Permanent exposure of mucin-secreting HT-29 cells to benzyl-N-acetyl-alpha-D-galactosaminide induces abnormal O-glycosylation of mucins and inhibits constitutive and stimulated MUC5AC secretion
持续将分泌粘蛋白的HT-29细胞暴露于苄基-N-乙酰基-α-D-半乳糖胺苷可诱导粘蛋白异常的O-糖基化，并抑制组成型和刺激型MUC5AC分泌。
期刊:Biochemical Journal
影响因子:4.3
doi:10.1042/bj3340283
Hennebicq-Reig, S; Lesuffleur, T; Capon, C; De Bolos, C; Kim, I; Moreau, O; Richet, C; Hémon, B; Recchi, M A; Maës, E; Aubert, J P; Real, F X; Zweibaum, A; Delannoy, P; Degand, P; Huet, G
