日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Antigen specificity shapes distinct aging trajectories of memory CD8⁺ T cells

抗原特异性塑造了记忆性CD8⁺ T细胞不同的衰老轨迹。

Ines Sturmlechner # ,Abhinav Jain # ,Bin Hu ,Rohit R Jadhav ,Wenqiang Cao ,Hirohisa Okuyama ,Lu Tian ,Cornelia M Weyand ,Jörg J Goronzy

IKZF1 and UBR4 gene variants drive autoimmunity and Th2 polarization in IgG4-related disease

IKZF1 和 UBR4 基因变异驱动 IgG4 相关疾病中的自身免疫和 Th2 极化

Qingxiang Liu ,Yanyan Zheng ,Ines Sturmlechner ,Abhinav Jain ,Maryam Own ,Qiankun Yang ,Huimin Zhang ,Filippo Pinto E Vairo ,Karen Cerosaletti ,Jane H Buckner ,Kenneth J Warrington ,Matthew J Koster ,Cornelia M Weyand ,Jörg J Goronzy

PREX1 improves homeostatic proliferation to maintain a naive CD4+ T cell compartment in older age

PREX1 改善稳态增殖,以维持老年人体内的初始 CD4+ T 细胞群。

Huimin Zhang ,Hirohisa Okuyama ,Abhinav Jain ,Rohit R Jadhav ,Bowen Wu ,Ines Sturmlechner ,Jose Morales ,Shozo Ohtsuki ,Cornelia M Weyand ,Jӧrg J Goronzy

Senescent alveolar macrophages promote early-stage lung tumorigenesis

衰老的肺泡巨噬细胞促进早期肺肿瘤的发生

Luis I Prieto ,Ines Sturmlechner ,Sara I Graves ,Cheng Zhang ,Nick P Goplen ,Eunhee S Yi ,Jie Sun ,Hu Li ,Darren J Baker

CISH impairs lysosomal function in activated T cells resulting in mitochondrial DNA release and inflammaging

CISH会损害活化T细胞的溶酶体功能,导致线粒体DNA释放和炎症衰老。

Jun Jin # ,Yunmei Mu # ,Huimin Zhang ,Ines Sturmlechner ,Chenyao Wang ,Rohit R Jadhav ,Qiong Xia ,Cornelia M Weyand ,Jorg J Goronzy

Stem-like CD4+ T cells in perivascular tertiary lymphoid structures sustain autoimmune vasculitis

血管周围三级淋巴结构中的干细胞样CD4+ T细胞维持自身免疫性血管炎

Yuki Sato ,Abhinav Jain ,Shozo Ohtsuki ,Hirohisa Okuyama ,Ines Sturmlechner ,Yoshinori Takashima ,Kevin-Phu C Le ,Melanie C Bois ,Gerald J Berry ,Kenneth J Warrington ,Jörg J Goronzy ,Cornelia M Weyand

TRIB2 safeguards naive T cell homeostasis during aging

TRIB2 在衰老过程中保护初始 T 细胞的稳态。

Wenqiang Cao ,Ines Sturmlechner ,Huimin Zhang ,Jun Jin ,Bin Hu ,Rohit R Jadhav ,Fengqin Fang ,Cornelia M Weyand ,Jörg J Goronzy

p21 induces a senescence program and skeletal muscle dysfunction

p21 诱导衰老程序和骨骼肌功能障碍

Davis A Englund, Alyssa Jolliffe, Zaira Aversa, Xu Zhang, Ines Sturmlechner, Ayumi E Sakamoto, Julianna D Zeidler, Gina M Warner, Colton McNinch, Thomas A White, Eduardo N Chini, Darren J Baker, Jan M van Deursen, Nathan K LeBrasseur

Transfer learning in a biomaterial fibrosis model identifies in vivo senescence heterogeneity and contributions to vascularization and matrix production across species and diverse pathologies

生物材料纤维化模型中的迁移学习可识别体内衰老异质性以及对跨物种和不同病理的血管化和基质产生的贡献

Christopher Cherry, James I Andorko, Kavita Krishnan, Joscelyn C Mejías, Helen Hieu Nguyen, Katlin B Stivers, Elise F Gray-Gaillard, Anna Ruta, Jin Han, Naomi Hamada, Masakazu Hamada, Ines Sturmlechner, Shawn Trewartha, John H Michel, Locke Davenport Huyer, Matthew T Wolf, Ada J Tam, Alexis N Peña, 

Senescent cells limit p53 activity via multiple mechanisms to remain viable

衰老细胞通过多种机制限制p53活性以保持活力

Ines Sturmlechner, Chance C Sine, Karthik B Jeganathan, Cheng Zhang, Raul O Fierro Velasco, Darren J Baker, Hu Li, Jan M van Deursen