Kagami M相关文献与解析，生知库 | 链接生命科学的每一步

Glandular and extraglandular expression of costimulatory molecules in patients with SjÃ¶gren's syndrome.

干燥综合征患者腺体和腺外共刺激分子的表达

期刊:Annals of the Rheumatic Diseases

影响因子:20.6

doi:10.1136/ard.60.5.473

Matsumura R, Umemiya K, Goto T, Nakazawa T, Kagami M, Tomioka H, Tanabe E, Sugiyama T, Sueishi M

其它

发表日期：2001

文献求助收藏

Role of JAK2 signal transductional pathway in activation and survival of human peripheral eosinophils by interferon-gamma (IFN-gamma)

JAK2信号转导通路在干扰素-γ（IFN-γ）激活和存活人外周血嗜酸性粒细胞中的作用

期刊:Clinical and Experimental Immunology

影响因子:3.8

doi:10.1046/j.1365-2249.1999.01068.x

Ochiai, K; Tanabe, E; Ishihara, C; Kagami, M; Sugiyama, T; Sueishi, M; Koya, N; Tomioka, H

SRO9, a multicopy suppressor of the bud growth defect in the Saccharomyces cerevisiae rho3-deficient cells, shows strong genetic interactions with tropomyosin genes, suggesting its role in organization of the actin cytoskeleton

SRO9是酿酒酵母rho3缺陷细胞中芽生长缺陷的多拷贝抑制因子，它与原肌球蛋白基因表现出强烈的遗传相互作用，表明其在肌动蛋白细胞骨架的组织中发挥作用。

期刊:Genetics

影响因子:5.1

doi:10.1093/genetics/147.3.1003

Kagami, M; Toh-e, A; Matsui, Y

细胞生物学

发表日期：1997

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IL-5 but not interferon-gamma (IFN-gamma) inhibits eosinophil apoptosis by up-regulation of bcl-2 expression

白细胞介素-5（IL-5）而非干扰素-γ（IFN-γ）通过上调bcl-2表达抑制嗜酸性粒细胞凋亡。

期刊:Clinical and Experimental Immunology

影响因子:3.8

doi:10.1046/j.1365-2249.1997.d01-884.x

Ochiai, K; Kagami, M; Matsumura, R; Tomioka, H

Human peripheral eosinophils express functional interferon-gamma receptors (IFN-gammaR)

人类外周血嗜酸性粒细胞表达功能性干扰素-γ受体（IFN-γR）

期刊:Clinical and Experimental Immunology

影响因子:3.8

doi:10.1046/j.1365-2249.1997.4511469.x

Ishihara, C; Ochiai, K; Kagami, M; Takashahi, H; Matsuyama, G; Yoshida, S; Tomioka, H; Koya, N

Parthenogenetic chimaerism/mosaicism with a Silver-Russell syndrome-like phenotype

具有类似Silver-Russell综合征表型的孤雌生殖嵌合体/嵌合体

Glandular and extraglandular expression of costimulatory molecules in patients with SjÃ¶gren's syndrome.

干燥综合征患者腺体和腺外共刺激分子的表达

Role of JAK2 signal transductional pathway in activation and survival of human peripheral eosinophils by interferon-gamma (IFN-gamma)

JAK2信号转导通路在干扰素-γ（IFN-γ）激活和存活人外周血嗜酸性粒细胞中的作用

SRO9, a multicopy suppressor of the bud growth defect in the Saccharomyces cerevisiae rho3-deficient cells, shows strong genetic interactions with tropomyosin genes, suggesting its role in organization of the actin cytoskeleton

SRO9是酿酒酵母rho3缺陷细胞中芽生长缺陷的多拷贝抑制因子，它与原肌球蛋白基因表现出强烈的遗传相互作用，表明其在肌动蛋白细胞骨架的组织中发挥作用。

IL-5 but not interferon-gamma (IFN-gamma) inhibits eosinophil apoptosis by up-regulation of bcl-2 expression

白细胞介素-5（IL-5）而非干扰素-γ（IFN-γ）通过上调bcl-2表达抑制嗜酸性粒细胞凋亡。

Human peripheral eosinophils express functional interferon-gamma receptors (IFN-gammaR)

人类外周血嗜酸性粒细胞表达功能性干扰素-γ受体（IFN-γR）