Cardiac-restricted overexpression of the A(2A)-adenosine receptor in FVB mice transiently increases contractile performance and rescues the heart failure phenotype in mice overexpressing the A(1)-adenosine receptor.
在 FVB 小鼠中，心脏特异性过表达 A(2A)-腺苷受体可暂时提高收缩性能，并挽救过表达 A(1)-腺苷受体的小鼠的心力衰竭表型
期刊:Cts-Clinical and Translational Science
影响因子:2.8
doi:10.1111/j.1752-8062.2008.00027.x
Chan Tung O, Funakoshi Hajime, Song Jianliang, Zhang Xue-Qian, Wang JuFang, Chung Paul H, DeGeorge Brent R Jr, Li Xue, Zhang Jin, Herrmann David E, Diamond Maura, Hamad Eman, Houser Steven R, Koch Walter J, Cheung Joseph Y, Feldman Arthur M
