日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Donor Macrophage Depletion Permits Post-Transplant Tolerance Induction in a Murine Islet Transplant Model

供体巨噬细胞清除可诱导小鼠胰岛移植模型中的移植后耐受性

Dilts, Miriam; Fay, Olivia K; Yu, Yang; Jordan, Collin Z; Luo, Xunrong

Targeting allograft inflammatory factor 1 reprograms kidney macrophages to enhance repair.

靶向同种异体移植炎症因子 1 可重编程肾脏巨噬细胞以增强修复

Husain Irma, Shah Holly, Jordan Collin Z, Natesh Naveen R, Fay Olivia K, Chen Yanting, Privratsky Jamie R, Kitai Hiroki, Souma Tomokazu, Varghese Shyni, Howell David N, Thorp Edward B, Luo Xunrong

Management of Labral Tears in the Hip: A Consensus Statement

髋关节盂唇撕裂的治疗:共识声明

Matache, Bogdan A; Belzile, Étienne L; Ayeni, Olufemi R; Garie, Luc De; Degen, Ryan M; Goudie, Richard; Heroux, Martin; Klett, Marie-Josee; Persson, Erika; Wong, Ivan; Al-Rawi, Firas; Baylis, Penny-Jane; Beaule, Paul E; Blanchet, Richard; Buchko, Jordan; Collin, Pierre; Crookham, Jason; Homayoon, Bobby; Hurley, Eoghan T; Johnston, Kelly; Khan, Moin; Lambert, Diane; Leblanc, Claire; Lemmex, Devin; Ling, Patrick; Lodhia, Parth; Longland, Billy; Kyle Martin, R; McConkey, Mark; McCormack, Bob; Moroz, Mickey; Nault, Marie-Lyne; Outerbridge, Ross; Peltz, Julie; Pozgay, Anita; Reid, David; Shallow, Scott; Shields, Ryan; Tucker, Allison; Urquhart, Nathan; Woodmass, Jarret

Defensive tolerance drives the reprogramming and dysfunction of infiltrating pathogenic B cells assuring the maintenance of tolerance.

防御性耐受驱动浸润性致病性 B 细胞的重编程和功能障碍,从而确保耐受性的维持

Hayashi Koki, Yokose Takahiro, Lancey Jenna, Szuter Edward S, Kwon Brandon, Murillo Fabian, Giarraputo Alessia, Rosales Ivy, Li Wenjun, Guinn Michael T, Cosimi Grant, Odutola Sarah Rose, Kim Joylyn, Sage Peter T, Tarantino Giuseppe, Huang Amy, Liu David, Effiom Derek, Boland Genevieve M, Cohen Sonia, McCallion Oliver, Issa Fadi, Jordan Collin, Luo Xunrong, Liss Andrew S, Ruddy David A, Piquet Michelle, Russell Paul S, Colvin Robert B, Madsen Joren C, Cosimi A Benedict, Kreisel Daniel, Alessandrini Alessandro

Heightened TLR7 signaling primes BCR-activated B cells in chronic graft-versus-host disease for effector functions

在慢性移植物抗宿主病中,增强的TLR7信号传导可使BCR激活的B细胞启动效应功能。

Bracken, Sonali J; Suthers, Amy N; DiCioccio, Rachel A; Su, Hsuan; Anand, Sarah; Poe, Jonathan C; Jia, Wei; Visentin, Jonathan; Basher, Fahmin; Jordan, Collin Z; McManigle, William C; Li, Zhiguo; Hakim, Frances T; Pavletic, Steven Z; Bhuiya, Nazmim S; Ho, Vincent T; Horwitz, Mitchell E; Chao, Nelson J; Sarantopoulos, Stefanie

Conversion of CD73hiFR4hi anergic T cells to IFN-γ-producing effector cells disrupts established immune tolerance

CD73hiFR4hi 无反应性 T 细胞向产生 IFN-γ 的效应细胞转化会破坏已建立的免疫耐受。

Dangi, Anil; Husain, Irma; Jordan, Collin Z; Yu, Shuangjin; Luo, Xunrong