Murakami, Masaaki; Fujiwara, Toshinobu; Hirano, Satoshi; Sabe, Hisataka