Horstkorte相关文献与解析，生知库 | 链接生命科学的每一步

Induced Muscle and Liver Absence of Gne in Postnatal Mice Does Not Result in Structural or Functional Muscle Impairment

出生后小鼠诱导的肌肉和肝脏缺乏 Gne 不会导致结构性或功能性肌肉损伤

Avi Harazi, Lena Yakovlev, Nili Ilouz, Philipp Selke, Rudiger Horstkorte, Yakov Fellig, Olga Lahat, Tzuri Lifschytz, Nathalie Abudi, Rinat Abramovitch, Zohar Argov, Stella Mitrani-Rosenbaum

信号转导

Glycation Leads to Increased Invasion of Glioblastoma Cells

糖化导致胶质母细胞瘤细胞侵袭性增强

期刊:Cells

影响因子:5.1

doi:10.3390/cells12091219

Paola Schildhauer, Philipp Selke, Christian Scheller, Christian Strauss, Rüdiger Horstkorte, Sandra Leisz, Maximilian Scheer

Glycation Interferes with the Expression of Sialyltransferases and Leads to Increased Polysialylation in Glioblastoma Cells

糖化干扰唾液酸转移酶的表达并导致胶质母细胞瘤细胞中的聚唾液酸化增加

期刊:Cells

影响因子:5.1

doi:10.3390/cells12232758

Paola Schildhauer, Philipp Selke, Martin S Staege, Anja Harder, Christian Scheller, Christian Strauss, Rüdiger Horstkorte, Maximilian Scheer, Sandra Leisz

Glycation Interferes with the Activity of the Bi-Functional UDP- N-Acetylglucosamine 2-Epimerase/ N-Acetyl-mannosamine Kinase (GNE)

糖化作用干扰双功能UDP-N-乙酰氨基葡萄糖2-差向异构酶/N-乙酰甘露糖胺激酶（GNE）的活性

期刊:Biomolecules

影响因子:4.8

doi:10.3390/biom13030422

Vanessa Hagenhaus,Jacob L Gorenflos López,Rebecca Rosenstengel,Carolin Neu,Christian P R Hackenberger,Arif Celik,Klara Weinert,Mai-Binh Nguyen,Kaya Bork,Rüdiger Horstkorte,Astrid Gesper

信号转导

发表日期：2023

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No Impact of PolySia-NCAM Expression on Treatment Response in Neuroendocrine Neoplasms of the Lung

PolySia-NCAM表达对肺神经内分泌肿瘤治疗反应无影响

期刊:Cancers

影响因子:4.4

doi:10.3390/cancers14184376

Gagiannis, Daniel; Scheil, Anna; Gagiannis, Sarah; Hackenbroch, Carsten; Horstkorte, Ruediger; Steinestel, Konrad

Disturbance of Key Cellular Subproteomes upon Propofol Treatment Is Associated with Increased Permeability of the Blood-Brain Barrier

丙泊酚治疗后关键细胞亚蛋白质组的紊乱与血脑屏障通透性增加有关

期刊:Proteomes

影响因子:4

doi:10.3390/proteomes10030028

Timo Längrich, Kaya Bork, Rüdiger Horstkorte, Veronika Weber, Britt Hofmann, Matt Fuszard, Heidi Olzscha

GMPPA defects cause a neuromuscular disorder with α-dystroglycan hyperglycosylation

GMPPA 缺陷会导致 α-肌营养不良症高糖基化引起的神经肌肉疾病

期刊:Journal of Clinical Investigation

影响因子:13.3

doi:10.1172/JCI139076

Patricia Franzka, Henriette Henze, M Juliane Jung, Svenja Caren Schüler, Sonnhild Mittag, Karina Biskup, Lutz Liebmann, Takfarinas Kentache, José Morales, Braulio Martínez, Istvan Katona, Tanja Herrmann, Antje-Kathrin Huebner, J Christopher Hennings, Susann Groth, Lennart Gresing, Rüdiger Horstkorte

神经

发表日期：2021

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Glycation Leads to Increased Polysialylation and Promotes the Metastatic Potential of Neuroblastoma Cells

糖化导致多聚唾液酸化增加并促进神经母细胞瘤细胞的转移潜能

期刊:Cells

影响因子:5.1

doi:10.3390/cells9040868

Maximilian Scheer, Kaya Bork, Frieder Simon, Manimozhi Nagasundaram, Rüdiger Horstkorte, Vinayaga Srinivasan Gnanapragassam

Gne-Depletion in C2C12 Myoblasts Leads to Alterations in Glycosylation and Myopathogene Expression.

C2C12 成肌细胞中 Gne 的缺失导致糖基化和肌病基因表达的改变。

GNE deficiency impairs Myogenesis in C2C12 cells and cannot be rescued by ManNAc supplementation

GNE 缺乏会损害 C2C12 细胞的肌肉生成，并且无法通过补充 ManNAc 来挽救

Induced Muscle and Liver Absence of Gne in Postnatal Mice Does Not Result in Structural or Functional Muscle Impairment

出生后小鼠诱导的肌肉和肝脏缺乏 Gne 不会导致结构性或功能性肌肉损伤

Glycation Leads to Increased Invasion of Glioblastoma Cells

糖化导致胶质母细胞瘤细胞侵袭性增强

Glycation Interferes with the Expression of Sialyltransferases and Leads to Increased Polysialylation in Glioblastoma Cells

糖化干扰唾液酸转移酶的表达并导致胶质母细胞瘤细胞中的聚唾液酸化增加

Glycation Interferes with the Activity of the Bi-Functional UDP- N-Acetylglucosamine 2-Epimerase/ N-Acetyl-mannosamine Kinase (GNE)

糖化作用干扰双功能UDP-N-乙酰氨基葡萄糖2-差向异构酶/N-乙酰甘露糖胺激酶（GNE）的活性

No Impact of PolySia-NCAM Expression on Treatment Response in Neuroendocrine Neoplasms of the Lung

PolySia-NCAM表达对肺神经内分泌肿瘤治疗反应无影响

Disturbance of Key Cellular Subproteomes upon Propofol Treatment Is Associated with Increased Permeability of the Blood-Brain Barrier

丙泊酚治疗后关键细胞亚蛋白质组的紊乱与血脑屏障通透性增加有关

GMPPA defects cause a neuromuscular disorder with α-dystroglycan hyperglycosylation

GMPPA 缺陷会导致 α-肌营养不良症高糖基化引起的神经肌肉疾病

Glycation Leads to Increased Polysialylation and Promotes the Metastatic Potential of Neuroblastoma Cells

糖化导致多聚唾液酸化增加并促进神经母细胞瘤细胞的转移潜能