Jesse Lyons相关文献与解析，生知库 | 链接生命科学的每一步

Samantha Dale Strasser, Phaedra C Ghazi, Alina Starchenko, Myriam Boukhali, Amanda Edwards, Lucia Suarez-Lopez, Jesse Lyons, Paul S Changelian, Joseph B Monahan, Jon Jacobsen, Douglas K Brubaker, Brian A Joughin, Michael B Yaffe, Wilhelm Haas, Douglas A Lauffenburger, Kevin M Haigis

免疫

肠炎

发表日期：2019

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The colonic epithelium plays an active role in promoting colitis by shaping the tissue cytokine profile

结肠上皮通过塑造组织细胞因子谱，在促进结肠炎的发生发展中发挥积极作用。

期刊:PLoS Biology

影响因子:7.8

doi:10.1371/journal.pbio.2002417

Jesse Lyons ，Phaedra C Ghazi ，Alina Starchenko ，Alessio Tovaglieri ，Katherine R Baldwin ，Emily J Poulin ，Jessica J Gierut ，Casie Genetti ，Vijay Yajnik ，David T Breault ，Douglas A Lauffenburger ，Kevin M Haigis

Integrated in vivo multiomics analysis identifies p21-activated kinase signaling as a driver of colitis

综合体内多组学分析确定 p21 活化激酶信号是结肠炎的驱动因素

期刊:Science Signaling

影响因子:

doi:10.1126/scisignal.aan3580

Jesse Lyons, Douglas K Brubaker, Phaedra C Ghazi, Katherine R Baldwin, Amanda Edwards, Myriam Boukhali, Samantha Dale Strasser, Lucia Suarez-Lopez, Yi-Jang Lin, Vijay Yajnik, Joseph L Kissil, Wilhelm Haas, Douglas A Lauffenburger, Kevin M Haigis

Oncogenic K-Ras promotes proliferation in quiescent intestinal stem cells

致癌基因K-Ras促进静止肠道干细胞增殖

期刊:Stem Cell Research

影响因子:0.8

doi:10.1016/j.scr.2015.06.003

Jessica J Gierut ，Jesse Lyons ，Manasvi S Shah ，Casie Genetti ，David T Breault ，Kevin M Haigis

Combined deficiency of p50 and cRel in CD4+ T cells reveals an essential requirement for nuclear factor kappaB in regulating mature T cell survival and in vivo function

CD4+ T 细胞中 p50 和 cRel 的联合缺乏揭示了核因子 κB 在调节成熟 T 细胞存活和体内功能方面必不可少

期刊:Journal of Experimental Medicine

影响因子:12.6

doi:10.1084/jem.20021610

Ye Zheng, Monika Vig, Jesse Lyons, Luk Van Parijs, Amer A Beg

Substrate-based kinase activity inference identifies MK2 as driver of colitis

基于底物的激酶活性推断 MK2 是结肠炎的驱动因素

The colonic epithelium plays an active role in promoting colitis by shaping the tissue cytokine profile

结肠上皮通过塑造组织细胞因子谱，在促进结肠炎的发生发展中发挥积极作用。

Integrated in vivo multiomics analysis identifies p21-activated kinase signaling as a driver of colitis

综合体内多组学分析确定 p21 活化激酶信号是结肠炎的驱动因素

Oncogenic K-Ras promotes proliferation in quiescent intestinal stem cells

致癌基因K-Ras促进静止肠道干细胞增殖

Combined deficiency of p50 and cRel in CD4+ T cells reveals an essential requirement for nuclear factor kappaB in regulating mature T cell survival and in vivo function

CD4+ T 细胞中 p50 和 cRel 的联合缺乏揭示了核因子 κB 在调节成熟 T 细胞存活和体内功能方面必不可少