Hedl Matija相关文献与解析，生知库 | 链接生命科学的每一步

IRF5 Is Required for Bacterial Clearance in Human M1-Polarized Macrophages, and IRF5 Immune-Mediated Disease Risk Variants Modulate This Outcome

IRF5是人类M1型极化巨噬细胞清除细菌所必需的，而IRF5免疫介导疾病风险变异体调节这一结果。

期刊:Journal of Immunology

影响因子:3.4

doi:10.4049/jimmunol.1800226

Hedl, Matija; Yan, Jie; Witt, Heiko; Abraham, Clara

Human

发表日期：2019

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MTMR3 risk allele enhances innate receptor-induced signaling and cytokines by decreasing autophagy and increasing caspase-1 activation

MTMR3风险等位基因通过降低自噬和增加caspase-1活化来增强先天性受体诱导的信号传导和细胞因子。

期刊:Proceedings of the National Academy of Sciences of the United States of America

影响因子:9.1

doi:10.1073/pnas.1501752112

Lahiri, Amit; Hedl, Matija; Abraham, Clara

Pattern recognition receptor signaling in human dendritic cells is enhanced by ICOS ligand and modulated by the Crohn's disease ICOSLG risk allele.

人类树突状细胞中的模式识别受体信号传导受到 ICOS 配体的增强，并受到克罗恩病 ICOSLG 风险等位基因的调节

期刊:Immunity

影响因子:26.3

doi:10.1016/j.immuni.2014.04.011

Hedl Matija, Lahiri Amit, Ning Kaida, Cho Judy H, Abraham Clara

IL-22BP is regulated by the inflammasome and modulates tumorigenesis in the intestine

IL-22BP受炎症小体调控，并调节肠道肿瘤的发生。

期刊:Nature

影响因子:48.5

doi:10.1038/nature11535

Huber, Samuel; Gagliani, Nicola; Zenewicz, Lauren A; Huber, Francis J; Bosurgi, Lidia; Hu, Bo; Hedl, Matija; Zhang, Wei; O'Connor, William Jr; Murphy, Andrew J; Valenzuela, David M; Yancopoulos, George D; Booth, Carmen J; Cho, Judy H; Ouyang, Wenjun; Abraham, Clara; Flavell, Richard A

Nod2-induced autocrine interleukin-1 alters signaling by ERK and p38 to differentially regulate secretion of inflammatory cytokines

Nod2诱导的自分泌白细胞介素-1通过改变ERK和p38信号通路来差异性地调节炎症细胞因子的分泌

期刊:Gastroenterology

影响因子:25.1

doi:10.1053/j.gastro.2012.08.048

Hedl, Matija; Abraham, Clara

IRF5 risk polymorphisms contribute to interindividual variance in pattern recognition receptor-mediated cytokine secretion in human monocyte-derived cells.

IRF5 风险多态性导致人类单核细胞衍生细胞中模式识别受体介导的细胞因子分泌存在个体差异

期刊:Journal of Immunology

影响因子:3.4

doi:10.4049/jimmunol.1103319

Hedl Matija, Abraham Clara

Secretory mediators regulate Nod2-induced tolerance in human macrophages

分泌介质调节人巨噬细胞中Nod2诱导的耐受性

期刊:Gastroenterology

影响因子:25.1

doi:10.1053/j.gastro.2010.09.009

Hedl, Matija; Abraham, Clara

Inhibition of the class II HMG-CoA reductase of Pseudomonas mevalonii

抑制假单胞菌II类HMG-CoA还原酶

期刊:Protein Science

影响因子:5.2

doi:10.1110/ps.03597504

Hedl, Matija; Rodwell, Victor W

发表日期：2004

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The E3 ubiquitin ligase RNF186 and RNF186 risk variants regulate innate receptor-induced outcomes

E3泛素连接酶RNF186及其风险变异体调控先天性受体诱导的结果

IRF5 Is Required for Bacterial Clearance in Human M1-Polarized Macrophages, and IRF5 Immune-Mediated Disease Risk Variants Modulate This Outcome

IRF5是人类M1型极化巨噬细胞清除细菌所必需的，而IRF5免疫介导疾病风险变异体调节这一结果。

MTMR3 risk allele enhances innate receptor-induced signaling and cytokines by decreasing autophagy and increasing caspase-1 activation

MTMR3风险等位基因通过降低自噬和增加caspase-1活化来增强先天性受体诱导的信号传导和细胞因子。

Pattern recognition receptor signaling in human dendritic cells is enhanced by ICOS ligand and modulated by the Crohn's disease ICOSLG risk allele.

人类树突状细胞中的模式识别受体信号传导受到 ICOS 配体的增强，并受到克罗恩病 ICOSLG 风险等位基因的调节

IL-22BP is regulated by the inflammasome and modulates tumorigenesis in the intestine

IL-22BP受炎症小体调控，并调节肠道肿瘤的发生。

Nod2-induced autocrine interleukin-1 alters signaling by ERK and p38 to differentially regulate secretion of inflammatory cytokines

Nod2诱导的自分泌白细胞介素-1通过改变ERK和p38信号通路来差异性地调节炎症细胞因子的分泌

IRF5 risk polymorphisms contribute to interindividual variance in pattern recognition receptor-mediated cytokine secretion in human monocyte-derived cells.

IRF5 风险多态性导致人类单核细胞衍生细胞中模式识别受体介导的细胞因子分泌存在个体差异

Secretory mediators regulate Nod2-induced tolerance in human macrophages

分泌介质调节人巨噬细胞中Nod2诱导的耐受性

Inhibition of the class II HMG-CoA reductase of Pseudomonas mevalonii

抑制假单胞菌II类HMG-CoA还原酶