Loss of UCP2 attenuates mitochondrial dysfunction without altering ROS production and uncoupling activity
UCP2 的缺失可减轻线粒体功能障碍,而不改变活性氧(ROS)的产生和解偶联活性。
期刊:PLoS Genetics
影响因子:3.7
doi:10.1371/journal.pgen.1004385
Kukat, Alexandra; Dogan, Sukru Anil; Edgar, Daniel; Mourier, Arnaud; Jacoby, Christoph; Maiti, Priyanka; Mauer, Jan; Becker, Christina; Senft, Katharina; Wibom, Rolf; Kudin, Alexei P; Hultenby, Kjell; Flögel, Ulrich; Rosenkranz, Stephan; Ricquier, Daniel; Kunz, Wolfram S; Trifunovic, Aleksandra