日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Macrophage ITGAV is dispensable for post-infarction remodeling in mice and does not mediate fibronectin responses.

巨噬细胞 ITGAV 对小鼠梗死后重塑并非必需,也不介导纤连蛋白反应。

Li Ruoshui, Huang Shuaibo, Hanna Anis, Hernandez Silvia C, Kubota Akihiko, Humeres Claudio, Tuleta Izabela, Frangogiannis Nikolaos G

Reformulation of the Necroptosis Pathway in Reperfused Myocardial Infarction

再灌注心肌梗死中坏死性凋亡途径的重新构建

Qin, Dongze; Modanwal, Radheshyam; Ghazi, Madeeha; Corbalan, J Jose; Jia, Xiaotong F; Axelrod, Joshua L; Mesidor, Roneldine; Jang, Hee Jae; Humeres, Claudio; Hanna, Anis; Varzideh, Fahimeh; Tallquist, Michelle D; Santulli, Gaetano; Liu, Qinghang; Kirshenbaum, Lorrie A; Ni, Hongmin; Kamp, Timothy J; Gundry, Rebekah L; Balachandran, Siddharth; Degterev, Alexei; Calvert, John W; Kitsis, Richard N

Fibroblast-specific TGF-β signaling mediates cardiac dysfunction, fibrosis, and hypertrophy in obese diabetic mice.

成纤维细胞特异性 TGF-β 信号传导介导肥胖糖尿病小鼠的心脏功能障碍、纤维化和肥大

Tuleta Izabela, Hanna Anis, Humeres Claudio, Aguilan Jennifer T, Sidoli Simone, Zhu Fenglan, Frangogiannis Nikolaos G

Fibroblast Smad7 Induction Protects the Remodeling Pressure-Overloaded Heart

成纤维细胞Smad7诱导可保护重塑期压力负荷过重的心脏

Humeres, Claudio; Shinde, Arti V; Tuleta, Izabela; Hernandez, Silvia C; Hanna, Anis; Huang, Shuaibo; Venugopal, Harikrishnan; Aguilan, Jennifer T; Conway, Simon J; Sidoli, Simone; Frangogiannis, Nikolaos G

Macrophages in the infarcted heart acquire a fibrogenic phenotype, expressing matricellular proteins, but do not undergo fibroblast conversion

梗死心脏中的巨噬细胞获得纤维化表型,表达细胞外基质蛋白,但不会转化为成纤维细胞。

Li, Ruoshui; Hanna, Anis; Huang, Shuaibo; Hernandez, Silvia C; Tuleta, Izabela; Kubota, Akihiko; Humeres, Claudio; Chen, Bijun; Liu, Yang; Zheng, Deyou; Frangogiannis, Nikolaos G

Properties and Functions of Fibroblasts and Myofibroblasts in Myocardial Infarction

心肌梗死中成纤维细胞和肌成纤维细胞的特性和功能

Venugopal, Harikrishnan; Hanna, Anis; Humeres, Claudio; Frangogiannis, Nikolaos G

The role of Smad signaling cascades in cardiac fibrosis

Smad信号通路在心脏纤维化中的作用

Hanna, Anis; Humeres, Claudio; Frangogiannis, Nikolaos G

Distinct roles of myofibroblast-specific Smad2 and Smad3 signaling in repair and remodeling of the infarcted heart.

肌成纤维细胞特异性 Smad2 和 Smad3 信号在梗死心脏的修复和重塑中发挥着不同的作用

Huang Shuaibo, Chen Bijun, Su Ya, Alex Linda, Humeres Claudio, Shinde Arti V, Conway Simon J, Frangogiannis Nikolaos G

Myocardial Galectin-3 Expression Is Associated with Remodeling of the Pressure-Overloaded Heart and May Delay the Hypertrophic Response without Affecting Survival, Dysfunction, and Cardiac Fibrosis

心肌半乳糖凝集素-3表达与压力负荷过重心脏的重塑相关,并可能延缓肥大反应,而不影响生存、功能障碍和心肌纤维化。

Frunza, Olga; Russo, Ilaria; Saxena, Amit; Shinde, Arti V; Humeres, Claudio; Hanif, Waqas; Rai, Vikrant; Su, Ya; Frangogiannis, Nikolaos G