日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Increased NMDAR activity and GluN2A-NMDAR silent synapse expansion induced by chronic benzodiazepine treatment

长期服用苯二氮卓类药物可增加NMDAR活性并诱导GluN2A-NMDAR沉默突触扩张。

Chapman, Caitlyn A; Povysheva, Nadya; Johnson, Jon W; Jacob, Tija C

Chronic benzodiazepine treatment triggers gephyrin scaffold destabilization and GABA(A)R subsynaptic reorganization.

长期服用苯二氮卓类药物会引发 gephyrin 支架不稳定和 GABA(A)R 突触下重组

Chapman Caitlyn A, Povysheva Nadya, Tarr Tyler B, Nuwer Jessica L, Meriney Stephen D, Johnson Jon W, Jacob Tija C

The Yin and Yang of GABAergic and Glutamatergic Synaptic Plasticity: Opposites in Balance by Crosstalking Mechanisms

GABA能和谷氨酸能突触可塑性的阴阳关系:通过相互作用机制实现对立面的平衡

Chapman, Caitlyn A; Nuwer, Jessica L; Jacob, Tija C

Ischemic Injury-Induced CaMKIIδ and CaMKIIγ Confer Neuroprotection Through the NF-κB Signaling Pathway

缺血性损伤诱导的CaMKIIδ和CaMKIIγ通过NF-κB信号通路发挥神经保护作用

Ye, Jing; Das, Sabyasachi; Roy, Adhiraj; Wei, Wenzhong; Huang, Huachen; Lorenz-Guertin, Joshua Michael; Xu, Qian; Jacob, Tija C; Wang, Bing; Sun, Dandan; Wang, Qiming Jane

Mutational analysis implicates the amyloid fibril as the toxic entity in Huntington's disease

突变分析表明,淀粉样蛋白原纤维是亨廷顿病中的毒性物质。

Drombosky, Kenneth W; Rode, Sascha; Kodali, Ravi; Jacob, Tija C; Palladino, Michael J; Wetzel, Ronald

γ2 GABA(A)R Trafficking and the Consequences of Human Genetic Variation

γ2 GABA(A)R 转运及其对人类遗传变异的影响

Lorenz-Guertin, Joshua M; Bambino, Matthew J; Jacob, Tija C

GABA type a receptor trafficking and the architecture of synaptic inhibition

GABA A 型受体转运和突触抑制的结构

Lorenz-Guertin, Joshua M; Jacob, Tija C

The EGL-21 carboxypeptidase E facilitates acetylcholine release at Caenorhabditis elegans neuromuscular junctions.

EGL-21 羧肽酶 E 促进秀丽隐杆线虫神经肌肉接头处乙酰胆碱的释放

Jacob Tija C, Kaplan Joshua M