日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Visualization of the Redox Status of Cytosolic Glutathione Using the Organelle- and Cytoskeleton-Targeted Redox Sensors

利用靶向细胞器和细胞骨架的氧化还原传感器可视化胞质谷胱甘肽的氧化还原状态

Hatori, Yuta; Kubo, Takanori; Sato, Yuichiro; Inouye, Sachiye; Akagi, Reiko; Seyama, Toshio

Local redox environment beneath biological membranes probed by palmitoylated-roGFP

利用棕榈酰化-roGFP探测生物膜下的局部氧化还原环境

Hatori, Yuta; Inouye, Sachiye; Akagi, Reiko; Seyama, Toshio

Heat shock factor 2 is required for maintaining proteostasis against febrile-range thermal stress and polyglutamine aggregation

热休克因子2对于维持蛋白质稳态,抵抗发热范围内的热应激和多聚谷氨酰胺聚集至关重要。

Shinkawa, Toyohide; Tan, Ke; Fujimoto, Mitsuaki; Hayashida, Naoki; Yamamoto, Kaoru; Takaki, Eiichi; Takii, Ryosuke; Prakasam, Ramachandran; Inouye, Sachiye; Mezger, Valerie; Nakai, Akira

A novel mouse HSF3 has the potential to activate nonclassical heat-shock genes during heat shock.

一种新型小鼠 HSF3 有可能在热休克期间激活非经典热休克基因

Fujimoto Mitsuaki, Hayashida Naoki, Katoh Takuma, Oshima Kouji, Shinkawa Toyohide, Prakasam Ramachandran, Tan Ke, Inouye Sachiye, Takii Ryosuke, Nakai Akira

Analysis of HSF4 binding regions reveals its necessity for gene regulation during development and heat shock response in mouse lenses

对 HSF4 结合区域的分析揭示了其在小鼠晶状体发育和热休克反应过程中基因调控的必要性

Fujimoto, Mitsuaki; Oshima, Koji; Shinkawa, Toyohide; Wang, Bei Bei; Inouye, Sachiye; Hayashida, Naoki; Takii, Ryosuke; Nakai, Akira

A novel HSF1-mediated death pathway that is suppressed by heat shock proteins

一种新型的HSF1介导的细胞死亡通路,可被热休克蛋白抑制

Hayashida, Naoki; Inouye, Sachiye; Fujimoto, Mitsuaki; Tanaka, Yasunori; Izu, Hanae; Takaki, Eiichi; Ichikawa, Hitoshi; Rho, Jaerang; Nakai, Akira

HSF4 is required for normal cell growth and differentiation during mouse lens development

HSF4是小鼠晶状体发育过程中正常细胞生长和分化所必需的

Fujimoto, Mitsuaki; Izu, Hanae; Seki, Keisuke; Fukuda, Ken; Nishida, Teruo; Yamada, Shu-Ichi; Kato, Kanefusa; Yonemura, Shigenobu; Inouye, Sachiye; Nakai, Akira

Activation of heat shock genes is not necessary for protection by heat shock transcription factor 1 against cell death due to a single exposure to high temperatures

热休克基因的激活对于热休克转录因子1抵抗单次高温暴露引起的细胞死亡的保护作用并非必要条件。

Inouye, Sachiye; Katsuki, Kensaku; Izu, Hanae; Fujimoto, Mitsuaki; Sugahara, Kazuma; Yamada, Shu-Ichi; Shinkai, Yoichi; Oka, Yoshitomo; Katoh, Yumiko; Nakai, Akira