日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Increased expression of SLC25A1/CIC causes an autistic-like phenotype with altered neuron morphology

SLC25A1/CIC 表达增加导致类似自闭症的表型,并伴有神经元形态改变

Michael J Rigby, Nicola Salvatore Orefice, Alexis J Lawton, Min Ma, Samantha L Shapiro, Sue Y Yi, Inca A Dieterich, Alyssa Frelka, Hannah N Miles, Robert A Pearce, John Paul J Yu, Lingjun Li, John M Denu, Luigi Puglielli

ATase inhibition rescues age-associated proteotoxicity of the secretory pathway

ATase抑制剂可挽救分泌途径中与年龄相关的蛋白毒性。

Maeghan Murie # ,Yajing Peng # ,Michael J Rigby ,Inca A Dieterich ,Mark A Farrugia ,Andreas Endresen ,Anita Bhattacharyya ,Luigi Puglielli

SLC13A5/sodium-citrate co-transporter overexpression causes disrupted white matter integrity and an autistic-like phenotype

SLC13A5/柠檬酸钠共转运体过表达导致白质完整性破坏和类似自闭症的表型

Michael J Rigby, Nicola Salvatore Orefice, Alexis J Lawton, Min Ma, Samantha L Shapiro, Sue Y Yi, Inca A Dieterich, Alyssa Frelka, Hannah N Miles, Robert A Pearce, John Paul J Yu, Lingjun Li, John M Denu, Luigi Puglielli

Acetyl-CoA flux from the cytosol to the ER regulates engagement and quality of the secretory pathway

从胞质溶胶到内质网的乙酰辅酶 A 通量调节分泌途径的参与和质量

Inca A Dieterich #, Yusi Cui #, Megan M Braun, Alexis J Lawton, Nicklaus H Robinson, Jennifer L Peotter, Qing Yu, Jason C Casler, Benjamin S Glick, Anjon Audhya, John M Denu, Lingjun Li, Luigi Puglielli

Acetyl-CoA flux regulates the proteome and acetyl-proteome to maintain intracellular metabolic crosstalk

乙酰辅酶 A 通量调节蛋白质组和乙酰蛋白质组,以维持细胞内代谢串扰

Inca A Dieterich, Alexis J Lawton, Yajing Peng, Qing Yu, Timothy W Rhoads, Katherine A Overmyer, Yusi Cui, Eric A Armstrong, Porsha R Howell, Maggie S Burhans, Lingjun Li, John M Denu, Joshua J Coon, Rozalyn M Anderson, Luigi Puglielli

Increased transport of acetyl-CoA into the endoplasmic reticulum causes a progeria-like phenotype

乙酰辅酶A向内质网转运增加会导致类似早衰症的表型

Peng, Yajing; Shapiro, Samantha L; Banduseela, Varuna C; Dieterich, Inca A; Hewitt, Kyle J; Bresnick, Emery H; Kong, Guangyao; Zhang, Jing; Schueler, Kathryn L; Keller, Mark P; Attie, Alan D; Hacker, Timothy A; Sullivan, Ruth; Kielar-Grevstad, Elle; Arriola Apelo, Sebastian I; Lamming, Dudley W; Anderson, Rozalyn M; Puglielli, Luigi