日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Fiber-type vulnerability and proteostasis reprogramming in skeletal muscle during pancreatic cancer cachexia

胰腺癌恶病质期间骨骼肌纤维类型脆弱性和蛋白质稳态重编程

Xu, Bowen; Joshi, Aniket S; Tomaz da Silva, Meiricris; Liu, Silin; Kumar, Ashok

Fibroblast growth factor-inducible 14 regulates satellite cell self-renewal and expansion during skeletal muscle repair

成纤维细胞生长因子诱导蛋白14在骨骼肌修复过程中调节卫星细胞的自我更新和扩增。

Tomaz da Silva, Meiricris; Joshi, Aniket S; Kumar, Ashok

Signaling networks governing skeletal muscle growth, atrophy, and cachexia

调控骨骼肌生长、萎缩和恶病质的信号网络

Joshi, Aniket S; Tomaz da Silva, Meiricris; Kumar, Savita; Kumar, Ashok

The TWEAK/Fn14 signaling mediates skeletal muscle wasting during cancer cachexia.

TWEAK/Fn14 信号通路介导癌症恶病质期间的骨骼肌萎缩

Tomaz da Silva Meiricris, Roy Anirban, Vuong Anh Tuan, Joshi Aniket S, Josphien Cristeena, Trivedi Meghana V, Hindi Sajedah M, Narkar Vihang A, Kumar Ashok

Fiber-type vulnerability and proteostasis reprogramming in skeletal muscle during pancreatic cancer cachexia

胰腺癌恶病质期间骨骼肌纤维类型脆弱性和蛋白质稳态重编程

Xu, Bowen; Joshi, Aniket S; da Silva, Meiricris Tomaz; Liu, Silin; Kumar, Ashok

TAK1 is a key regulator of oncogenic signaling and differentiation blockade in rhabdomyosarcoma

TAK1是横纹肌肉瘤中致癌信号传导和分化阻滞的关键调节因子

Vuong, Anh Tuan; Joshi, Aniket S; Roy, Anirban; Mathukumalli, Kavya; Ho, Phuong T; Bhat, Raksha; da Silva, Meiricris Tomaz; Samanta, Tagari; Trivedi, Meghana V; Guo, Bin; Kaipparettu, Benny A; Kumar, Ashok

ER stress and ERO1: Potential therapeutic targets for inherited myopathies

内质网应激和ERO1:遗传性肌病的潜在治疗靶点

Roy, Anirban; Joshi, Aniket S; Kumar, Ashok

Mechanisms of gelofusine protection in an in vitro model of polymyxin B-associated renal injury.

凝胶渗透剂在多粘菌素 B 相关肾损伤体外模型中的保护机制

Hudson Cole S, Roy Anirban, Li Qingtian, Joshi Aniket S, Yin Taijun, Kumar Ashok, Sheikh-Hamad David, Tam Vincent H

Fn14 promotes myoblast fusion during regenerative myogenesis

Fn14促进再生性肌生成过程中成肌细胞的融合

Tomaz da Silva, Meiricris; Joshi, Aniket S; Castillo, Micah B; Koike, Tatiana E; Roy, Anirban; Gunaratne, Preethi H; Kumar, Ashok

Polygenic genetic variation affecting antibody formation underlies hypertensive renal injury in the stroke-prone spontaneously hypertensive rat

影响抗体形成的多基因遗传变异是易卒中自发性高血压大鼠高血压肾损伤的根本原因。

Dhande, Isha S; Zhu, Yaming; Joshi, Aniket S; Hicks, M John; Braun, Michael C; Doris, Peter A