日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Evaluating current practice and knowledge about antibiotic stewardship principles in paediatric tertiary hospitals to identify target areas for future teaching activities

评估儿科三级医院目前在抗菌药物管理原则方面的实践和知识,以确定未来教学活动的目标领域

Kolberg, Laura; Buschbeck, Judith; Wagner, Annabelle; Jonat, Susanne; Wolf, Gerhard; Peters, Jochen; Behrends, Uta; Steinhauser, Maximilian; Huebner, Johannes; von Both, Ulrich

Offspring born to influenza A virus infected pregnant mice have increased susceptibility to viral and bacterial infections in early life

感染甲型流感病毒的怀孕小鼠所产的后代,在生命早期更容易感染病毒和细菌。

Henning Jacobsen ,Kerstin Walendy-Gnirß ,Nilgün Tekin-Bubenheim ,Nancy Mounogou Kouassi ,Isabel Ben-Batalla ,Nikolaus Berenbrok ,Martin Wolff ,Vinicius Pinho Dos Reis ,Martin Zickler ,Lucas Scholl ,Annette Gries ,Hanna Jania ,Andreas Kloetgen ,Arne Düsedau ,Gundula Pilnitz-Stolze ,Aicha Jeridi ,Ali Önder Yildirim ,Helmut Fuchs ,Valerie Gailus-Durner ,Claudia Stoeger ,Martin Hrabe de Angelis ,Tatjana Manuylova ,Karin Klingel ,Fiona J Culley ,Jochen Behrends ,Sonja Loges ,Bianca Schneider ,Susanne Krauss-Etschmann ,Peter Openshaw ,Gülsah Gabriel

CD73-mediated adenosine production by CD8 T cell-derived extracellular vesicles constitutes an intrinsic mechanism of immune suppression

CD73 介导的 CD8 T 细胞衍生的细胞外囊泡产生腺苷是免疫抑制的内在机制

Enja Schneider #, Riekje Winzer #, Anne Rissiek, Isabell Ricklefs, Catherine Meyer-Schwesinger, Franz L Ricklefs, Andreas Bauche, Jochen Behrends, Rudolph Reimer, Santra Brenna, Hauke Wasielewski, Melchior Lauten, Björn Rissiek, Berta Puig, Filippo Cortesi, Tim Magnus, Ralf Fliegert, Christa E Mülle

IL-6 is not Absolutely Essential for the Development of a TH17 Immune Response after an Aerosol Infection with Mycobacterium Tuberculosis H37rv

IL-6 对于结核分枝杆菌 H37rv 气溶胶感染后 TH17 免疫反应的产生并非绝对必要

Kristina Ritter, Jan Christian Sodenkamp, Alexandra Hölscher, Jochen Behrends, Christoph Hölscher

Blocking IL-10 receptor signaling ameliorates Mycobacterium tuberculosis infection during influenza-induced exacerbation

阻断IL-10受体信号传导可减轻流感诱发的结核分枝杆菌感染加重期症状

Sarah Ring ,Lars Eggers ,Jochen Behrends ,Adam Wutkowski ,Dominik Schwudke ,Andrea Kröger ,Alexandra Maximiliane Hierweger ,Christoph Hölscher ,Gülsah Gabriel ,Bianca E Schneider

Inactivation of Bacteria by γ-Irradiation to Investigate the Interaction with Antimicrobial Peptides

γ 射线灭活细菌并研究其与抗菌肽的相互作用

Wilmar Correa, Julius Brandenburg, Jochen Behrends, Lena Heinbockel, Norbert Reiling, Laura Paulowski, Dominik Schwudke, Kerstin Stephan, Guillermo Martinez-de-Tejada, Klaus Brandenburg, Thomas Gutsmann

The increased protection and pathology in Mycobacterium tuberculosis-infected IL-27R-alpha-deficient mice is supported by IL-17A and is associated with the IL-17A-induced expansion of multifunctional T cells

结核分枝杆菌感染的 IL-27R-alpha 缺陷小鼠的保护力和病理性增强由 IL-17A 支持,并且与 IL-17A 诱导的多功能 T 细胞扩增有关

Hanna Erdmann, Jochen Behrends, Kristina Ritter, Alexandra Hölscher, Johanna Volz, Ida Rosenkrands, Christoph Hölscher

De Novo Fatty Acid Synthesis During Mycobacterial Infection Is a Prerequisite for the Function of Highly Proliferative T Cells, But Not for Dendritic Cells or Macrophages

分枝杆菌感染期间的从头脂肪酸合成是高度增殖 T 细胞功能的先决条件,但不是树突状细胞或巨噬细胞的先决条件

Philipp Stüve, Lucía Minarrieta, Hanna Erdmann, Catharina Arnold-Schrauf, Maxine Swallow, Melanie Guderian, Freyja Krull, Alexandra Hölscher, Peyman Ghorbani, Jochen Behrends, Wolf-Rainer Abraham, Christoph Hölscher, Tim D Sparwasser, Luciana Berod

MED15 overexpression in prostate cancer arises during androgen deprivation therapy via PI3K/mTOR signaling

前列腺癌中的 MED15 过度表达在雄激素剥夺疗法期间通过 PI3K/mTOR 信号传导出现

Anne Offermann #, Ignacija Vlasic #, Isabella Syring, Wenzel Vogel, Christian Ruiz, Tobias Zellweger, Cyrill A Rentsch, Susanne Hagedorn, Jochen Behrends, Michael Nowak, Axel Merseburger, Lukas Bubendorf, Jutta Kirfel, Stefan Duensing, David Adler #, Sven Perner #

Epstein-Barr virus-induced gene 3 suppresses T helper type 1, type 17 and type 2 immune responses after Trypanosoma cruzi infection and inhibits parasite replication by interfering with alternative macrophage activation

Epstein-Barr 病毒诱导的基因 3 可抑制克氏锥虫感染后的 T 辅助细胞 1 型、17 型和 2 型免疫反应,并通过干扰替代巨噬细胞活化来抑制寄生虫复制

Julia Böhme, Caroline Roßnagel, Thomas Jacobs, Jochen Behrends, Christoph Hölscher, Hanna Erdmann