Helble Jennifer相关文献与解析，生知库 | 链接生命科学的每一步

Borrelia burgdorferi initiates early transcriptional re-programming in macrophages that supports long-term suppression of inflammation

伯氏疏螺旋体启动巨噬细胞早期转录重编程，从而支持炎症的长期抑制。

期刊:PLoS Pathogens

影响因子:4.9

doi:10.1371/journal.ppat.1011886

Petnicki-Ocwieja, Tanja; McCarthy, Julie E; Powale, Urmila; Langston, P Kent; Helble, Jennifer D; Hu, Linden T

The PD-1/PD-L1 pathway is induced during Borrelia burgdorferi infection and inhibits T cell joint infiltration without compromising bacterial clearance

PD-1/PD-L1通路在伯氏疏螺旋体感染期间被激活，并抑制T细胞关节浸润，但不影响细菌清除。

期刊:PLoS Pathogens

影响因子:4.9

doi:10.1371/journal.ppat.1010903

Helble, Jennifer D; McCarthy, Julie E; Sawden, Machlan; Starnbach, Michael N; Hu, Linden T

T cell responses to Chlamydia

衣原体引起的T细胞反应

期刊:Pathogens and Disease

影响因子:2.7

doi:10.1093/femspd/ftab014

Helble, Jennifer D; Starnbach, Michael N

Interactions between Borrelia burgdorferi and its hosts across the enzootic cycle

在地方性流行周期中，伯氏疏螺旋体与其宿主之间的相互作用

期刊:Parasite Immunology

影响因子:2.1

doi:10.1111/pim.12816

Helble, Jennifer D; McCarthy, Julie E; Hu, Linden T

发表日期：2021

文献求助收藏

Antigen-specific memory and naïve CD4+ T cells following secondary Chlamydia trachomatis infection

继发性沙眼衣原体感染后的抗原特异性记忆和初始CD4+ T细胞

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0240670

Helble, Jennifer D; Mann, Alexander O; Starnbach, Michael N

A FACS-Based Genome-wide CRISPR Screen Reveals a Requirement for COPI in Chlamydia trachomatis Invasion

基于FACS的全基因组CRISPR筛选揭示了COPI在沙眼衣原体入侵中的必要性

期刊:iScience

影响因子:4.1

doi:10.1016/j.isci.2018.12.011

Park, Joseph S; Helble, Jennifer D; Lazarus, Jacob E; Yang, Guanhua; Blondel, Carlos J; Doench, John G; Starnbach, Michael N; Waldor, Matthew K

发表日期：2019

文献求助收藏

Early Colonization of the Upper Genital Tract by Chlamydia muridarum Is Associated with Enhanced Inflammation Later in Infection

鼠衣原体早期定植于上生殖道与感染后期炎症加剧有关

期刊:Infection and Immunity

影响因子:2.8

doi:10.1128/IAI.00405-19

Helble, Jennifer D; Reinhold-Larsson, Nicole V; Starnbach, Michael N

炎症/感染

Immunology/Inflammation

Single-cell cloning of human T-cell lines reveals clonal variation in cell death responses to chemotherapeutics.

人类 T 细胞系的单细胞克隆揭示了细胞对化疗药物死亡反应的克隆变异

期刊:Cancer Genetics

影响因子:2.1

doi:10.1016/j.cancergen.2019.06.003

Hanlon Kathleen, Thompson Alex, Pantano Lorena, Hutchinson John N, Al-Obeidi Arshed, Wang Shu, Bliss-Moreau Meghan, Helble Jennifer, Alexe Gabriela, Stegmaier Kimberly, Bauer Daniel E, Croker Ben A

Pathology after Chlamydia trachomatis infection is driven by nonprotective immune cells that are distinct from protective populations

沙眼衣原体感染后的病理变化是由非保护性免疫细胞驱动的，这些细胞与保护性免疫细胞群截然不同。

期刊:Proceedings of the National Academy of Sciences of the United States of America

影响因子:

doi:10.1073/pnas.1711356115

Lijek, Rebeccah S; Helble, Jennifer D; Olive, Andrew J; Seiger, Kyra W; Starnbach, Michael N

Cytotoxic CD4+ T Cells Are Induced during Infection with Chlamydia trachomatis

沙眼衣原体感染期间会诱导细胞毒性 CD4+ T 细胞的产生

Borrelia burgdorferi initiates early transcriptional re-programming in macrophages that supports long-term suppression of inflammation

伯氏疏螺旋体启动巨噬细胞早期转录重编程，从而支持炎症的长期抑制。

The PD-1/PD-L1 pathway is induced during Borrelia burgdorferi infection and inhibits T cell joint infiltration without compromising bacterial clearance

PD-1/PD-L1通路在伯氏疏螺旋体感染期间被激活，并抑制T细胞关节浸润，但不影响细菌清除。

T cell responses to Chlamydia

衣原体引起的T细胞反应

Interactions between Borrelia burgdorferi and its hosts across the enzootic cycle

在地方性流行周期中，伯氏疏螺旋体与其宿主之间的相互作用

Antigen-specific memory and naïve CD4+ T cells following secondary Chlamydia trachomatis infection

继发性沙眼衣原体感染后的抗原特异性记忆和初始CD4+ T细胞

A FACS-Based Genome-wide CRISPR Screen Reveals a Requirement for COPI in Chlamydia trachomatis Invasion

基于FACS的全基因组CRISPR筛选揭示了COPI在沙眼衣原体入侵中的必要性

Early Colonization of the Upper Genital Tract by Chlamydia muridarum Is Associated with Enhanced Inflammation Later in Infection

鼠衣原体早期定植于上生殖道与感染后期炎症加剧有关

Single-cell cloning of human T-cell lines reveals clonal variation in cell death responses to chemotherapeutics.

人类 T 细胞系的单细胞克隆揭示了细胞对化疗药物死亡反应的克隆变异

Pathology after Chlamydia trachomatis infection is driven by nonprotective immune cells that are distinct from protective populations

沙眼衣原体感染后的病理变化是由非保护性免疫细胞驱动的，这些细胞与保护性免疫细胞群截然不同。