日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Arrhythmogenic calmodulin variants D131E and Q135P disrupt interaction with the L-type voltage-gated Ca(2+) channel (Ca(v)1.2) and reduce Ca(2+)-dependent inactivation

致心律失常的钙调蛋白变体D131E和Q135P破坏了与L型电压门控Ca(2+)通道(Ca(v)1.2)的相互作用,并降低了Ca(2+)依赖性失活。

Gupta, Nitika; Richards, Ella M B; Morris, Vanessa S; Morris, Rachael; Wadmore, Kirsty; Held, Marie; McCormick, Liam; Prakash, Ohm; Dart, Caroline; Helassa, Nordine

Structure-Function Diversity of Calcium-Binding Proteins (CaBPs): Key Roles in Cell Signalling and Disease

钙结合蛋白(CaBPs)的结构-功能多样性:在细胞信号传导和疾病中的关键作用

Morris, Vanessa S; Richards, Ella M B; Morris, Rachael; Dart, Caroline; Helassa, Nordine

Calmodulin D133H Disrupts Ca(v)1.2 and K(v)7.1 Regulation to Prolong Cardiac Action Potentials in Long QT Syndrome

钙调蛋白D133H破坏Ca(v)1.2和K(v)7.1的调节,从而延长长QT综合征患者的心肌动作电位

Gupta, Nitika; McCormick, Liam F; Richards, Ella M B; Wadmore, Kirsty; Morris, Rachael; Morris, Vanessa S; Kirilenko, Pavel; Fowler, Ewan D; Dart, Caroline; Helassa, Nordine

Proximity labelling reveals effects of disease-causing mutation on the DNAJC5/cysteine string protein α interactome

邻近标记揭示致病突变对 DNAJC5/半胱氨酸串蛋白 α 相互作用组的影响

Eleanor Barker, Amy Milburn, Nordine Helassa, Dean Hammond, Natalia Sanchez-Soriano, Alan Morgan, Jeff Barclay

Author Correction: Highly specific and non-invasive imaging of Piezo1-dependent activity across scales using GenEPi

作者更正:利用 GenEPi 对 Piezo1 依赖性活性进行跨尺度的高特异性、非侵入性成像

Yaganoglu, Sine; Kalyviotis, Konstantinos; Vagena-Pantoula, Christina; Jülich, Dörthe; Gaub, Benjamin M; Welling, Maaike; Lopes, Tatiana; Lachowski, Dariusz; Tang, See Swee; Del Rio Hernandez, Armando; Salem, Victoria; Müller, Daniel J; Holley, Scott A; Vermot, Julien; Shi, Jian; Helassa, Nordine; Török, Katalin; Pantazis, Periklis

Highly specific and non-invasive imaging of Piezo1-dependent activity across scales using GenEPi

利用 GenEPi 对不同尺度下 Piezo1 依赖性活性进行高度特异性和非侵入性成像

Sine Yaganoglu # ,Konstantinos Kalyviotis # ,Christina Vagena-Pantoula ,Dörthe Jülich ,Benjamin M Gaub ,Maaike Welling ,Tatiana Lopes ,Dariusz Lachowski ,See Swee Tang ,Armando Del Rio Hernandez ,Victoria Salem ,Daniel J Müller ,Scott A Holley ,Julien Vermot ,Jian Shi ,Nordine Helassa ,Katalin Török ,Periklis Pantazis

Long QT syndrome-associated calmodulin variants disrupt the activity of the slowly activating delayed rectifier potassium channel

长QT综合征相关的钙调蛋白变异体破坏了慢激活延迟整流钾通道的活性。

McCormick, Liam; Wadmore, Kirsty; Milburn, Amy; Gupta, Nitika; Morris, Rachael; Held, Marie; Prakash, Ohm; Carr, Joseph; Barrett-Jolley, Richard; Dart, Caroline; Helassa, Nordine

Calmodulin variant E140G associated with long QT syndrome impairs CaMKIIδ autophosphorylation and L-type calcium channel inactivation

与长 QT 综合征相关的钙调蛋白变体 E140G 会损害 CaMKIIδ 自身磷酸化和 L 型钙通道失活

Ohm Prakash, Nitika Gupta, Amy Milburn, Liam McCormick, Vishvangi Deugi, Pauline Fisch, Jacob Wyles, N Lowri Thomas, Svetlana Antonyuk, Caroline Dart, Nordine Helassa

Editorial: The role of calcium and calcium binding proteins in cell physiology and disease

社论:钙和钙结合蛋白在细胞生理和疾病中的作用

Thomas, N Lowri; Dart, C; Helassa, N

Vesicular release probability sets the strength of individual Schaffer collateral synapses.

囊泡释放概率决定了单个 Schaffer 侧支突触的强度

Dürst Céline D, Wiegert J Simon, Schulze Christian, Helassa Nordine, Török Katalin, Oertner Thomas G