日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Inhibition of Mertk Signaling Enhances Bone Healing after Tooth Extraction

抑制Mertk信号通路可促进拔牙后骨愈合

Decker, A M; Matsumoto, M; Decker, J T; Roh, A; Inohara, N; Sugai, J; Martin, K; Taichman, R; Kaigler, D; Shea, L D; Núñez, G

A specific gene-microbe interaction drives the development of Crohn's disease-like colitis in mice

特定的基因-微生物相互作用驱动小鼠发生类似克罗恩病的结肠炎。

Caruso, R; Mathes, T; Martens, E C; Kamada, N; Nusrat, A; Inohara, N; Núñez, G

Mechanisms of inflammation-driven bacterial dysbiosis in the gut

炎症驱动的肠道菌群失调机制

Zeng, M Y; Inohara, N; Nuñez, G

The Role of Oral Pathobionts in Dysbiosis during Periodontitis Development

口腔致病菌在牙周炎发展过程中菌群失调中的作用

Jiao, Y; Hasegawa, M; Inohara, N

Divergence of the systemic immune response following oral infection with distinct strains of Porphyromonas gingivalis.

不同牙龈卟啉单胞菌菌株引起的口腔感染后,全身免疫反应存在差异

Marchesan J T, Morelli T, Lundy S K, Jiao Y, Lim S, Inohara N, Nunez G, Fox D A, Giannobile W V

CIDE, a novel family of cell death activators with homology to the 45 kDa subunit of the DNA fragmentation factor

CIDE是一类新型细胞死亡激活因子,与DNA片段化因子的45 kDa亚基具有同源性。

Inohara, N; Koseki, T; Chen, S; Wu, X; Núñez, G

harakiri, a novel regulator of cell death, encodes a protein that activates apoptosis and interacts selectively with survival-promoting proteins Bcl-2 and Bcl-X(L)

harakiri 是一种新型的细胞死亡调节因子,它编码一种能够激活细胞凋亡并与促进生存的蛋白 Bcl-2 和 Bcl-X(L) 选择性相互作用的蛋白质。

Inohara, N; Ding, L; Chen, S; Núñez, G

Auxin-binding protein located in the endoplasmic reticulum of maize shoots: molecular cloning and complete primary structure

玉米幼苗内质网中生长素结合蛋白:分子克隆和完整一级结构

Inohara, N; Shimomura, S; Fukui, T; Futai, M