日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

ATG16L1 WD domain and linker regulates lipid trafficking to maintain plasma membrane integrity to limit influenza virus infection.

ATG16L1 WD 结构域和连接子调节脂质运输,以维持质膜完整性,从而限制流感病毒感染

Bone Benjamin, Griffith Luke, Jefferson Matthew, Yamauchi Yohei, Wileman Thomas, Powell Penny P

The TECPR1:ATG5-ATG12 complex conjugates LC3/ATG8 to damaged lysosomes that expose luminal glycans in response to osmotic imbalance.

TECPR1:ATG5-ATG12 复合物将 LC3/ATG8 连接到受损的溶酶体上,从而在渗透压失衡时暴露腔内聚糖

Wang Yingxue, Jefferson Matthew, Ramos Maria, Whelband Matthew, Kreuzer Kristin, Khuu Grace, Lazarou Michael, Mccoll James, Lazenby James, Whitchurch Cynthia B, Verkade Paul, Mayer Ulrike, Wileman Thomas

Exploring Endotypes in Chronic Rhinosinusitis (ExpRess): Protocol for a cohort study

探索慢性鼻窦炎的内型(ExpRess):队列研究方案

Gokani, Shyam Ajay; Jefferson, Matthew; Gavrilovic, Jelena; Clark, Allan; Hildebrand, Falk; Wileman, Tom; Hopkins, Claire; Philpott, Carl

Control of infection by LC3-associated phagocytosis, CASM, and detection of raised vacuolar pH by the V-ATPase-ATG16L1 axis

LC3相关吞噬作用、CASM对感染的控制以及V-ATPase-ATG16L1轴对液泡pH值升高的检测

Wang, Yingxue; Ramos, Maria; Jefferson, Matthew; Zhang, Weijiao; Beraza, Naiara; Carding, Simon; Powell, Penny P; Stewart, James P; Mayer, Ulrike; Wileman, Thomas

Application of Topical Sucralfate and Topical Platelet-Rich Plasma Improves Wound Healing in Diabetic Ulcer Rats Wound Model

局部应用硫糖铝和富血小板血浆可改善糖尿病溃疡大鼠伤口模型的伤口愈合

Yuniati, Renni; Innelya, Innelya; Rachmawati, Arti; Charlex, Harold Jefferson Matthew; Rahmatika, Alfi; Khrisna, Matthew Brian; Mundhofir, Farmaditya E P; Hario Seno, K Heri Nugroho; Kristina, Tri Nur

Bidirectional Homeostatic Regulation of a Depression-Related Brain State by Gamma-Aminobutyric Acidergic Deficits and Ketamine Treatment

通过 γ-氨基丁酸能缺陷和氯胺酮治疗对抑郁相关脑状态进行双向稳态调节

Zhen Ren, Horia Pribiag, Sarah J Jefferson, Matthew Shorey, Thomas Fuchs, David Stellwagen, Bernhard Luscher

Genetic diseases conferring resistance to infectious diseases

赋予对传染病抵抗力的遗传性疾病

Withrock, Isabelle C; Anderson, Stephen J; Jefferson, Matthew A; McCormack, Garrett R; Mlynarczyk, Gregory S A; Nakama, Aron; Lange, Jennifer K; Berg, Carrie A; Acharya, Sreemoyee; Stock, Matthew L; Lind, Melissa S; Luna, K C; Kondru, Naveen C; Manne, Sireesha; Patel, Bhavika B; de la Rosa, Bierlein M; Huang, Kuei-Pin; Sharma, Shaunik; Hu, Hilary Z; Kanuri, Sri Harsha; Carlson, Steve A

The pestivirus N terminal protease N(pro) redistributes to mitochondria and peroxisomes suggesting new sites for regulation of IRF3 by N(pro.)

瘟病毒N末端蛋白酶Npro重新分布到线粒体和过氧化物酶体,提示Npro对IRF3有新的调控位点。

Matthew Jefferson, Matthew Whelband, Irina Mohorianu, Penny P Powell