日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

β-TrCP1 degradation is a novel action mechanism of PI3K/mTOR inhibitors in triple-negative breast cancer cells

β-TrCP1降解是PI3K/mTOR抑制剂在三阴性乳腺癌细胞中的新作用机制

Yong Weon Yi, Hyo Jin Kang, Edward Jeong Bae, Seunghoon Oh, Yeon-Sun Seong, Insoo Bae

Dual inhibition of EGFR and MET induces synthetic lethality in triple-negative breast cancer cells through downregulation of ribosomal protein S6

EGFR 和 MET 的双重抑制通过下调核糖体蛋白 S6 诱导三阴性乳腺癌细胞的合成致死

Yong Weon Yi, Kyusic You, Edward Jeong Bae, Sahng-June Kwak, Yeon-Sun Seong, Insoo Bae

Disruption of STAT3-DNMT1 interaction by SH-I-14 induces re-expression of tumor suppressor genes and inhibits growth of triple-negative breast tumor

SH-I-14 破坏 STAT3-DNMT1 相互作用可诱导肿瘤抑制基因重新表达并抑制三阴性乳腺肿瘤的生长

Hyo Jin Kang #, Yong Weon Yi #, Shu-Jie Hou #, Hee Jeong Kim, Yali Kong, Insoo Bae, Milton L Brown

Novel carbazole inhibits phospho-STAT3 through induction of protein-tyrosine phosphatase PTPN6

新型咔唑通过诱导蛋白酪氨酸磷酸酶 PTPN6 来抑制磷酸化 STAT3

Shujie Hou, Yong Weon Yi, Hyo Jin Kang, Li Zhang, Hee Jeong Kim, Yali Kong, Yong Liu, Kan Wang, Hye-Sik Kong, Scott Grindrod, Insoo Bae, Milton L Brown

Inhibition of NRF2 by PIK-75 augments sensitivity of pancreatic cancer cells to gemcitabine

PIK-75 抑制 NRF2 可增强胰腺癌细胞对吉西他滨的敏感性

Hong-Quan Duong, Yong Weon Yi, Hyo Jin Kang, Young Bin Hong, Wenxi Tang, Antai Wang, Yeon-Sun Seong, Insoo Bae

HER2 confers drug resistance of human breast cancer cells through activation of NRF2 by direct interaction

HER2通过直接相互作用激活NRF2,使人类乳腺癌细胞产生耐药性

Hyo Jin Kang, Yong Weon Yi, Young Bin Hong, Hee Jeong Kim, Young-Joo Jang, Yeon-Sun Seong, Insoo Bae

Inhibition of checkpoint kinase 2 (CHK2) enhances sensitivity of pancreatic adenocarcinoma cells to gemcitabine

抑制检查点激酶 2 (CHK2) 可增强胰腺腺癌细胞对吉西他滨的敏感性

Hong-Quan Duong, Young Bin Hong, Jung Soon Kim, Hee-Seok Lee, Yong Weon Yi, Yeon Jeong Kim, Antai Wang, Wenjing Zhao, Chi Heum Cho, Yeon-Sun Seong, Insoo Bae

Inhibition of the PI3K/AKT pathway potentiates cytotoxicity of EGFR kinase inhibitors in triple-negative breast cancer cells

抑制 PI3K/AKT 通路可增强 EGFR 激酶抑制剂对三阴性乳腺癌细胞的细胞毒性

Yong Weon Yi, Wooyoung Hong, Hyo Jin Kang, Hee Jeong Kim, Wenjing Zhao, Antai Wang, Yeon-Sun Seong, Insoo Bae

Inhibition of constitutively activated phosphoinositide 3-kinase/AKT pathway enhances antitumor activity of chemotherapeutic agents in breast cancer susceptibility gene 1-defective breast cancer cells

抑制组成性激活的磷酸肌醇 3-激酶/AKT 通路可增强化疗药物在乳腺癌易感基因 1 缺陷乳腺癌细胞中的抗肿瘤活性

Yong Weon Yi, Hyo Jin Kang, Hee Jeong Kim, Jae Seok Hwang, Antai Wang, Insoo Bae

Targeting mutant p53 by a SIRT1 activator YK-3-237 inhibits the proliferation of triple-negative breast cancer cells

SIRT1 激活剂 YK-3-237 靶向突变 p53 可抑制三阴性乳腺癌细胞的增殖

Yong Weon Yi, Hyo Jin Kang, Hee Jeong Kim, Yali Kong, Milton L Brown, Insoo Bae