Enhancing KLF15 activity in cardiomyocytes: a novel approach to prevent pathological reprogramming and fibrosis via nuclease-deficient dCas9VPR
增强心肌细胞中 KLF15 的活性：一种通过核酸酶缺陷型 dCas9VPR 预防病理性重编程和纤维化的新方法
期刊:Signal Transduction and Targeted Therapy
影响因子:
doi:10.1038/s41392-026-02593-9
Schoger, Eric; Kim, Rosa; Bleckwedel, Federico; Peralta, Tomás M; Priesmeier, Laura; Fischer, Janek A; Stengel, Laura; Rocha, Cheila; Santos, Gabriela L; Lutz, Susanne; Boileau, Etienne; Baumgarten, Nina; Schulz, Marcel H; Dieterich, Christoph; Müller, Oliver J; Cyganek, Lukas; Cabrera-Orefice, Alfredo; Eberl, Hanna; Maack, Christoph; Streckfuss-Bömeke, Katrin; Pavez-Giani, Mario G; Doroudgar, Shirin; Sossalla, Samuel; Zelarayán, Laura C
