Jesper Falkesgaard相关文献与解析，生知库

Liqiong Jiang #, Lars P Lunding #, William S Webber, Karsten Beckmann, Tania Azam, Jesper Falkesgaard Højen, Jesus Amo-Aparicio, Alberto Dinarello, Tom T Nguyen, Ulrich Pessara, Daniel Parera, David J Orlicky, Stephan Fischer, Michael Wegmann, Charles A Dinarello, Suzhao Li

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Goat

IgG F(ab')2

发表日期：2024

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Pharmacologic inhibition of NLRP3 reduces the levels of α-synuclein and protects dopaminergic neurons in a model of Parkinson's disease

药物抑制 NLRP3 可降低帕金森病模型中的 α-突触核蛋白水平并保护多巴胺能神经元

期刊:Journal of Neuroinflammation

影响因子:9.3

doi:10.1186/s12974-023-02830-w

Jesus Amo-Aparicio, Jonathan Daly, Jesper Falkesgaard Højen, Charles A Dinarello

Cell targeting and immunostimulatory properties of a novel Fcγ-receptor-independent agonistic anti-CD40 antibody in rhesus macaques

一种新型Fcγ受体非依赖性激动型抗CD40抗体在恒河猴中的细胞靶向性和免疫刺激特性

期刊:Cellular and Molecular Life Sciences

影响因子:6.2

doi:10.1007/s00018-023-04828-2

Xianglei Yan ，Sebastian Ols ，Rodrigo Arcoverde Cerveira ，Klara Lenart ，Fredrika Hellgren ，Kewei Ye ，Alberto Cagigi ，Marcus Buggert ，Falk Nimmerjahn ，Jesper Falkesgaard Højen ，Daniel Parera ，Ulrich Pessara ，Stephan Fischer ，Karin Loré

A novel anti-human IL-1R7 antibody reduces IL-18-mediated inflammatory signaling

新型抗人 IL-1R7 抗体可降低 IL-18 介导的炎症信号

期刊:Journal of Biological Chemistry

影响因子:4

doi:10.1016/j.jbc.2021.100630

Suzhao Li, Liqiong Jiang, Karsten Beckmann, Jesper Falkesgaard Højen, Ulrich Pessara, Nicholas E Powers, Dennis M de Graaf, Tania Azam, Jared Lindenberger, Elan Z Eisenmesser, Stephan Fischer, Charles A Dinarello

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Immunology/Inflammation

The Use of Toll-Like Receptor Agonists in HIV-1 Cure Strategies

Toll样受体激动剂在HIV-1治愈策略中的应用

期刊:Frontiers in Immunology

影响因子:5.9

doi:10.3389/fimmu.2020.01112

Martinsen, Janne Tegder; Gunst, Jesper Damsgaard; Højen, Jesper Falkesgaard; Tolstrup, Martin; Søgaard, Ole Schmeltz

IL-1R3 blockade broadly attenuates the functions of six members of the IL-1 family, revealing their contribution to models of disease

IL-1R3 阻断广泛减弱了 IL-1 家族六个成员的功能，揭示了它们对疾病模型的贡献

期刊:Nature Immunology

影响因子:27.7

doi:10.1038/s41590-019-0467-1

Jesper Falkesgaard Højen, Marie Louise Vindvad Kristensen, Amy S McKee, Megan Taylor Wade, Tania Azam, Lars P Lunding, Dennis M de Graaf, Benjamin J Swartzwelter, Michael Wegmann, Martin Tolstrup, Karsten Beckman, Mayumi Fujita, Stephan Fischer, Charles A Dinarello0

发表日期：2019

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Treatment of HIV-Infected Individuals with the Histone Deacetylase Inhibitor Panobinostat Results in Increased Numbers of Regulatory T Cells and Limits Ex Vivo Lipopolysaccharide-Induced Inflammatory Responses

用组蛋白去乙酰化酶抑制剂帕比司他治疗 HIV 感染者可增加调节性 T 细胞的数量并限制体外脂多糖诱导的炎症反应。

期刊:mSphere

影响因子:3.7

doi:10.1128/mSphere.00616-17

Christel Rothe Brinkmann # ，Jesper Falkesgaard Højen # ，Thomas Aagaard Rasmussen ，Anne Sofie Kjær ，Rikke Olesen ，Paul W Denton ，Lars Østergaard ，Zhengyu Ouyang ，Mathias Lichterfeld ，Xu Yu ，Ole Schmeltz Søgaard ，Charles Dinarello ，Martin Tolstrup

An antibody to IL-1 receptor 7 protects mice from LPS-induced tissue and systemic inflammation

IL-1 受体 7 抗体可保护小鼠免受 LPS 诱发的组织和全身炎症

Pharmacologic inhibition of NLRP3 reduces the levels of α-synuclein and protects dopaminergic neurons in a model of Parkinson's disease

药物抑制 NLRP3 可降低帕金森病模型中的 α-突触核蛋白水平并保护多巴胺能神经元

Cell targeting and immunostimulatory properties of a novel Fcγ-receptor-independent agonistic anti-CD40 antibody in rhesus macaques

一种新型Fcγ受体非依赖性激动型抗CD40抗体在恒河猴中的细胞靶向性和免疫刺激特性

A novel anti-human IL-1R7 antibody reduces IL-18-mediated inflammatory signaling

新型抗人 IL-1R7 抗体可降低 IL-18 介导的炎症信号

The Use of Toll-Like Receptor Agonists in HIV-1 Cure Strategies

Toll样受体激动剂在HIV-1治愈策略中的应用

IL-1R3 blockade broadly attenuates the functions of six members of the IL-1 family, revealing their contribution to models of disease

IL-1R3 阻断广泛减弱了 IL-1 家族六个成员的功能，揭示了它们对疾病模型的贡献

Treatment of HIV-Infected Individuals with the Histone Deacetylase Inhibitor Panobinostat Results in Increased Numbers of Regulatory T Cells and Limits Ex Vivo Lipopolysaccharide-Induced Inflammatory Responses

用组蛋白去乙酰化酶抑制剂帕比司他治疗 HIV 感染者可增加调节性 T 细胞的数量并限制体外脂多糖诱导的炎症反应。