日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

HIV-Nef Protein Transfer to Endothelial Cells Requires Rac1 Activation and Leads to Endothelial Dysfunction Implications for Statin Treatment in HIV Patients

HIV-Nef蛋白向内皮细胞的转移需要Rac1激活,并导致内皮功能障碍,这对HIV患者的他汀类药物治疗具有重要意义。

Chelvanambi, Sarvesh; Gupta, Samir K; Chen, Xingjuan; Ellis, Bradley W; Maier, Bernhard F; Colbert, Tyler M; Kuriakose, Jithin; Zorlutuna, Pinar; Jolicoeur, Paul; Obukhov, Alexander G; Clauss, Matthias

Altered immune response differentially enhances susceptibility to Cryptococcus neoformans and Cryptococcus gattii infection in mice expressing the HIV-1 transgene

免疫反应的改变会差异性地增强表达 HIV-1 转基因小鼠对新型隐球菌和格特隐球菌感染的易感性。

Leongson, Kassandre; Cousineau-Côté, Vincent; Goupil, Mathieu; Aumont, Francine; Sénéchal, Serge; Gaboury, Louis; Jolicoeur, Paul; Kronstad, James W; de Repentigny, Louis

Xenotropic and other murine leukemia virus-related viruses in humans

人类中的异嗜性病毒和其他鼠白血病病毒相关病毒

Khan, Arifa S; McClure, Myra; Kubo, Yoshinao; Jolicoeur, Paul

Macrophage-mediated responses to Candida albicans in mice expressing the human immunodeficiency virus type 1 transgene

表达人类免疫缺陷病毒1型转基因的小鼠中巨噬细胞介导的对白色念珠菌的反应

Goupil, Mathieu; Trudelle, Emilie Bélanger; Dugas, Véronique; Racicot-Bergeron, Catherine; Aumont, Francine; Sénéchal, Serge; Hanna, Zaher; Jolicoeur, Paul; de Repentigny, Louis

Overexpression of activated murine Notch1 and Notch3 in transgenic mice blocks mammary gland development and induces mammary tumors.

转基因小鼠中激活的鼠源 Notch1 和 Notch3 的过度表达会阻碍乳腺发育并诱发乳腺肿瘤

Hu Chunyan, Diévart Anne, Lupien Mathieu, Calvo Ezequiel, Tremblay Gilles, Jolicoeur Paul

CD8+ T cells but not polymorphonuclear leukocytes are required to limit chronic oral carriage of Candida albicans in transgenic mice expressing human immunodeficiency virus type 1

CD8+ T 细胞而非多形核白细胞是限制表达 1 型人类免疫缺陷病毒的转基因小鼠体内白色念珠菌慢性口腔携带所必需的。

Marquis, Miriam; Lewandowski, Daniel; Dugas, Véronique; Aumont, Francine; Sénéchal, Serge; Jolicoeur, Paul; Hanna, Zaher; de Repentigny, Louis

Oligodendrocyte-specific expression of human immunodeficiency virus type 1 Nef in transgenic mice leads to vacuolar myelopathy and alters oligodendrocyte phenotype in vitro.

转基因小鼠中少突胶质细胞特异性表达人类免疫缺陷病毒 1 型 Nef 会导致空泡性脊髓病,并改变体外少突胶质细胞的表型

Radja Fatiha, Kay Denis G, Albrecht Steffen, Jolicoeur Paul

Cardiac disease in transgenic mice expressing human immunodeficiency virus-1 nef in cells of the immune system

转基因小鼠在免疫系统细胞中表达人类免疫缺陷病毒-1 nef 基因,导致心脏疾病

Kay, Denis G; Yue, Ping; Hanna, Zaher; Jothy, Serge; Tremblay, Etienne; Jolicoeur, Paul