Kanno Yosuke相关文献与解析，生知库 | 链接生命科学的每一步

Î±2-Antiplasmin is associated with macrophage activation and fibrin deposition in a macrophage activation syndrome mouse model.

在巨噬细胞活化综合征小鼠模型中，α2-抗纤溶酶与巨噬细胞活化和纤维蛋白沉积有关

期刊:Clinical and Experimental Immunology

影响因子:3.8

doi:10.1093/cei/uxae021

Kanno Yosuke, Toyama Kinomi, Shibata Haruna, Matsuo Osamu, Ozaki Kei-Ichi

α2-Antiplasmin as a Potential Therapeutic Target for Systemic Sclerosis

α2-抗纤溶酶作为系统性硬化症的潜在治疗靶点

期刊:Life-Basel

影响因子:3.4

doi:10.3390/life12030396

Kanno, Yosuke; Shu, En

Alternatively activated macrophages are associated with the α2AP production that occurs with the development of dermal fibrosis : The role of alternatively activated macrophages on the development of fibrosis

替代激活的巨噬细胞与真皮纤维化发展过程中发生的α2AP产生有关：替代激活的巨噬细胞在纤维化发展中的作用

期刊:Arthritis Research & Therapy

影响因子:4.6

doi:10.1186/s13075-020-02159-2

Kanno, Yosuke; Shu, En; Niwa, Hirofumi; Kanoh, Hiroyuki; Seishima, Mariko

α2AP regulates vascular alteration by inhibiting VEGF signaling in systemic sclerosis: the roles of α2AP in vascular dysfunction in systemic sclerosis

α2AP通过抑制VEGF信号通路调节系统性硬化症中的血管改变：α2AP在系统性硬化症血管功能障碍中的作用

期刊:Arthritis Research & Therapy

影响因子:4.6

doi:10.1186/s13075-017-1227-y

Kanno, Yosuke; Shu, En; Kanoh, Hiroyuki; Matsuda, Ayaka; Seishima, Mariko

uPA-derived peptide, Å6 is involved in the suppression of lipopolysaccaride-promoted inflammatory osteoclastogenesis and the resultant bone loss

uPA衍生肽Å6参与抑制脂多糖促进的炎症性破骨细胞生成以及由此导致的骨丢失。

期刊:Immunity Inflammation and Disease

影响因子:2.7

doi:10.1002/iid3.169

Kanno, Yosuke; Maruyama, Chihiro; Matsuda, Ayaka; Ishisaki, Akira

The blocking of uPAR suppresses lipopolysaccharide-induced inflammatory osteoclastogenesis and the resultant bone loss through attenuation of integrin β3/Akt pathway

阻断uPAR可通过减弱整合素β3/Akt通路来抑制脂多糖诱导的炎症性破骨细胞生成及其导致的骨丢失。

期刊:Immunity Inflammation and Disease

影响因子:2.7

doi:10.1002/iid3.116

Kanno, Yosuke; Ishisaki, Akira; Miyashita, Mei; Matsuo, Osamu

Altered behavior in mice with deletion of the alpha2-antiplasmin gene

α2-抗纤溶酶基因缺失小鼠的行为改变

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0097947

Kawashita, Eri; Kanno, Yosuke; Ikeda, Kanako; Kuretake, Hiromi; Matsuo, Osamu; Matsuno, Hiroyuki

发表日期：2014

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Bridge Formation in Phosphorylase Kinase♦: Structure and Location of the Regulatory β Subunits in the (αβγδ)(4) Phosphorylase Kinase Complex

磷酸化酶激酶中的桥接形成♦：(αβγδ)(4)磷酸化酶激酶复合物中调节性β亚基的结构和位置

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.A112.347690

Niimi, Shinichiro; Arakawa-Takeuchi, Shiho; Uranbileg, Baasanjav; Park, Jung-ha; Jinno, Shigeki; Okayama, Hiroto; Nguyen, Laura A; Kim, Dong-Hyun; Daly, Michele B; Allan, Kevin C; Kim, Baek; Shao, HanJuan; Lee, Seakwoo; Gae-Scott, Sherri; Nakata, Chiaki; Chen, Shoujun; Hamad, Abdel R; Chakravarti, Shukti; Kanno, Yosuke; Ishisaki, Akira; Kawashita, Eri; Chosa, Naoyuki; Nakajima, Keiichi; Nishihara, Tatsuji; Toyoshima, Kuniaki; Okada, Kiyotaka; Ueshima, Shigeru; Matsushita, Kenji; Matsuo, Osamu; Matsuno, Hiroyuki; Schlam, Daniel; Bohdanowicz, Michal; Chatgilialoglu, Alexandros; Steinberg, Benjamin E; Ueyama, Takehiko; Du, Guangwei; Grinstein, Sergio; Fairn, Gregory D; Kong, Guangyao; Du, Juan; Liu, Yangang; Meline, Benjamin; Chang, Yuan-I; Ranheim, Erik A; Wang, Jinyong; Zhang, Jing; Zhao, Xue; Yang, Bo; Solakyildirim, Kemal; Joo, Eun Ji; Toida, Toshihiko; Higashi, Kyohei; Linhardt, Robert J; Li, Lingyun

Plasminogen/plasmin modulates bone metabolism by regulating the osteoblast and osteoclast function

纤溶酶原/纤溶酶通过调节成骨细胞和破骨细胞的功能来调节骨代谢。

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.M110.152181

Kanno, Yosuke; Ishisaki, Akira; Kawashita, Eri; Chosa, Naoyuki; Nakajima, Keiichi; Nishihara, Tatsuji; Toyoshima, Kuniaki; Okada, Kiyotaka; Ueshima, Shigeru; Matsushita, Kenji; Matsuo, Osamu; Matsuno, Hiroyuki

Pathogenic and Regulatory Roles of Fibrinolytic Factors in Autoimmune Diseases

纤溶因子在自身免疫性疾病中的致病和调控作用

Î±2-Antiplasmin is associated with macrophage activation and fibrin deposition in a macrophage activation syndrome mouse model.

在巨噬细胞活化综合征小鼠模型中，α2-抗纤溶酶与巨噬细胞活化和纤维蛋白沉积有关

α2-Antiplasmin as a Potential Therapeutic Target for Systemic Sclerosis

α2-抗纤溶酶作为系统性硬化症的潜在治疗靶点

Alternatively activated macrophages are associated with the α2AP production that occurs with the development of dermal fibrosis : The role of alternatively activated macrophages on the development of fibrosis

替代激活的巨噬细胞与真皮纤维化发展过程中发生的α2AP产生有关：替代激活的巨噬细胞在纤维化发展中的作用

α2AP regulates vascular alteration by inhibiting VEGF signaling in systemic sclerosis: the roles of α2AP in vascular dysfunction in systemic sclerosis

α2AP通过抑制VEGF信号通路调节系统性硬化症中的血管改变：α2AP在系统性硬化症血管功能障碍中的作用

uPA-derived peptide, Å6 is involved in the suppression of lipopolysaccaride-promoted inflammatory osteoclastogenesis and the resultant bone loss

uPA衍生肽Å6参与抑制脂多糖促进的炎症性破骨细胞生成以及由此导致的骨丢失。

The blocking of uPAR suppresses lipopolysaccharide-induced inflammatory osteoclastogenesis and the resultant bone loss through attenuation of integrin β3/Akt pathway

阻断uPAR可通过减弱整合素β3/Akt通路来抑制脂多糖诱导的炎症性破骨细胞生成及其导致的骨丢失。

Altered behavior in mice with deletion of the alpha2-antiplasmin gene

α2-抗纤溶酶基因缺失小鼠的行为改变

Bridge Formation in Phosphorylase Kinase♦: Structure and Location of the Regulatory β Subunits in the (αβγδ)(4) Phosphorylase Kinase Complex

磷酸化酶激酶中的桥接形成♦：(αβγδ)(4)磷酸化酶激酶复合物中调节性β亚基的结构和位置

Plasminogen/plasmin modulates bone metabolism by regulating the osteoblast and osteoclast function

纤溶酶原/纤溶酶通过调节成骨细胞和破骨细胞的功能来调节骨代谢。