Imam-Aliagan相关文献与解析，生知库 | 链接生命科学的每一步

Mitofilin Heterozygote Mice Display an Increase in Myocardial Injury and Inflammation after Ischemia/Reperfusion

线粒体丝裂素杂合子小鼠在缺血/再灌注后心肌损伤和炎症增加

期刊:Antioxidants

影响因子:

doi:10.3390/antiox12040921

Yansheng Feng, Abdulhafiz Imam Aliagan, Nathalie Tombo, Jean C Bopassa

信号转导

发表日期：2023

文献求助收藏

RIP3 Translocation into Mitochondria Promotes Mitofilin Degradation to Increase Inflammation and Kidney Injury after Renal Ischemia-Reperfusion

RIP3易位进入线粒体促进线粒体丝裂蛋白降解，增加肾缺血再灌注后的炎症和肾脏损伤

期刊:Cells

影响因子:5.1

doi:10.3390/cells11121894

Yansheng Feng, Abdulhafiz Imam Aliagan, Nathalie Tombo, Derrick Draeger, Jean C Bopassa

Cardiac ischemia/reperfusion stress reduces inner mitochondrial membrane protein (mitofilin) levels during early reperfusion

心脏缺血/再灌注应激降低早期再灌注期间内线粒体膜蛋白（线粒体丝裂蛋白）的水平

期刊:Free Radical Biology and Medicine

影响因子:7.1

doi:10.1016/j.freeradbiomed.2020.06.039

Nathalie Tombo, Abdulhafiz D Imam Aliagan, Yansheng Feng, Harpreet Singh, Jean C Bopassa

Chronic GPER1 Activation Protects Against Oxidative Stress-Induced Cardiomyoblast Death via Preservation of Mitochondrial Integrity and Deactivation of Mammalian Sterile-20-Like Kinase/Yes-Associated Protein Pathway

慢性 GPER1 激活可通过保护线粒体完整性和失活哺乳动物不育-20 样激酶/Yes 相关蛋白通路来防止氧化应激诱导的心肌细胞死亡

期刊:Frontiers in Endocrinology

影响因子:

doi:10.3389/fendo.2020.579161

Abdulhafiz Imam Aliagan, Ngonidzashe B Madungwe, Nathalie Tombo, Yansheng Feng, Jean C Bopassa

Liproxstatin-1 protects the mouse myocardium against ischemia/reperfusion injury by decreasing VDAC1 levels and restoring GPX4 levels

Liproxstatin-1 通过降低 VDAC1 水平和恢复 GPX4 水平保护小鼠心肌免受缺血/再灌注损伤

期刊:Biochemical and Biophysical Research Communications

影响因子:2.5

doi:10.1016/j.bbrc.2019.10.006

Yansheng Feng, Ngonidzashe B Madungwe, Abdulhafiz D Imam Aliagan, Nathalie Tombo, Jean C Bopassa

Clinical applications of circulating biomarkers in non-small cell lung cancer

循环生物标志物在非小细胞肺癌中的临床应用

Mitofilin Heterozygote Mice Display an Increase in Myocardial Injury and Inflammation after Ischemia/Reperfusion

线粒体丝裂素杂合子小鼠在缺血/再灌注后心肌损伤和炎症增加

RIP3 Translocation into Mitochondria Promotes Mitofilin Degradation to Increase Inflammation and Kidney Injury after Renal Ischemia-Reperfusion

RIP3易位进入线粒体促进线粒体丝裂蛋白降解，增加肾缺血再灌注后的炎症和肾脏损伤

Cardiac ischemia/reperfusion stress reduces inner mitochondrial membrane protein (mitofilin) levels during early reperfusion

心脏缺血/再灌注应激降低早期再灌注期间内线粒体膜蛋白（线粒体丝裂蛋白）的水平

Chronic GPER1 Activation Protects Against Oxidative Stress-Induced Cardiomyoblast Death via Preservation of Mitochondrial Integrity and Deactivation of Mammalian Sterile-20-Like Kinase/Yes-Associated Protein Pathway

慢性 GPER1 激活可通过保护线粒体完整性和失活哺乳动物不育-20 样激酶/Yes 相关蛋白通路来防止氧化应激诱导的心肌细胞死亡

Liproxstatin-1 protects the mouse myocardium against ischemia/reperfusion injury by decreasing VDAC1 levels and restoring GPX4 levels

Liproxstatin-1 通过降低 VDAC1 水平和恢复 GPX4 水平保护小鼠心肌免受缺血/再灌注损伤