Jun-ichiro Inoue相关文献与解析，生知库 | 链接生命科学的每一步

Michishige Harada,Takehisa Matsumoto,Mizuki Yamamoto,Jin Goda,Akiko Idei,Kenichi Ohtaki,Natsuki Kojima,Natsumi Yoneda,Kosuke Miyauchi,Kazushige Katsura,Mariko Ikeda,Kazuharu Hanada,Yoshiko Ishizuka-Katsura,Toshiaki Hosaka,Tamao Hisano,Toshie Kaizuka,Takako Yamamoto,Masashi Matsuda,Manabu Nakayama,Akiko Sugimoto-Ishige,Machie Sakuma,Rina Hashimoto,Kazuo Takayama,Misako Nakayama,Cong Thanh Nguyen,Hirohito Ishigaki,Yasushi Itoh,Yoshinobu Hashizume,Minoru Yoshida,Yasushi Kawaguchi,Makoto Takeda,Haruhiko Koseki,Mikako Shirouzu,Jun-Ichiro Inoue,Takashi Saito

新冠

Human

炎症/感染

SARS-CoV-2

发表日期：2025

文献求助收藏

TRAF7 determines circadian period through ubiquitination and degradation of DBP

TRAF7 通过泛素化和 DBP 降解来决定昼夜节律

期刊:Communications Biology

影响因子:5.2

doi:10.1038/s42003-024-07002-x

Shusaku Masuda #, Nobuhiro Kurabayashi #, Rina Nunokawa, Yuta Otobe, Hiroko Kozuka-Hata, Masaaki Oyama, Yuri Shibata, Jun-Ichiro Inoue, Michinori Koebis, Atsu Aiba, Hikari Yoshitane, Yoshitaka Fukada

表观遗传

发表日期：2024

文献求助收藏

Efficacy of an eHealth self-management program in reducing irritable bowel syndrome symptom severity: a randomized controlled trial

电子健康自我管理项目在减轻肠易激综合征症状严重程度方面的疗效：一项随机对照试验

期刊:Scientific Reports

影响因子:3.9

doi:10.1038/s41598-023-50293-z

Tayama, Jun; Hamaguchi, Toyohiro; Koizumi, Kohei; Yamamura, Ryodai; Okubo, Ryo; Kawahara, Jun-Ichiro; Inoue, Kenji; Takeoka, Atsushi; Fukudo, Shin

肠易激综合征

发表日期：2024

文献求助收藏

Aberrant accumulation of NIK promotes tumor growth by dysregulating translation and post-translational modifications in breast cancer

NIK 异常积累通过失调乳腺癌中的翻译和翻译后修饰促进肿瘤生长

期刊:Cancer Cell International

影响因子:5.3

doi:10.1186/s12935-023-02904-y

Yusuke Hayashi, Jun Nakayama, Mizuki Yamamoto, Masashi Maekawa, Shinya Watanabe, Shigeki Higashiyama, Jun-Ichiro Inoue, Yusuke Yamamoto, Kentaro Semba

Translational Control

发表日期：2023

文献求助收藏

Real-Time Search-Assisted Multiplexed Quantitative Proteomics Reveals System-Wide Translational Regulation of Non-Canonical Short Open Reading Frames

实时搜索辅助多路复用定量蛋白质组学揭示非规范短开放阅读框架的系统范围翻译调控

期刊:Biomolecules

影响因子:4.8

doi:10.3390/biom13060979

Hiroko Kozuka-Hata, Tomoko Hiroki, Naoaki Miyamura, Aya Kitamura, Kouhei Tsumoto, Jun-Ichiro Inoue, Masaaki Oyama

信号转导

发表日期：2023

文献求助收藏

In Silico and In Vitro Evaluation of Some Amidine Derivatives as Hit Compounds towards Development of Inhibitors against Coronavirus Diseases

对一些脒衍生物作为冠状病毒疾病抑制剂开发的有效化合物进行计算机模拟和体外评估

期刊:Viruses-Basel

影响因子:3.8

doi:10.3390/v15051171

Ahmed H E Hassan, Selwan M El-Sayed, Mizuki Yamamoto, Jin Gohda, Takehisa Matsumoto, Mikako Shirouzu, Jun-Ichiro Inoue, Yasushi Kawaguchi, Reem M A Mansour, Abtin Anvari, Abdelbasset A Farahat

Dysfunctional Sars-CoV-2-M protein-specific cytotoxic T lymphocytes in patients recovering from severe COVID-19

重症新冠肺炎康复患者体内SARS-CoV-2-M蛋白特异性细胞毒性T淋巴细胞功能异常

期刊:Nature Communications

影响因子:14.7

doi:10.1038/s41467-022-34655-1

Hideki Ogura ，Jin Gohda ，Xiuyuan Lu ，Mizuki Yamamoto ，Yoshio Takesue ，Aoi Son ，Sadayuki Doi ，Kazuyuki Matsushita ，Fumitaka Isobe ，Yoshihiro Fukuda ，Tai-Ping Huang ，Takamasa Ueno ，Naomi Mambo ，Hiromoto Murakami ，Yasushi Kawaguchi ，Jun-Ichiro Inoue ，Kunihiro Shirai ，Sho Yamasaki ，Jun-Ichi Hirata ，Satoshi Ishido

Metalloproteinase-Dependent and TMPRSS2-Independent Cell Surface Entry Pathway of SARS-CoV-2 Requires the Furin Cleavage Site and the S2 Domain of Spike Protein

SARS-CoV-2 的金属蛋白酶依赖性和非 TMPRSS2 依赖性细胞表面进入途径需要弗林蛋白酶切割位点和刺突蛋白的 S2 结构域

期刊:mBio

影响因子:5.1

doi:10.1128/mbio.00519-22

Mizuki Yamamoto, Jin Gohda, Ayako Kobayashi, Keiko Tomita, Youko Hirayama, Naohiko Koshikawa, Motoharu Seiki, Kentaro Semba, Tetsu Akiyama, Yasushi Kawaguchi, Jun-Ichiro Inoue

Pharmacological inhibition of Mint3 attenuates tumour growth, metastasis, and endotoxic shock

Mint3 的药理学抑制可减弱肿瘤生长、转移和内毒素休克。

期刊:Communications Biology

影响因子:5.2

doi:10.1038/s42003-021-02701-1

Takeharu Sakamoto ，Yuya Fukui ，Yasumitsu Kondoh ，Kaori Honda ，Takeshi Shimizu ，Toshiro Hara ，Tetsuro Hayashi ，Yurika Saitoh ，Yoshinori Murakami ，Jun-Ichiro Inoue ，Shuichi Kaneko ，Hiroyuki Osada ，Motoharu Seiki

N-(4-Hydroxyphenyl) Retinamide Suppresses SARS-CoV-2 Spike Protein-Mediated Cell-Cell Fusion by a Dihydroceramide Δ4-Desaturase 1-Independent Mechanism

N-(4-羟基苯基)视黄酰胺通过二氢神经酰胺 Δ4-去饱和酶 1 非依赖性机制抑制 SARS-CoV-2 刺突蛋白介导的细胞间融合

期刊:Journal of Virology

影响因子:4

doi:10.1128/JVI.00807-21

Yasuhiro Hayashi, Kiyoto Tsuchiya, Mizuki Yamamoto, Yoko Nemoto-Sasaki, Kazunari Tanigawa, Kotaro Hama, Yusuke Ueda, Takashi Tanikawa, Jin Gohda, Kenji Maeda, Jun-Ichiro Inoue, Atsushi Yamashita

Monoclonal antibodies against human TMPRSS2 prevent infection by any SARS-CoV-2 variant

针对人TMPRSS2的单克隆抗体可预防任何SARS-CoV-2变种的感染

TRAF7 determines circadian period through ubiquitination and degradation of DBP

TRAF7 通过泛素化和 DBP 降解来决定昼夜节律

Efficacy of an eHealth self-management program in reducing irritable bowel syndrome symptom severity: a randomized controlled trial

电子健康自我管理项目在减轻肠易激综合征症状严重程度方面的疗效：一项随机对照试验

Aberrant accumulation of NIK promotes tumor growth by dysregulating translation and post-translational modifications in breast cancer

NIK 异常积累通过失调乳腺癌中的翻译和翻译后修饰促进肿瘤生长

Real-Time Search-Assisted Multiplexed Quantitative Proteomics Reveals System-Wide Translational Regulation of Non-Canonical Short Open Reading Frames

实时搜索辅助多路复用定量蛋白质组学揭示非规范短开放阅读框架的系统范围翻译调控

In Silico and In Vitro Evaluation of Some Amidine Derivatives as Hit Compounds towards Development of Inhibitors against Coronavirus Diseases

对一些脒衍生物作为冠状病毒疾病抑制剂开发的有效化合物进行计算机模拟和体外评估

Dysfunctional Sars-CoV-2-M protein-specific cytotoxic T lymphocytes in patients recovering from severe COVID-19

重症新冠肺炎康复患者体内SARS-CoV-2-M蛋白特异性细胞毒性T淋巴细胞功能异常

Metalloproteinase-Dependent and TMPRSS2-Independent Cell Surface Entry Pathway of SARS-CoV-2 Requires the Furin Cleavage Site and the S2 Domain of Spike Protein

SARS-CoV-2 的金属蛋白酶依赖性和非 TMPRSS2 依赖性细胞表面进入途径需要弗林蛋白酶切割位点和刺突蛋白的 S2 结构域

Pharmacological inhibition of Mint3 attenuates tumour growth, metastasis, and endotoxic shock

Mint3 的药理学抑制可减弱肿瘤生长、转移和内毒素休克。

N-(4-Hydroxyphenyl) Retinamide Suppresses SARS-CoV-2 Spike Protein-Mediated Cell-Cell Fusion by a Dihydroceramide Δ4-Desaturase 1-Independent Mechanism

N-(4-羟基苯基)视黄酰胺通过二氢神经酰胺 Δ4-去饱和酶 1 非依赖性机制抑制 SARS-CoV-2 刺突蛋白介导的细胞间融合