Jennifer Veth相关文献与解析，生知库 | 链接生命科学的每一步

Identification of new drugs to counteract anti-spike IgG-induced hyperinflammation in severe COVID-19

鉴定可对抗重症 COVID-19 患者抗刺突蛋白 IgG 诱发的过度炎症的新药

doi:10.26508/lsa.202302106

Chiara E Geyer, Hung-Jen Chen, Alexander P Bye, Xue D Manz, Denise Guerra, Tom G Caniels, Tom Pl Bijl, Guillermo R Griffith, Willianne Hoepel, Steven W de Taeye, Jennifer Veth, Alexander Pj Vlaar; Amsterdam UMC COVID-19 Biobank; Gestur Vidarsson, Harm Jan Bogaard, Jurjan Aman, Jonathan M Gibbins, Ma

免疫

新冠

Immunology/Inflammation

发表日期：2023

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Transcriptome analysis reveals the contribution of oligodendrocyte and radial glia-derived cues for maintenance of microglia identity

转录组分析揭示了少突胶质细胞和放射状胶质细胞衍生的线索对维持小胶质细胞身份的贡献

期刊:Glia

影响因子:5.4

doi:10.1002/glia.24136

Raissa Timmerman, Ella A Zuiderwijk-Sick, Nynke Oosterhof, Anke E J 't Jong, Jennifer Veth, Saskia M Burm, Tjakko J van Ham, Jeffrey J Bajramovic

Exposure of Microglia to Interleukin-4 Represses NF-κB-Dependent Transcription of Toll-Like Receptor-Induced Cytokines

小胶质细胞暴露于白细胞介素 4 可抑制 NF-κB 依赖的 Toll 样受体诱导的细胞因子转录

期刊:Frontiers in Immunology

影响因子:5.7

doi:10.3389/fimmu.2021.771453

Ella A Zuiderwijk-Sick, Céline van der Putten, Raissa Timmerman, Jennifer Veth, Erica M Pasini, Linda van Straalen, Paul van der Valk, Sandra Amor, Jeffrey J Bajramovic

Inflammasome-induced IL-1β secretion in microglia is characterized by delayed kinetics and is only partially dependent on inflammatory caspases

炎症小体诱导的小胶质细胞 IL-1β 分泌具有延迟动力学特征，并且仅部分依赖于炎症 caspase

期刊:Journal of Neuroscience

影响因子:4.4

doi:10.1523/JNEUROSCI.2510-14.2015

Saskia M Burm, Ella A Zuiderwijk-Sick, Anke E J 't Jong, Céline van der Putten, Jennifer Veth, Ivanela Kondova, Jeffrey J Bajramovic

Testosterone and estradiol reduce inflammation of human macrophages induced by anti-SARS-CoV-2 IgG

睾酮和雌二醇可减轻抗SARS-CoV-2 IgG诱导的人类巨噬细胞炎症。

IgA2 immune complexes selectively promote inflammation by human CD103+ dendritic cells

IgA2 免疫复合物选择性地促进人类 CD103+ 树突状细胞的炎症

Identification of new drugs to counteract anti-spike IgG-induced hyperinflammation in severe COVID-19

鉴定可对抗重症 COVID-19 患者抗刺突蛋白 IgG 诱发的过度炎症的新药

Transcriptome analysis reveals the contribution of oligodendrocyte and radial glia-derived cues for maintenance of microglia identity

转录组分析揭示了少突胶质细胞和放射状胶质细胞衍生的线索对维持小胶质细胞身份的贡献

Exposure of Microglia to Interleukin-4 Represses NF-κB-Dependent Transcription of Toll-Like Receptor-Induced Cytokines

小胶质细胞暴露于白细胞介素 4 可抑制 NF-κB 依赖的 Toll 样受体诱导的细胞因子转录

Inflammasome-induced IL-1β secretion in microglia is characterized by delayed kinetics and is only partially dependent on inflammatory caspases

炎症小体诱导的小胶质细胞 IL-1β 分泌具有延迟动力学特征，并且仅部分依赖于炎症 caspase