Junya Awata相关文献与解析，生知库 | 链接生命科学的每一步

A Barth Syndrome Patient-Derived D75H Point Mutation in TAFAZZIN Drives Progressive Cardiomyopathy in Mice

巴特综合征患者 TAFAZZIN 基因 D75H 点突变导致小鼠发生进行性心肌病

期刊:International Journal of Molecular Sciences

Paige L Snider, Elizabeth A Sierra Potchanant, Zejin Sun, Donna M Edwards, Ka-Kui Chan, Catalina Matias, Junya Awata, Aditya Sheth, P Melanie Pride, R Mark Payne, Michael Rubart, Jeffrey J Brault, Michael T Chin, Grzegorz Nalepa, Simon J Conway

A novel αB-crystallin R123W variant drives hypertrophic cardiomyopathy by promoting maladaptive calcium-dependent signal transduction

一种新型 αB-晶体蛋白 R123W 变体通过促进适应不良的钙依赖性信号转导来驱动肥厚性心肌病

期刊:Frontiers in Cardiovascular Medicine

影响因子:2.8

doi:10.3389/fcvm.2023.1223244

Chun Chou, Gregory L Martin, Gayani Perera, Junya Awata, Amy Larson, Robert Blanton, Michael T Chin

Intramuscular delivery of formulated RNA encoding six linked nanobodies is highly protective for exposures to three Botulinum neurotoxin serotypes

肌肉内注射编码六个连接纳米抗体的配制 RNA 可高度保护人体免受三种肉毒杆菌毒素血清型的侵害

期刊:Scientific Reports

影响因子:3.8

doi:10.1038/s41598-022-15876-2

Jean Mukherjee, Celinia A Ondeck, Jacqueline M Tremblay, Jacob Archer, Michelle Debatis, Alexa Foss, Junya Awata, Jesse H Erasmus, Patrick M McNutt, Charles B Shoemaker

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发表日期：2022

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Single Nucleus RNA-sequencing Reveals Altered Intercellular Communication and Dendritic Cell Activation in Nonobstructive Hypertrophic Cardiomyopathy

单核 RNA 测序揭示非阻塞性肥厚型心肌病中细胞间通讯和树突状细胞活化的改变

期刊:

影响因子:

doi:10.26502/fccm.92920277

Christina J Codden, Amy Larson, Junya Awata, Gayani Perera, Michael T Chin

DRC3 connects the N-DRC to dynein g to regulate flagellar waveform

DRC3 将 N-DRC 与动力蛋白连接起来，调节鞭毛波形

期刊:Molecular Biology of the Cell

影响因子:3.1

doi:10.1091/mbc.E15-01-0018

Junya Awata, Kangkang Song, Jianfeng Lin, Stephen M King, Michael J Sanderson, Daniela Nicastro, George B Witman

发表日期：2015

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Cell-Penetrating Peptide Enhances Tafazzin Gene Therapy in Mouse Model of Barth Syndrome

细胞穿透肽增强 Barth 综合征小鼠模型中的 Tafazzin 基因治疗

A Barth Syndrome Patient-Derived D75H Point Mutation in TAFAZZIN Drives Progressive Cardiomyopathy in Mice

巴特综合征患者 TAFAZZIN 基因 D75H 点突变导致小鼠发生进行性心肌病

A novel αB-crystallin R123W variant drives hypertrophic cardiomyopathy by promoting maladaptive calcium-dependent signal transduction

一种新型 αB-晶体蛋白 R123W 变体通过促进适应不良的钙依赖性信号转导来驱动肥厚性心肌病

Intramuscular delivery of formulated RNA encoding six linked nanobodies is highly protective for exposures to three Botulinum neurotoxin serotypes

肌肉内注射编码六个连接纳米抗体的配制 RNA 可高度保护人体免受三种肉毒杆菌毒素血清型的侵害

Single Nucleus RNA-sequencing Reveals Altered Intercellular Communication and Dendritic Cell Activation in Nonobstructive Hypertrophic Cardiomyopathy

单核 RNA 测序揭示非阻塞性肥厚型心肌病中细胞间通讯和树突状细胞活化的改变

DRC3 connects the N-DRC to dynein g to regulate flagellar waveform

DRC3 将 N-DRC 与动力蛋白连接起来，调节鞭毛波形