Hayden Matthew S相关文献与解析，生知库 | 链接生命科学的每一步

Intraoperative margin assessment for basal cell carcinoma with deep learning and histologic tumor mapping to surgical site

利用深度学习和组织学肿瘤定位技术对基底细胞癌进行术中切缘评估

Levy, Joshua J; Davis, Matthew J; Chacko, Rachael S; Davis, Michael J; Fu, Lucy J; Goel, Tarushii; Pamal, Akash; Nafi, Irfan; Angirekula, Abhinav; Suvarna, Anish; Vempati, Ram; Christensen, Brock C; Hayden, Matthew S; Vaickus, Louis J; LeBoeuf, Matthew R

细胞生物学

PDK1 Is Required for Maintenance of CD4(+) Foxp3(+) Regulatory T Cell Function

PDK1是维持CD4(+)Foxp3(+)调节性T细胞功能所必需的

期刊:Journal of Immunology

影响因子:3.4

doi:10.4049/jimmunol.2000051

Oh, Hyunju; Zhao, Jingyao; Grinberg-Bleyer, Yenkel; Postler, Thomas S; Wang, Pingzhang; Park, Sung-Gyoo; Rabadan, Raul; Hayden, Matthew S; Ghosh, Sankar

Gene editing in dermatology: Harnessing CRISPR for the treatment of cutaneous disease

皮肤病学中的基因编辑：利用 CRISPR 技术治疗皮肤疾病

期刊:F1000Research

影响因子:

doi:10.12688/f1000research.23185.2

Baker, Catherine; Hayden, Matthew S

发表日期：2020

文献求助收藏

The Alternative NF-ÎºB Pathway in Regulatory T Cell Homeostasis and Suppressive Function.

调节性 T 细胞稳态和抑制功能中的替代 NF-κB 通路

期刊:Journal of Immunology

影响因子:3.4

doi:10.4049/jimmunol.1800042

Grinberg-Bleyer Yenkel, Caron Rachel, Seeley John J, De Silva Nilushi S, Schindler Christian W, Hayden Matthew S, Klein Ulf, Ghosh Sankar

Cutting edge: NF-κB p65 and c-Rel control epidermal development and immune homeostasis in the skin

前沿研究：NF-κB p65 和 c-Rel 控制皮肤表皮发育和免疫稳态

期刊:Journal of Immunology

影响因子:3.4

doi:10.4049/jimmunol.1402608

Grinberg-Bleyer, Yenkel; Dainichi, Teruki; Oh, Hyunju; Heise, Nicole; Klein, Ulf; Schmid, Roland M; Hayden, Matthew S; Ghosh, Sankar

Regulation of NF-κB by TNF family cytokines

TNF家族细胞因子对NF-κB的调控

期刊:Seminars in Immunology

影响因子:7.8

doi:10.1016/j.smim.2014.05.004

Hayden, Matthew S; Ghosh, Sankar

NF-κB

发表日期：2014

文献求助收藏

Transition from heterotypic to homotypic PDK1 homodimerization is essential for TCR-mediated NF-κB activation

PDK1同源二聚体从异型向同型转变是TCR介导的NF-κB激活所必需的。

期刊:Journal of Immunology

影响因子:3.4

doi:10.4049/jimmunol.1202923

Kang, Jung-Ah; Jeong, Sang Phil; Park, Daeho; Hayden, Matthew S; Ghosh, Sankar; Park, Sung-Gyoo

The kinase PDK1 is essential for B-cell receptor mediated survival signaling

激酶PDK1对于B细胞受体介导的存活信号传导至关重要。

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0055378

Park, Sung-Gyoo; Long, Meixiao; Kang, Jung-Ah; Kim, Woo-Seok; Lee, Cho-Rong; Im, Sin-Hyeog; Strickland, Ian; Schulze-Luehrmann, Jan; Hayden, Matthew S; Ghosh, Sankar

A role for NF-κB activity in skin hyperplasia and the development of keratoacanthomata in mice

NF-κB活性在小鼠皮肤增生和角化棘皮瘤发生发展中的作用

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0071887

Poligone, Brian; Hayden, Matthew S; Chen, Luojing; Pentland, Alice P; Jimi, Eijiro; Ghosh, Sankar

NF-κB

发表日期：2013

文献求助收藏

A CRISPR/LbCas12a system for Borrelia burgdorferi sensu stricto detection in blacklegged ticks

用于检测黑腿蜱中伯氏疏螺旋体（Borrelia burgdorferi sensu stricto）的CRISPR/LbCas12a系统

Intraoperative margin assessment for basal cell carcinoma with deep learning and histologic tumor mapping to surgical site

利用深度学习和组织学肿瘤定位技术对基底细胞癌进行术中切缘评估

PDK1 Is Required for Maintenance of CD4(+) Foxp3(+) Regulatory T Cell Function

PDK1是维持CD4(+)Foxp3(+)调节性T细胞功能所必需的

Gene editing in dermatology: Harnessing CRISPR for the treatment of cutaneous disease

皮肤病学中的基因编辑：利用 CRISPR 技术治疗皮肤疾病

The Alternative NF-ÎºB Pathway in Regulatory T Cell Homeostasis and Suppressive Function.

调节性 T 细胞稳态和抑制功能中的替代 NF-κB 通路

Cutting edge: NF-κB p65 and c-Rel control epidermal development and immune homeostasis in the skin

前沿研究：NF-κB p65 和 c-Rel 控制皮肤表皮发育和免疫稳态

Regulation of NF-κB by TNF family cytokines

TNF家族细胞因子对NF-κB的调控

Transition from heterotypic to homotypic PDK1 homodimerization is essential for TCR-mediated NF-κB activation

PDK1同源二聚体从异型向同型转变是TCR介导的NF-κB激活所必需的。

The kinase PDK1 is essential for B-cell receptor mediated survival signaling

激酶PDK1对于B细胞受体介导的存活信号传导至关重要。

A role for NF-κB activity in skin hyperplasia and the development of keratoacanthomata in mice

NF-κB活性在小鼠皮肤增生和角化棘皮瘤发生发展中的作用