Kadariya Yuwaraj相关文献与解析，生知库 | 链接生命科学的每一步

Inflammation-Related IL1β/IL1R Signaling Promotes the Development of Asbestos-Induced Malignant Mesothelioma

炎症相关的IL1β/IL1R信号通路促进石棉诱发恶性间皮瘤的发生发展

doi:10.1158/1940-6207.CAPR-15-0347

Kadariya, Yuwaraj; Menges, Craig W; Talarchek, Jacqueline; Cai, Kathy Q; Klein-Szanto, Andres J; Pietrofesa, Ralph A; Christofidou-Solomidou, Melpo; Cheung, Mitchell; Mossman, Brooke T; Shukla, Arti; Testa, Joseph R

炎症/感染

信号转导

Immunology/Inflammation

炎症

发表日期：2016

文献求助收藏

Germline BAP1 mutation in a family with high incidence of multiple primary cancers and a potential gene-environment interaction

在一个多原发性癌症高发的家族中发现生殖系BAP1突变，并可能存在基因-环境相互作用。

期刊:Cancer Letters

影响因子:10.1

doi:10.1016/j.canlet.2015.09.011

Cheung, Mitchell; Kadariya, Yuwaraj; Talarchek, Jacqueline; Pei, Jianming; Ohar, Jill A; Kayaleh, Omar R; Testa, Joseph R

An asbestos-exposed family with multiple cases of pleural malignant mesothelioma without inheritance of a predisposing BAP1 mutation

一个曾接触石棉的家族中有多例胸膜恶性间皮瘤病例，但未遗传到易感基因BAP1突变。

期刊:Cancer Genetics

影响因子:2.1

doi:10.1016/j.cancergen.2015.07.004

Cheung, Mitchell; Kadariya, Yuwaraj; Pei, Jianming; Talarchek, Jacqueline; Facciolo, Francesco; Visca, Paolo; Righi, Luisella; Cozzi, Ilaria; Testa, Joseph R; Ascoli, Valeria

BAP1

发表日期：2015

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Merlin deficiency predicts FAK inhibitor sensitivity: a synthetic lethal relationship

Merlin 缺陷预示着对 FAK 抑制剂的敏感性：一种合成致死关系

期刊:Science Translational Medicine

影响因子:14.6

doi:10.1126/scitranslmed.3008639

Shapiro, Irina M; Kolev, Vihren N; Vidal, Christian M; Kadariya, Yuwaraj; Ring, Jennifer E; Wright, Quentin; Weaver, David T; Menges, Craig; Padval, Mahesh; McClatchey, Andrea I; Xu, Qunli; Testa, Joseph R; Pachter, Jonathan A

Expression of MTAP inhibits tumor-related phenotypes in HT1080 cells via a mechanism unrelated to its enzymatic function.

MTAP 的表达通过与其酶功能无关的机制抑制 HT1080 细胞中的肿瘤相关表型

期刊:G3-Genes Genomes Genetics

影响因子:2.2

doi:10.1534/g3.114.014555

Tang Baiqing, Kadariya Yuwaraj, Chen Yibai, Slifker Michael, Kruger Warren D

Regulation of human methylthioadenosine phosphorylase gene by the CBF (CCAAT binding factor)/NF-Y (nuclear factor-Y)

CBF（CCAAT结合因子）/NF-Y（核因子-Y）对人类甲基硫腺苷磷酸化酶基因的调控

期刊:Biochemical Journal

影响因子:4.3

doi:10.1042/BJ20041472

Kadariya, Yuwaraj; Nakatani, Kaname; Nishioka, Junji; Fujikawa, Takahiko; Kruger, Warren D; Nobori, Tsutomu

Molecular characterization of 5'-deoxy-5'-methylthioadenosine phosphorylase-deficient mutant clones of murine lymphoma cell line R1.1

小鼠淋巴瘤细胞系 R1.1 的 5'-脱氧-5'-甲基硫代腺苷磷酸化酶缺陷突变克隆的分子特征

期刊:Cancer Science

影响因子:4.3

doi:10.1111/j.1349-7006.2003.tb01476.x

Kadariya, Yuwaraj; Nishioka, Junji; Nakamura, Akiko; Kato-Nakazawa, Keiko; Nobori, Tsutomu

Spontaneous Mesotheliomas in Germline Bap1 Heterozygous Mice from Different Genetic Backgrounds

不同遗传背景的生殖系Bap1杂合子小鼠自发性间皮瘤

Modeling Malignant Mesothelioma in Genetically Engineered Mice.

在基因工程小鼠中构建恶性间皮瘤模型

Inflammation-Related IL1β/IL1R Signaling Promotes the Development of Asbestos-Induced Malignant Mesothelioma

炎症相关的IL1β/IL1R信号通路促进石棉诱发恶性间皮瘤的发生发展

Germline BAP1 mutation in a family with high incidence of multiple primary cancers and a potential gene-environment interaction

在一个多原发性癌症高发的家族中发现生殖系BAP1突变，并可能存在基因-环境相互作用。

An asbestos-exposed family with multiple cases of pleural malignant mesothelioma without inheritance of a predisposing BAP1 mutation

一个曾接触石棉的家族中有多例胸膜恶性间皮瘤病例，但未遗传到易感基因BAP1突变。

Merlin deficiency predicts FAK inhibitor sensitivity: a synthetic lethal relationship

Merlin 缺陷预示着对 FAK 抑制剂的敏感性：一种合成致死关系

Expression of MTAP inhibits tumor-related phenotypes in HT1080 cells via a mechanism unrelated to its enzymatic function.

MTAP 的表达通过与其酶功能无关的机制抑制 HT1080 细胞中的肿瘤相关表型

Regulation of human methylthioadenosine phosphorylase gene by the CBF (CCAAT binding factor)/NF-Y (nuclear factor-Y)

CBF（CCAAT结合因子）/NF-Y（核因子-Y）对人类甲基硫腺苷磷酸化酶基因的调控

Molecular characterization of 5'-deoxy-5'-methylthioadenosine phosphorylase-deficient mutant clones of murine lymphoma cell line R1.1

小鼠淋巴瘤细胞系 R1.1 的 5'-脱氧-5'-甲基硫代腺苷磷酸化酶缺陷突变克隆的分子特征