Calmodulin/CaMKII inhibition improves intercellular communication and impulse propagation in the heart and is antiarrhythmic under conditions when fibrosis is absent
钙调蛋白/CaMKII 抑制可改善心脏细胞间通讯和脉冲传播，并且在没有纤维化的情况下具有抗心律失常作用
期刊:Cardiovascular Research
影响因子:10.2
doi:10.1093/cvr/cvw173
Hiroki Takanari, Vincent J A Bourgonje, Magda S C Fontes, Antonia J A Raaijmakers, Helen Driessen, John A Jansen, Roel van der Nagel, Bart Kok, Leonie van Stuijvenberg, Mohamed Boulaksil, Yoshio Takemoto, Masatoshi Yamazaki, Yukiomi Tsuji, Haruo Honjo, Kaichiro Kamiya, Itsuo Kodama, Mark E Anderson,
