日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Cardiomyocyte and endothelial cells play distinct roles in the tumour necrosis factor (TNF)-dependent atrial responses and increased atrial fibrillation vulnerability induced by endurance exercise training in mice

心肌细胞和内皮细胞在小鼠耐力运动训练引起的肿瘤坏死因子 (TNF) 依赖性心房反应和心房颤动脆弱性增加中发挥着不同的作用

Robert Lakin, Nazari Polidovitch, Sibao Yang, Mihir Parikh, Xueyan Liu, Ryan Debi, Xiaodong Gao, Wenliang Chen, Camilo Guzman, Simona Yakobov, Farzad Izaddoustdar, Marianne Wauchop, Qian Lei, Weimin Xu, Sergei A Nedospasov, Vincent M Christoffels, Peter H Backx

Transcriptomic Bioinformatic Analyses of Atria Uncover Involvement of Pathways Related to Strain and Post-translational Modification of Collagen in Increased Atrial Fibrillation Vulnerability in Intensely Exercised Mice

心房转录组生物信息学分析揭示与应变和胶原蛋白翻译后修饰相关的通路参与了高强度运动小鼠心房颤动脆弱性的增加

Yena Oh, Sibao Yang, Xueyan Liu, Sayantan Jana, Farzad Izaddoustdar, Xiaodong Gao, Ryan Debi, Dae-Kyum Kim, Kyoung-Han Kim, Ping Yang, Zamaneh Kassiri, Robert Lakin, Peter H Backx

Changes in Heart Rate and Its Regulation by the Autonomic Nervous System Do Not Differ Between Forced and Voluntary Exercise in Mice

小鼠在强迫运动和自愿运动时心率变化及其自主神经系统调节并无差异

Robert Lakin, Camilo Guzman, Farzad Izaddoustdar, Nazari Polidovitch, Jack M Goodman, Peter H Backx

Increased atrial arrhythmia susceptibility induced by intense endurance exercise in mice requires TNFα

小鼠剧烈耐力运动引起心房心律失常易感性增加需要 TNFα

Roozbeh Aschar-Sobbi, Farzad Izaddoustdar, Adam S Korogyi, Qiongling Wang, Gerrie P Farman, FengHua Yang, Wallace Yang, David Dorian, Jeremy A Simpson, Jari M Tuomi, Douglas L Jones, Kumaraswamy Nanthakumar, Brian Cox, Xander H T Wehrens, Paul Dorian, Peter H Backx

Leptin-induced cardiomyocyte hypertrophy reveals both calcium-dependent and calcium-independent/RhoA-dependent calcineurin activation and NFAT nuclear translocation

瘦素诱导的心肌细胞肥大揭示了钙依赖性和钙非依赖性/RhoA 依赖性的钙调神经磷酸酶激活以及 NFAT 核转位

Venkatesh Rajapurohitam, Farzad Izaddoustdar, Eduardo Martinez-Abundis, Morris Karmazyn

Iron overload decreases CaV1.3-dependent L-type Ca2+ currents leading to bradycardia, altered electrical conduction, and atrial fibrillation

铁过载会降低 CaV1.3 依赖的 L 型 Ca2+ 电流,导致心动过缓、电传导改变和心房颤动。

Rose, Robert A; Sellan, Michael; Simpson, Jeremy A; Izaddoustdar, Farzad; Cifelli, Carlo; Panama, Brian K; Davis, Mark; Zhao, Dongling; Markhani, Moniba; Murphy, Geoffrey G; Striessnig, Joerg; Liu, Peter P; Heximer, Scott P; Backx, Peter H